ResearcherUniversity Hospital CologneActive in the last 2 years
Zhefang Wang
Matched on ORCID iD 0000-0002-2297-2586; OpenAlex author ID A5012897253. Sources: OpenAlex.
Their work
- Papers
- 6
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 18
2019: 1 paper · 2020: 1 paper · 2021: 2 papers · 2022: none · 2023: none · 2024: none · 2025: none · 2026: 2 papers
Papers 6
- LSD1-GLS2 axis drives subtype-specific chemoresistance in pancreatic cancer through glutaminolysis reprogramming. Co-author DOI 10.1038/s41419-026-09075-4↗
- Mitophagy in pancreatic cancer: mechanistic insights and implications for novel therapeutic strategies. Co-author DOI 10.1038/s41420-026-02948-9↗
- The molecular biology of pancreatic adenocarcinoma: translational challenges and clinical perspectives. Co-author DOI 10.1038/s41392-021-00659-4↗
- IL-17B/RB Activation in Pancreatic Stellate Cells Promotes Pancreatic Cancer Metabolism and Growth. Co-author DOI 10.3390/cancers13215338↗
- Inflammatory IFIT3 renders chemotherapy resistance by regulating post-translational modification of VDAC2 in pancreatic cancer. Co-author DOI 10.7150/thno.43093↗
- Tumor-Derived Extracellular Vesicles Inhibit Natural Killer Cell Function in Pancreatic Cancer. Co-author DOI 10.3390/cancers11060874↗
Closest collaborators
The 18 strongest of 18 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Christiane J. Bruns · 6 shared papers, 2019–2026 · strength 0.97
- Paper Mitophagy in pancreatic cancer: mechanistic insights and implications for novel therapeutic strategies. DOI 10.1038/s41420-026-02948-9↗
- Paper LSD1-GLS2 axis drives subtype-specific chemoresistance in pancreatic cancer through glutaminolysis reprogramming. DOI 10.1038/s41419-026-09075-4↗
- Paper IL-17B/RB Activation in Pancreatic Stellate Cells Promotes Pancreatic Cancer Metabolism and Growth. DOI 10.3390/cancers13215338↗
- Yue Zhao · 6 shared papers, 2019–2026 · strength 0.97
- Paper Mitophagy in pancreatic cancer: mechanistic insights and implications for novel therapeutic strategies. DOI 10.1038/s41420-026-02948-9↗
- Paper LSD1-GLS2 axis drives subtype-specific chemoresistance in pancreatic cancer through glutaminolysis reprogramming. DOI 10.1038/s41419-026-09075-4↗
- Paper IL-17B/RB Activation in Pancreatic Stellate Cells Promotes Pancreatic Cancer Metabolism and Growth. DOI 10.3390/cancers13215338↗
- Qiongzhu Dong · 4 shared papers, 2020–2026 · strength 0.77
- Paper Mitophagy in pancreatic cancer: mechanistic insights and implications for novel therapeutic strategies. DOI 10.1038/s41420-026-02948-9↗
- Paper LSD1-GLS2 axis drives subtype-specific chemoresistance in pancreatic cancer through glutaminolysis reprogramming. DOI 10.1038/s41419-026-09075-4↗
- Paper The molecular biology of pancreatic adenocarcinoma: translational challenges and clinical perspectives. DOI 10.1038/s41392-021-00659-4↗
- Felix Popp · 4 shared papers, 2019–2026 · strength 0.61
- Paper Mitophagy in pancreatic cancer: mechanistic insights and implications for novel therapeutic strategies. DOI 10.1038/s41420-026-02948-9↗
- Paper IL-17B/RB Activation in Pancreatic Stellate Cells Promotes Pancreatic Cancer Metabolism and Growth. DOI 10.3390/cancers13215338↗
- Paper Inflammatory IFIT3 renders chemotherapy resistance by regulating post-translational modification of VDAC2 in pancreatic… DOI 10.7150/thno.43093↗
- Jiangang Zhao · 3 shared papers, 2019–2026 · strength 0.43
- Paper LSD1-GLS2 axis drives subtype-specific chemoresistance in pancreatic cancer through glutaminolysis reprogramming. DOI 10.1038/s41419-026-09075-4↗
- Paper Inflammatory IFIT3 renders chemotherapy resistance by regulating post-translational modification of VDAC2 in pancreatic… DOI 10.7150/thno.43093↗
- Paper Tumor-Derived Extracellular Vesicles Inhibit Natural Killer Cell Function in Pancreatic Cancer. DOI 10.3390/cancers11060874↗
- Alexander Quaas · 2 shared papers, 2021–2026 · strength 0.36
- Jiahui Li · 3 shared papers, 2019–2021 · strength 0.29
- Paper IL-17B/RB Activation in Pancreatic Stellate Cells Promotes Pancreatic Cancer Metabolism and Growth. DOI 10.3390/cancers13215338↗
- Paper Inflammatory IFIT3 renders chemotherapy resistance by regulating post-translational modification of VDAC2 in pancreatic… DOI 10.7150/thno.43093↗
- Paper Tumor-Derived Extracellular Vesicles Inhibit Natural Killer Cell Function in Pancreatic Cancer. DOI 10.3390/cancers11060874↗
- Maximilian Reichert · 1 shared paper, 2026 · strength 0.25
- Hana Algül · 1 shared paper, 2026 · strength 0.25
- Bo Kong · 2 shared papers, 2020–2021 · strength 0.21
- Rainer Lothar Heuchel · 1 shared paper, 2021 · strength 0.11
- Feng Yang · 1 shared paper, 2021 · strength 0.11
- Beiyuan Hu · 1 shared paper, 2021 · strength 0.11
- Yan Zheng · 1 shared paper, 2021 · strength 0.11
- Jiayan Yan · 1 shared paper, 2021 · strength 0.11
- Shun Wang · 1 shared paper, 2021 · strength 0.11
- Deliang Fu · 1 shared paper, 2021 · strength 0.11
- Hans Schlößer · 1 shared paper, 2019 · strength 0.08
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.