ResearcherCincinnati Children's Hospital Medical CenterActive in the last 2 years
Maximilian Reichert
Matched on ORCID iD 0000-0002-8611-5639; OpenAlex author ID A5073445337. Sources: OpenAlex.
Their work
- Papers
- 58 (first author 2, last author 7)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 268
2012: 1 paper · 2013: 3 papers · 2014: none · 2015: none · 2016: 4 papers · 2017: 1 paper · 2018: 4 papers · 2019: 1 paper · 2020: 4 papers · 2021: 6 papers · 2022: 9 papers · 2023: 2 papers · 2024: 6 papers · 2025: 10 papers · 2026: 7 papers
Papers 50 of 58
- The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1002/mco2.70794↗
- Precision Medicine in Pancreatic Cancer: Targeting KRAS and Beyond. Co-author DOI 10.1007/s11523-026-01236-x↗
- LSD1-GLS2 axis drives subtype-specific chemoresistance in pancreatic cancer through glutaminolysis reprogramming. Co-author DOI 10.1038/s41419-026-09075-4↗
- Oncogenic KRAS-driven type I interferon signalling primes pancreatic cancer for necroptosis. Co-author DOI 10.1038/s41467-026-73189-8↗
- Multicellular rosette formation guides epithelial tissue assembly in pancreatic ductal adenocarcinoma cell organoids. Co-author DOI 10.1073/pnas.2512284123↗
- Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. Co-author DOI 10.1158/2767-9764.crc-25-0670↗
- Modeling pancreatic cancer tumor stroma co-evolution in an in ovo model Last author DOI 10.64898/2026.06.26.734719↗
- KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. Co-author DOI 10.1002/1878-0261.13725↗
- Development of a translational radiobiology platform using pancreatic cancer patient-derived organoids for personalized radiation oncology. Co-author DOI 10.1016/j.tranon.2025.102535↗
- Disrupting AGR2/IGF1 paracrine and reciprocal signaling for pancreatic cancer therapy. Co-author DOI 10.1016/j.xcrm.2024.101927↗
- A decision point between transdifferentiation and programmed cell death priming controls KRAS-dependent pancreatic cancer development. Co-author DOI 10.1038/s41467-025-56493-7↗
- Heterogeneity-driven phenotypic plasticity and treatment response in branched-organoid models of pancreatic ductal adenocarcinoma. Last author DOI 10.1038/s41551-024-01273-9↗
- Gemcitabine and ATR inhibitors synergize to kill PDAC cells by blocking DNA damage response. Co-author DOI 10.1038/s44320-025-00085-6↗
- A 3D patternoid model for the reproducible characterization of invasive phenotypes and drug sensitivity in PDAC. Co-author DOI 10.1039/d5lc00203f↗
- The Biology in the Pattern: Metastatic Organotropism and Clinical Outcome Depend on DNA Damage Response and Immune Interactions in Pancreatic Cancer. First author DOI 10.1158/0008-5472.can-25-1085↗
- Label-free single-cell phenotyping to determine tumor cell heterogeneity in pancreatic cancer in real time. Last author DOI 10.1172/jci.insight.169105↗
- c-Rel drives pancreatic cancer metastasis through fibronectin-integrin signaling-induced isolation stress resistance and EMT. Co-author DOI 10.1186/s12943-025-02486-5↗
- A Novel AMPK Inhibitor Sensitizes Pancreatic Cancer Cells to Ferroptosis Induction. Co-author DOI 10.1002/advs.202307695↗
- Trametinib in combination with hydroxychloroquine or palbociclib in advanced metastatic pancreatic cancer: data from a retrospective, multicentric cohort (AIO … Co-author DOI 10.1007/s00432-024-05954-5↗
- Coexisting mechanisms of luminogenesis in pancreatic cancer-derived organoids. Co-author DOI 10.1016/j.isci.2024.110299↗
- Alternating gemcitabine plus nab-paclitaxel and gemcitabine alone versus continuous gemcitabine plus nab-paclitaxel after induction treatment of metastatic pan… Co-author DOI 10.1016/s2468-1253(24)00197-3↗
- An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. Co-author DOI 10.1053/j.gastro.2023.10.026↗
- Comparison of endoscopic ultrasound-guided fine-needle aspiration and fine-needle biopsy to generate pancreatic cancer organoids: Randomized trial. Co-author DOI 10.1055/a-2257-3171↗
- In-vitro model to mimic T cell subset change in human PDAC organoid co-culture. Co-author DOI 10.1007/s00432-023-05100-7↗
- Phenotype screens of murine pancreatic cancer identify a Tgf-α-Ccl2-paxillin axis driving human-like neural invasion. Co-author DOI 10.1172/jci166333↗
- Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. Co-author DOI 10.1002/cac2.12280↗
- Detecting drug resistance in pancreatic cancer organoids guides optimized chemotherapy treatment. Co-author DOI 10.1002/path.5906↗
- AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. Co-author DOI 10.1007/s00018-022-04638-y↗
- A Pancreatic Cancer Patient-Derived Xenograft Model for Adoptive Immunotherapy Using Autologous Tumor-Infiltrating Lymphocytes. Last author DOI 10.1016/j.gastha.2022.08.008↗
- Selective multi-kinase inhibition sensitizes mesenchymal pancreatic cancer to immune checkpoint blockade by remodeling the tumor microenvironment. Co-author DOI 10.1038/s43018-021-00326-1↗
- NOXA expression drives synthetic lethality to RUNX1 inhibition in pancreatic cancer. Co-author DOI 10.1073/pnas.2105691119↗
- HDAC2 Facilitates Pancreatic Cancer Metastasis. Co-author DOI 10.1158/0008-5472.can-20-3209↗
- Epigenetic drug screening defines a PRMT5 inhibitor-sensitive pancreatic cancer subtype. Co-author DOI 10.1172/jci.insight.151353↗
- Identification of treatment-induced vulnerabilities in pancreatic cancer patients using functional model systems. Last author DOI 10.15252/emmm.202114876↗
- Mesenchymal Plasticity Regulated by Prrx1 Drives Aggressive Pancreatic Cancer Biology. Last author DOI 10.1053/j.gastro.2020.09.010↗
- New Insights Into Pancreatic Cancer: Notes from a Virtual Meeting. Co-author DOI 10.1053/j.gastro.2021.04.082↗
- AGR2-Dependent Nuclear Import of RNA Polymerase II Constitutes a Specific Target of Pancreatic Ductal Adenocarcinoma in the Context of Wild-Type p53. Co-author DOI 10.1053/j.gastro.2021.07.030↗
- Phosphoproteomics Identifies PI3K Inhibitor-selective Adaptive Responses in Pancreatic Cancer Cell Therapy and Resistance. Co-author DOI 10.1158/1535-7163.mct-20-0981↗
- PALLD mutation in a European family conveys a stromal predisposition for familial pancreatic cancer. Co-author DOI 10.1172/jci.insight.141532↗
- [18F]FDG PET/MRI enables early chemotherapy response prediction in pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s13550-021-00808-4↗
- Targeting the ubiquitin-proteasome system in a pancreatic cancer subtype with hyperactive MYC. Co-author DOI 10.1002/1878-0261.12835↗
- SUMO pathway inhibition targets an aggressive pancreatic cancer subtype. Co-author DOI 10.1136/gutjnl-2018-317856↗
- Loss of Wasl improves pancreatic cancer outcome. Co-author DOI 10.1172/jci.insight.127275↗
- Implementing cell-free DNA of pancreatic cancer patient-derived organoids for personalized oncology. Last author DOI 10.1172/jci.insight.137809↗
- PRRX1 isoforms cooperate with FOXM1 to regulate the DNA damage response in pancreatic cancer cells. Co-author DOI 10.1038/s41388-019-0725-6↗
- β2 Adrenergic-Neurotrophin Feedforward Loop Promotes Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2017.11.007↗
- Regulation of Epithelial Plasticity Determines Metastatic Organotropism in Pancreatic Cancer. Co-author DOI 10.1016/j.devcel.2018.05.025↗
- Pancreas 3D Organoids: Current and Future Aspects as a Research Platform for Personalized Medicine in Pancreatic Cancer. Co-author DOI 10.1016/j.jcmgh.2017.12.004↗
- MTOR inhibitor-based combination therapies for pancreatic cancer. Co-author DOI 10.1038/bjc.2017.421↗
- HDAC1 and HDAC2 integrate the expression of p53 mutants in pancreatic cancer. Co-author DOI 10.1038/onc.2016.344↗
Closest collaborators
The 25 strongest of 268 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Günter Schneider · 27 shared papers, 2013–2026 · strength 3.10
- Paper Modeling pancreatic cancer tumor stroma co-evolution in an in ovo model DOI 10.64898/2026.06.26.734719↗
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Dieter Saur · 25 shared papers, 2013–2026 · strength 3.01
- Paper Coexisting mechanisms of luminogenesis in pancreatic cancer-derived organoids. DOI 10.1016/j.isci.2024.110299↗
- Paper Modeling pancreatic cancer tumor stroma co-evolution in an in ovo model DOI 10.64898/2026.06.26.734719↗
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Roland Michael Schmid · 23 shared papers, 2013–2026 · strength 2.68
- Paper Modeling pancreatic cancer tumor stroma co-evolution in an in ovo model DOI 10.64898/2026.06.26.734719↗
- Paper Development of a translational radiobiology platform using pancreatic cancer patient-derived organoids for personalized… DOI 10.1016/j.tranon.2025.102535↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Roland Rad · 21 shared papers, 2017–2026 · strength 2.48
- Paper Modeling pancreatic cancer tumor stroma co-evolution in an in ovo model DOI 10.64898/2026.06.26.734719↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper Label-free single-cell phenotyping to determine tumor cell heterogeneity in pancreatic cancer in real time. DOI 10.1172/jci.insight.169105↗
- Matthias Wirth · 17 shared papers, 2013–2026 · strength 1.82
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Felix Orben · 11 shared papers, 2020–2026 · strength 1.51
- Paper Development of a translational radiobiology platform using pancreatic cancer patient-derived organoids for personalized… DOI 10.1016/j.tranon.2025.102535↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper Comparison of endoscopic ultrasound-guided fine-needle aspiration and fine-needle biopsy to generate pancreatic cancer … DOI 10.1055/a-2257-3171↗
- Christian Schneeweis · 11 shared papers, 2018–2026 · strength 1.36
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Paper Heterogeneity-driven phenotypic plasticity and treatment response in branched-organoid models of pancreatic ductal aden… DOI 10.1038/s41551-024-01273-9↗
- Aristeidis Papargyriou · 7 shared papers, 2020–2025 · strength 1.33
- Paper The Biology in the Pattern: Metastatic Organotropism and Clinical Outcome Depend on DNA Damage Response and Immune Inte… DOI 10.1158/0008-5472.can-25-1085↗
- Paper Label-free single-cell phenotyping to determine tumor cell heterogeneity in pancreatic cancer in real time. DOI 10.1172/jci.insight.169105↗
- Paper A decision point between transdifferentiation and programmed cell death priming controls KRAS-dependent pancreatic canc… DOI 10.1038/s41467-025-56493-7↗
- Carolin Schneider · 9 shared papers, 2022–2026 · strength 1.32
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Lukas Krauß · 8 shared papers, 2022–2026 · strength 1.19
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Andreas R. Bausch · 4 shared papers, 2024–2026 · strength 1.15
- Paper Multicellular rosette formation guides epithelial tissue assembly in pancreatic ductal adenocarcinoma cell organoids. DOI 10.1073/pnas.2512284123↗
- Paper A 3D patternoid model for the reproducible characterization of invasive phenotypes and drug sensitivity in PDAC. DOI 10.1039/d5lc00203f↗
- Paper Coexisting mechanisms of luminogenesis in pancreatic cancer-derived organoids. DOI 10.1016/j.isci.2024.110299↗
- Elisabeth Heßmann · 7 shared papers, 2021–2026 · strength 1.05
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Hana Algül · 8 shared papers, 2013–2026 · strength 0.99
- Paper LSD1-GLS2 axis drives subtype-specific chemoresistance in pancreatic cancer through glutaminolysis reprogramming. DOI 10.1038/s41419-026-09075-4↗
- Paper c-Rel drives pancreatic cancer metastasis through fibronectin-integrin signaling-induced isolation stress resistance an… DOI 10.1186/s12943-025-02486-5↗
- Paper A decision point between transdifferentiation and programmed cell death priming controls KRAS-dependent pancreatic canc… DOI 10.1038/s41467-025-56493-7↗
- H. Carlo Maurer · 7 shared papers, 2018–2026 · strength 0.96
- Paper Modeling pancreatic cancer tumor stroma co-evolution in an in ovo model DOI 10.64898/2026.06.26.734719↗
- Paper Development of a translational radiobiology platform using pancreatic cancer patient-derived organoids for personalized… DOI 10.1016/j.tranon.2025.102535↗
- Paper Label-free single-cell phenotyping to determine tumor cell heterogeneity in pancreatic cancer in real time. DOI 10.1172/jci.insight.169105↗
- Volker Ellenrieder · 6 shared papers, 2021–2026 · strength 0.93
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Oliver Holger Krämer · 9 shared papers, 2013–2026 · strength 0.93
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Wilko Weichert · 9 shared papers, 2018–2025 · strength 0.89
- Paper Development of a translational radiobiology platform using pancreatic cancer patient-derived organoids for personalized… DOI 10.1016/j.tranon.2025.102535↗
- Paper Identification of treatment-induced vulnerabilities in pancreatic cancer patients using functional model systems. DOI 10.15252/emmm.202114876↗
- Paper PALLD mutation in a European family conveys a stromal predisposition for familial pancreatic cancer. DOI 10.1172/jci.insight.141532↗
- İhsan Ekin Demir · 6 shared papers, 2021–2025 · strength 0.87
- Paper Development of a translational radiobiology platform using pancreatic cancer patient-derived organoids for personalized… DOI 10.1016/j.tranon.2025.102535↗
- Paper Label-free single-cell phenotyping to determine tumor cell heterogeneity in pancreatic cancer in real time. DOI 10.1172/jci.insight.169105↗
- Paper c-Rel drives pancreatic cancer metastasis through fibronectin-integrin signaling-induced isolation stress resistance an… DOI 10.1186/s12943-025-02486-5↗
- Katja Peschke · 6 shared papers, 2020–2025 · strength 0.86
- Paper A Pancreatic Cancer Patient-Derived Xenograft Model for Adoptive Immunotherapy Using Autologous Tumor-Infiltrating Lymp… DOI 10.1016/j.gastha.2022.08.008↗
- Paper c-Rel drives pancreatic cancer metastasis through fibronectin-integrin signaling-induced isolation stress resistance an… DOI 10.1186/s12943-025-02486-5↗
- Paper Heterogeneity-driven phenotypic plasticity and treatment response in branched-organoid models of pancreatic ductal aden… DOI 10.1038/s41551-024-01273-9↗
- Marian Grade · 5 shared papers, 2022–2026 · strength 0.83
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Zahra Dantes · 9 shared papers, 2018–2025 · strength 0.83
- Paper Disrupting AGR2/IGF1 paracrine and reciprocal signaling for pancreatic cancer therapy. DOI 10.1016/j.xcrm.2024.101927↗
- Paper Phosphoproteomics Identifies PI3K Inhibitor-selective Adaptive Responses in Pancreatic Cancer Cell Therapy and Resistan… DOI 10.1158/1535-7163.mct-20-0981↗
- Paper Mesenchymal Plasticity Regulated by Prrx1 Drives Aggressive Pancreatic Cancer Biology. DOI 10.1053/j.gastro.2020.09.010↗
- Zonera Hassan · 8 shared papers, 2017–2022 · strength 0.77
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Stefanie Bärthel · 5 shared papers, 2022–2026 · strength 0.76
- Paper Modeling pancreatic cancer tumor stroma co-evolution in an in ovo model DOI 10.64898/2026.06.26.734719↗
- Paper Label-free single-cell phenotyping to determine tumor cell heterogeneity in pancreatic cancer in real time. DOI 10.1172/jci.insight.169105↗
- Paper Heterogeneity-driven phenotypic plasticity and treatment response in branched-organoid models of pancreatic ductal aden… DOI 10.1038/s41551-024-01273-9↗
- Bernhard Küster · 5 shared papers, 2022–2025 · strength 0.73
- Paper Gemcitabine and ATR inhibitors synergize to kill PDAC cells by blocking DNA damage response. DOI 10.1038/s44320-025-00085-6↗
- Paper Label-free single-cell phenotyping to determine tumor cell heterogeneity in pancreatic cancer in real time. DOI 10.1172/jci.insight.169105↗
- Paper A Novel AMPK Inhibitor Sensitizes Pancreatic Cancer Cells to Ferroptosis Induction. DOI 10.1002/advs.202307695↗
- Michael B. Ghadimi · 4 shared papers, 2024–2026 · strength 0.71
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.