ResearcherPhilipps University of MarburgActive in the last 2 years
Volker Ellenrieder
Matched on ORCID iD 0000-0001-9981-8571; OpenAlex author ID A5046422102. Sources: OpenAlex.
Their work
- Papers
- 50 (first author 3, last author 7)
- Trials
- 0
- Grants
- 0
- Labs led
- 2
- Collaborators
- 143
2011: 2 papers · 2012: 1 paper · 2013: 1 paper · 2014: 4 papers · 2015: 3 papers · 2016: 4 papers · 2017: 1 paper · 2018: 1 paper · 2019: 5 papers · 2020: 2 papers · 2021: 4 papers · 2022: 3 papers · 2023: 2 papers · 2024: 7 papers · 2025: 4 papers · 2026: 6 papers
Papers 50
- The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1002/mco2.70794↗
- Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarc… Co-author DOI 10.1038/s41591-026-04234-8↗
- GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induction of homologous recombination repair. Last author DOI 10.1136/gutjnl-2025-336227↗
- Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. Co-author DOI 10.1158/2767-9764.crc-25-0670↗
- High-Dimensional Immunophenotyping of Plasma-Derived Small Extracellular Vesicles in Pancreatic Cancer: An Exploratory Proof-of-Principle Study. Co-author DOI 10.3390/biom16070942↗
- Benchmarking the Intratumoral Microbiome in Pancreatic Ductal Adenocarcinoma: A Longitudinal Assessment of Contamination Sources and Decontamination Strategies Co-author DOI 10.64898/2026.08.24.746744↗
- KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. Co-author DOI 10.1002/1878-0261.13725↗
- Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. Co-author DOI 10.1038/s41467-024-55330-7↗
- TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. Co-author DOI 10.1126/sciadv.adu2339↗
- Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identity in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2024-334374↗
- A Novel AMPK Inhibitor Sensitizes Pancreatic Cancer Cells to Ferroptosis Induction. Co-author DOI 10.1002/advs.202307695↗
- Expression of gemcitabine metabolizing enzymes and stromal components reveal complexities of preclinical pancreatic cancer models for therapeutic testing. Co-author DOI 10.1016/j.neo.2024.101002↗
- Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarc… Co-author DOI 10.1038/s41591-024-03054-y↗
- An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. Co-author DOI 10.1053/j.gastro.2023.10.026↗
- A single-cell strategy for the identification of intronic variants related to mis-splicing in pancreatic cancer. Co-author DOI 10.1093/nargab/lqae057↗
- A Concept for Mining Transitive Sequential Patterns from Pancreatic Cancer Patient Journeys. Co-author DOI 10.3233/shti240862↗
- Prolonged survival by combination treatment with a standardized herbal extract from Japanese Kampo-medicine (Juzentaihoto) and gemcitabine in an orthotopic tra… Co-author DOI 10.3389/fonc.2024.1454291↗
- Dynamics of intestinal and intratumoral microbiome signatures in genetically engineered mice and human pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.pan.2023.07.008↗
- Neutrophil-derived complement factor P induces cytotoxicity in basal-like cells via caspase 3/7 activation in pancreatic cancer Co-author DOI 10.1101/2023.10.28.564512↗
- Discussion: Current Multimodality Treatment Options in Pancreatic Cancer in Clinical Practice - What Is the Future Impact of Molecular Biological Profiling? First author DOI 10.1159/000521847↗
- Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancreatic cancer. Co-author DOI 10.1172/jci.insight.154475↗
- TP53-Status-Dependent Oncogenic EZH2 Activity in Pancreatic Cancer. Co-author DOI 10.3390/cancers14143451↗
- TNF-α-producing macrophages determine subtype identity and prognosis via AP1 enhancer reprogramming in pancreatic cancer. Co-author DOI 10.1038/s43018-021-00258-w↗
- New Insights Into Pancreatic Cancer: Notes from a Virtual Meeting. Co-author DOI 10.1053/j.gastro.2021.04.082↗
- STIM1 Mediates Calcium-Dependent Epigenetic Reprogramming in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-20-2874↗
- Chromatin-Independent Interplay of NFATc1 and EZH2 in Pancreatic Cancer. Co-author DOI 10.3390/cells10123463↗
- Microenvironmental Determinants of Pancreatic Cancer. Co-author DOI 10.1152/physrev.00042.2019↗
- EZH2 Regulates Pancreatic Cancer Subtype Identity and Tumor Progression via Transcriptional Repression of GATA6. Co-author DOI 10.1158/0008-5472.can-20-0672↗
- [New milestone in adjuvant therapy for pancreatic cancer]. First author DOI 10.1007/s00108-019-0628-1↗
- Cytosolic 5'-nucleotidase 1A is overexpressed in pancreatic cancer and mediates gemcitabine resistance by reducing intracellular gemcitabine metabolites. Co-author DOI 10.1016/j.ebiom.2019.01.037↗
- Aberrant NFATc1 signaling counteracts TGFβ-mediated growth arrest and apoptosis induction in pancreatic cancer progression. Co-author DOI 10.1038/s41419-019-1682-2↗
- Preclinical Evaluation of 1,2-Diamino-4,5-Dibromobenzene in Genetically Engineered Mouse Models of Pancreatic Cancer. Co-author DOI 10.3390/cells8060563↗
- Stromal Features of the Primary Tumor Are Not Prognostic in Genetically Engineered Mice of Pancreatic Cancer. Co-author DOI 10.3390/cells9010058↗
- Utilizing High Resolution Ultrasound to Monitor Tumor Onset and Growth in Genetically Engineered Pancreatic Cancer Models. Co-author DOI 10.3791/56979↗
- Epigenetic treatment of pancreatic cancer: is there a therapeutic perspective on the horizon? Last author DOI 10.1136/gutjnl-2016-312539↗
- MYC in pancreatic cancer: novel mechanistic insights and their translation into therapeutic strategies. Co-author DOI 10.1038/onc.2015.216↗
- NFATc4 Regulates Sox9 Gene Expression in Acinar Cell Plasticity and Pancreatic Cancer Initiation. Co-author DOI 10.1155/2016/5272498↗
- GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. Co-author DOI 10.1158/1535-7163.mct-15-0309↗
- Current Standard and Future Perspectives in First- and Second-Line Treatment of Metastatic Pancreatic Adenocarcinoma. Co-author DOI 10.1159/000447739↗
- Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/nm.3952↗
- [Pancreatic cancer stroma: oncologist's ally or foe?]. First author DOI 10.1055/s-0034-1398955↗
- Antithetical NFATc1-Sox2 and p53-miR200 signaling networks govern pancreatic cancer cell plasticity. Last author DOI 10.15252/embj.201489574↗
- Differential activity of GSK-3 isoforms regulates NF-κB and TRAIL- or TNFα induced apoptosis in pancreatic cancer cells. Co-author DOI 10.1038/cddis.2014.102↗
- Differential activity of GSK-3 isoforms regulates NF-κB and TRAIL- or TNFα induced apoptosis in pancreatic cancer cells Co-author DOI 10.1038/cddis.2014.341↗
- nab-Paclitaxel: novel clinical and experimental evidence in pancreatic cancer. Last author DOI 10.1055/s-0034-1366002↗
- Inflammation-induced NFATc1-STAT3 transcription complex promotes pancreatic cancer initiation by KrasG12D. Last author DOI 10.1158/2159-8290.cd-13-0593↗
- WNT5A-NFAT signaling mediates resistance to apoptosis in pancreatic cancer. Co-author DOI 10.1593/neo.121312↗
- Restricted heterochromatin formation links NFATc2 repressor activity with growth promotion in pancreatic cancer. Last author DOI 10.1053/j.gastro.2011.11.001↗
- Transcriptional control of gene expression in pancreatic cancer: from sequence-specific transcription factors to nuclear architecture. Co-author DOI 10.1007/s12029-011-9254-4↗
- Disruption of a nuclear NFATc2 protein stabilization loop confers breast and pancreatic cancer growth suppression by zoledronic acid. Last author DOI 10.1074/jbc.m110.197533↗
Closest collaborators
The 25 strongest of 143 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Elisabeth Heßmann · 33 shared papers, 2014–2026 · strength 4.25
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper Expression of gemcitabine metabolizing enzymes and stromal components reveal complexities of preclinical pancreatic can… DOI 10.1016/j.neo.2024.101002↗
- Albrecht Neeße · 19 shared papers, 2014–2026 · strength 2.18
- Paper Benchmarking the Intratumoral Microbiome in Pancreatic Ductal Adenocarcinoma: A Longitudinal Assessment of Contaminatio… DOI 10.64898/2026.08.24.746744↗
- Paper Dynamics of intestinal and intratumoral microbiome signatures in genetically engineered mice and human pancreatic ducta… DOI 10.1016/j.pan.2023.07.008↗
- Paper Expression of gemcitabine metabolizing enzymes and stromal components reveal complexities of preclinical pancreatic can… DOI 10.1016/j.neo.2024.101002↗
- Shiv K. Singh · 19 shared papers, 2011–2026 · strength 2.12
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper Expression of gemcitabine metabolizing enzymes and stromal components reveal complexities of preclinical pancreatic can… DOI 10.1016/j.neo.2024.101002↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Günter Schneider · 8 shared papers, 2016–2026 · strength 1.22
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Steven A. Johnsen · 9 shared papers, 2017–2026 · strength 1.11
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Matthias Wirth · 7 shared papers, 2015–2026 · strength 1.08
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Lukas Klein · 7 shared papers, 2019–2026 · strength 1.07
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Christoph Ammer‐Herrmenau · 5 shared papers, 2023–2026 · strength 1.06
- Paper Benchmarking the Intratumoral Microbiome in Pancreatic Ductal Adenocarcinoma: A Longitudinal Assessment of Contaminatio… DOI 10.64898/2026.08.24.746744↗
- Paper Dynamics of intestinal and intratumoral microbiome signatures in genetically engineered mice and human pancreatic ducta… DOI 10.1016/j.pan.2023.07.008↗
- Paper Expression of gemcitabine metabolizing enzymes and stromal components reveal complexities of preclinical pancreatic can… DOI 10.1016/j.neo.2024.101002↗
- Maximilian Reichert · 6 shared papers, 2021–2026 · strength 0.93
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Michael B. Ghadimi · 5 shared papers, 2024–2026 · strength 0.93
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Ulrich Sax · 5 shared papers, 2024–2026 · strength 0.92
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper A Concept for Mining Transitive Sequential Patterns from Pancreatic Cancer Patient Journeys. DOI 10.3233/shti240862↗
- Alexander Otto König · 11 shared papers, 2011–2024 · strength 0.86
- Paper A Concept for Mining Transitive Sequential Patterns from Pancreatic Cancer Patient Journeys. DOI 10.3233/shti240862↗
- Paper [New milestone in adjuvant therapy for pancreatic cancer]. DOI 10.1007/s00108-019-0628-1↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Carolin Schneider · 5 shared papers, 2024–2026 · strength 0.84
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Dieter Saur · 5 shared papers, 2021–2026 · strength 0.80
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Jochen Gaedcke · 7 shared papers, 2015–2025 · strength 0.79
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Paper Chromatin-Independent Interplay of NFATc1 and EZH2 in Pancreatic Cancer. DOI 10.3390/cells10123463↗
- Laura Urbach · 5 shared papers, 2020–2025 · strength 0.73
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Neutrophil-derived complement factor P induces cytotoxicity in basal-like cells via caspase 3/7 activation in pancreati… DOI 10.1101/2023.10.28.564512↗
- Jens Thomas Siveke · 8 shared papers, 2014–2026 · strength 0.71
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Epigenetic treatment of pancreatic cancer: is there a therapeutic perspective on the horizon? DOI 10.1136/gutjnl-2016-312539↗
- Marian Grade · 4 shared papers, 2024–2026 · strength 0.71
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Lukas Krauß · 4 shared papers, 2024–2026 · strength 0.71
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Roland Michael Schmid · 5 shared papers, 2015–2026 · strength 0.65
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper The Protein Phosphatase Inhibitor LB100 Targets the Mesenchymal Lineage of Pancreatic Ductal Adenocarcinoma. DOI 10.1002/mco2.70794↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Stephan A. Hahn · 5 shared papers, 2015–2026 · strength 0.61
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Shilpa Patil · 6 shared papers, 2018–2022 · strength 0.60
- Paper TP53-Status-Dependent Oncogenic EZH2 Activity in Pancreatic Cancer. DOI 10.3390/cancers14143451↗
- Paper Chromatin-Independent Interplay of NFATc1 and EZH2 in Pancreatic Cancer. DOI 10.3390/cells10123463↗
- Paper Utilizing High Resolution Ultrasound to Monitor Tumor Onset and Growth in Genetically Engineered Pancreatic Cancer Mode… DOI 10.3791/56979↗
- Lena‐Christin Conradi · 3 shared papers, 2025–2026 · strength 0.58
- Paper Targeting PRMT5 Inhibitor-Induced Adaptation in Pancreatic Cancer with the RBM39 Degrader Indisulam. DOI 10.1158/2767-9764.crc-25-0670↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper KRAS G 12C-inhibitor-based combination therapies for pancreatic cancer: insights from drug screening. DOI 10.1002/1878-0261.13725↗
- Uday Kishore · 4 shared papers, 2021–2025 · strength 0.55
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Neutrophil-derived complement factor P induces cytotoxicity in basal-like cells via caspase 3/7 activation in pancreati… DOI 10.1101/2023.10.28.564512↗
- Paper Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancre… DOI 10.1172/jci.insight.154475↗
- Mengyu Tu · 4 shared papers, 2021–2025 · strength 0.55
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Neutrophil-derived complement factor P induces cytotoxicity in basal-like cells via caspase 3/7 activation in pancreati… DOI 10.1101/2023.10.28.564512↗
- Paper Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancre… DOI 10.1172/jci.insight.154475↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.