ResearcherUniversitätsmedizin GöttingenActive in the last 2 years
Lukas Klein
Matched on ORCID iD 0000-0003-3725-068X; OpenAlex author ID A5046866105. Sources: OpenAlex.
Their work
- Papers
- 10 (first author 4, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 52
2019: 1 paper · 2020: none · 2021: 1 paper · 2022: 2 papers · 2023: 2 papers · 2024: none · 2025: 2 papers · 2026: 2 papers
Papers 10
- Targeting addiction to HMGB2-driven transcriptional programs in pancreatic cancer. Co-author DOI 10.1016/j.celrep.2026.117649↗
- GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induction of homologous recombination repair. Co-author DOI 10.1136/gutjnl-2025-336227↗
- Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. Co-author DOI 10.1038/s41467-024-55330-7↗
- TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. Co-author DOI 10.1126/sciadv.adu2339↗
- Neutrophil-derived complement factor P induces cytotoxicity in basal-like cells via caspase 3/7 activation in pancreatic cancer Co-author DOI 10.1101/2023.10.28.564512↗
- IL-17A-producing CD8+ T cells promote PDAC via induction of inflammatory cancer-associated fibroblasts. Co-author DOI 10.1136/gutjnl-2022-327855↗
- Mechanisms of PDAC subtype heterogeneity and therapy response. First author DOI 10.1016/j.trecan.2022.08.005↗
- Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancreatic cancer. First author DOI 10.1172/jci.insight.154475↗
- TNF-α-producing macrophages determine subtype identity and prognosis via AP1 enhancer reprogramming in pancreatic cancer. First author DOI 10.1038/s43018-021-00258-w↗
- Stromal Features of the Primary Tumor Are Not Prognostic in Genetically Engineered Mice of Pancreatic Cancer. First author DOI 10.3390/cells9010058↗
Closest collaborators
The 25 strongest of 52 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Shiv K. Singh · 9 shared papers, 2021–2026 · strength 1.53
- Paper Mechanisms of PDAC subtype heterogeneity and therapy response. DOI 10.1016/j.trecan.2022.08.005↗
- Paper Targeting addiction to HMGB2-driven transcriptional programs in pancreatic cancer. DOI 10.1016/j.celrep.2026.117649↗
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Volker Ellenrieder · 7 shared papers, 2019–2026 · strength 1.07
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Elisabeth Heßmann · 5 shared papers, 2021–2026 · strength 0.82
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Mengyu Tu · 5 shared papers, 2021–2025 · strength 0.67
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Neutrophil-derived complement factor P induces cytotoxicity in basal-like cells via caspase 3/7 activation in pancreati… DOI 10.1101/2023.10.28.564512↗
- Paper IL-17A-producing CD8+ T cells promote PDAC via induction of inflammatory cancer-associated fibroblasts. DOI 10.1136/gutjnl-2022-327855↗
- Laura Urbach · 4 shared papers, 2021–2025 · strength 0.64
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Neutrophil-derived complement factor P induces cytotoxicity in basal-like cells via caspase 3/7 activation in pancreati… DOI 10.1101/2023.10.28.564512↗
- Elisa Espinet Espinet · 4 shared papers, 2021–2025 · strength 0.62
- Paper Mechanisms of PDAC subtype heterogeneity and therapy response. DOI 10.1016/j.trecan.2022.08.005↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancre… DOI 10.1172/jci.insight.154475↗
- Uday Kishore · 4 shared papers, 2021–2025 · strength 0.55
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Neutrophil-derived complement factor P induces cytotoxicity in basal-like cells via caspase 3/7 activation in pancreati… DOI 10.1101/2023.10.28.564512↗
- Paper Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancre… DOI 10.1172/jci.insight.154475↗
- Argyris Papantonis · 3 shared papers, 2021–2026 · strength 0.50
- Paper Targeting addiction to HMGB2-driven transcriptional programs in pancreatic cancer. DOI 10.1016/j.celrep.2026.117649↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper TNF-α-producing macrophages determine subtype identity and prognosis via AP1 enhancer reprogramming in pancreatic cance… DOI 10.1038/s43018-021-00258-w↗
- Lena‐Christin Conradi · 2 shared papers, 2026 · strength 0.42
- Ulrich Sax · 2 shared papers, 2025–2026 · strength 0.42
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Rebecca Diya Samuel · 2 shared papers, 2025 · strength 0.40
- Paper TP53 missense-specific transcriptional plasticity drives resistance against cell cycle inhibitors in pancreatic cancer. DOI 10.1126/sciadv.adu2339↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Andreas Trumpp · 3 shared papers, 2021–2025 · strength 0.40
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancre… DOI 10.1172/jci.insight.154475↗
- Paper TNF-α-producing macrophages determine subtype identity and prognosis via AP1 enhancer reprogramming in pancreatic cance… DOI 10.1038/s43018-021-00258-w↗
- Muhammad Umair Latif · 2 shared papers, 2025–2026 · strength 0.39
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Albrecht Neeße · 3 shared papers, 2019–2022 · strength 0.32
- Paper Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancre… DOI 10.1172/jci.insight.154475↗
- Paper Stromal Features of the Primary Tumor Are Not Prognostic in Genetically Engineered Mice of Pancreatic Cancer. DOI 10.3390/cells9010058↗
- Paper TNF-α-producing macrophages determine subtype identity and prognosis via AP1 enhancer reprogramming in pancreatic cance… DOI 10.1038/s43018-021-00258-w↗
- Adi Danieli‐Mackay · 2 shared papers, 2021–2026 · strength 0.31
- Niklas Krebs · 2 shared papers, 2022–2025 · strength 0.30
- Paper Spatial tumor immune heterogeneity facilitates subtype co-existence and therapy response in pancreatic cancer. DOI 10.1038/s41467-024-55330-7↗
- Paper Axon guidance receptor ROBO3 modulates subtype identity and prognosis via AXL-associated inflammatory network in pancre… DOI 10.1172/jci.insight.154475↗
- Ellen Puré · 1 shared paper, 2022 · strength 0.22
- Gabriela Salinas Riester · 1 shared paper, 2026 · strength 0.22
- Tiago De Oliveira · 1 shared paper, 2026 · strength 0.22
- Emre Taylan Duman · 1 shared paper, 2026 · strength 0.22
- Matthias Dobbelstein · 1 shared paper, 2026 · strength 0.22
- Michael B. Ghadimi · 1 shared paper, 2025 · strength 0.22
- Ramona Schulz‐Heddergott · 1 shared paper, 2025 · strength 0.22
- Nelson Dusetti · 1 shared paper, 2025 · strength 0.22
- Steven A. Johnsen · 1 shared paper, 2026 · strength 0.20
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.