ResearcherMayo ClinicActive in the last 2 years
Steven A. Johnsen
Matched on ORCID iD 0000-0003-1198-5805; OpenAlex author ID A5059210191. Sources: OpenAlex.
Their work
- Papers
- 24 (first author 5, last author 7)
- Trials
- 0
- Grants
- 0
- Labs led
- 2
- Collaborators
- 98
2016: 1 paper · 2017: 2 papers · 2018: 2 papers · 2019: 2 papers · 2020: 2 papers · 2021: 4 papers · 2022: none · 2023: 2 papers · 2024: 2 papers · 2025: 3 papers · 2026: 4 papers
Papers 24
- Epigenetic context defines the transcriptional activity of canonical and noncanonical NF-κB signaling in pancreatic cancer. Last author DOI 10.1038/s41420-026-03019-9↗
- Epigenetic and transcriptional control of classical and basal-like cell states in pancreatic ductal adenocarcinoma. First author DOI 10.1093/gastro/goag028↗
- KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. Last author DOI 10.1126/sciadv.aea2106↗
- GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induction of homologous recombination repair. Co-author DOI 10.1136/gutjnl-2025-336227↗
- Patient-derived tumor organoids highlight the potential of precision medicine in managing pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.35443↗
- Therapeutic targeting of FOSL1 and RELA-dependent transcriptional mechanisms to suppress pancreatic cancer metastasis. Last author DOI 10.1038/s41419-025-07810-x↗
- Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identity in pancreatic cancer. Last author DOI 10.1136/gutjnl-2024-334374↗
- An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. Co-author DOI 10.1053/j.gastro.2023.10.026↗
- Pancreatic cancer acquires resistance to MAPK pathway inhibition by clonal expansion and adaptive DNA hypermethylation. Co-author DOI 10.1186/s13148-024-01623-z↗
- Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. Last author DOI 10.1136/gutjnl-2022-328154↗
- Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1541-7786.mcr-22-0916↗
- New Insights Into Pancreatic Cancer: Notes from a Virtual Meeting. Co-author DOI 10.1053/j.gastro.2021.04.082↗
- STIM1 Mediates Calcium-Dependent Epigenetic Reprogramming in Pancreatic Cancer. Last author DOI 10.1158/0008-5472.can-20-2874↗
- Telomerase and Pluripotency Factors Jointly Regulate Stemness in Pancreatic Cancer Stem Cells. Co-author DOI 10.3390/cancers13133145↗
- Store-Operated Calcium Entry: Shaping the Transcriptional and Epigenetic Landscape in Pancreatic Cancer. First author DOI 10.3390/cells10050966↗
- Characterization of a dual BET/HDAC inhibitor for treatment of pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.33137↗
- EZH2 Regulates Pancreatic Cancer Subtype Identity and Tumor Progression via Transcriptional Repression of GATA6. Co-author DOI 10.1158/0008-5472.can-20-0672↗
- Cytosolic 5'-nucleotidase 1A is overexpressed in pancreatic cancer and mediates gemcitabine resistance by reducing intracellular gemcitabine metabolites. Co-author DOI 10.1016/j.ebiom.2019.01.037↗
- Aberrant NFATc1 signaling counteracts TGFβ-mediated growth arrest and apoptosis induction in pancreatic cancer progression. Co-author DOI 10.1038/s41419-019-1682-2↗
- DeltaNp63-dependent super enhancers define molecular identity in pancreatic cancer by an interconnected transcription factor network. First author DOI 10.1073/pnas.1812915116↗
- Epigenetic Targeting of Aberrant Transcriptional Modulation in Pancreatic Cancer First author DOI 10.3390/epigenomes2020008↗
- Histone deacetylase class-I inhibition promotes epithelial gene expression in pancreatic cancer cells in a BRD4- and MYC-dependent manner. Last author DOI 10.1093/nar/gkx212↗
- Epigenetic treatment of pancreatic cancer: is there a therapeutic perspective on the horizon? First author DOI 10.1136/gutjnl-2016-312539↗
- Relevance of Sp Binding Site Polymorphism in WWOX for Treatment Outcome in Pancreatic Cancer. Co-author DOI 10.1093/jnci/djv387↗
Closest collaborators
The 25 strongest of 98 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Elisabeth Heßmann · 12 shared papers, 2017–2026 · strength 1.44
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Feda H. Hamdan · 10 shared papers, 2018–2024 · strength 1.34
- Paper Store-Operated Calcium Entry: Shaping the Transcriptional and Epigenetic Landscape in Pancreatic Cancer. DOI 10.3390/cells10050966↗
- Paper DeltaNp63-dependent super enhancers define molecular identity in pancreatic cancer by an interconnected transcription f… DOI 10.1073/pnas.1812915116↗
- Paper Epigenetic Targeting of Aberrant Transcriptional Modulation in Pancreatic Cancer DOI 10.3390/epigenomes2020008↗
- Amro M. Abdelrahman · 6 shared papers, 2023–2026 · strength 1.20
- Paper Therapeutic targeting of FOSL1 and RELA-dependent transcriptional mechanisms to suppress pancreatic cancer metastasis. DOI 10.1038/s41419-025-07810-x↗
- Paper Epigenetic context defines the transcriptional activity of canonical and noncanonical NF-κB signaling in pancreatic can… DOI 10.1038/s41420-026-03019-9↗
- Paper KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. DOI 10.1126/sciadv.aea2106↗
- Mark Joseph Truty · 6 shared papers, 2023–2026 · strength 1.20
- Paper Therapeutic targeting of FOSL1 and RELA-dependent transcriptional mechanisms to suppress pancreatic cancer metastasis. DOI 10.1038/s41419-025-07810-x↗
- Paper Epigenetic context defines the transcriptional activity of canonical and noncanonical NF-κB signaling in pancreatic can… DOI 10.1038/s41420-026-03019-9↗
- Paper KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. DOI 10.1126/sciadv.aea2106↗
- Zeynab Najafova · 8 shared papers, 2017–2026 · strength 1.17
- Paper KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. DOI 10.1126/sciadv.aea2106↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Volker Ellenrieder · 9 shared papers, 2017–2026 · strength 1.11
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Christina Parassiadis · 2 shared papers, 2025–2026 · strength 0.82
- Paper Epigenetic and transcriptional control of classical and basal-like cell states in pancreatic ductal adenocarcinoma. DOI 10.1093/gastro/goag028↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Ana P. Kutschat · 5 shared papers, 2021–2025 · strength 0.79
- Paper Store-Operated Calcium Entry: Shaping the Transcriptional and Epigenetic Landscape in Pancreatic Cancer. DOI 10.3390/cells10050966↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Jochen Gaedcke · 7 shared papers, 2016–2025 · strength 0.78
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Jens Thomas Siveke · 7 shared papers, 2017–2024 · strength 0.77
- Paper Pancreatic cancer acquires resistance to MAPK pathway inhibition by clonal expansion and adaptive DNA hypermethylation. DOI 10.1186/s13148-024-01623-z↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Joana E. Aggrey-Fynn · 4 shared papers, 2023–2026 · strength 0.77
- Paper Therapeutic targeting of FOSL1 and RELA-dependent transcriptional mechanisms to suppress pancreatic cancer metastasis. DOI 10.1038/s41419-025-07810-x↗
- Paper Epigenetic context defines the transcriptional activity of canonical and noncanonical NF-κB signaling in pancreatic can… DOI 10.1038/s41420-026-03019-9↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Xin Wang · 5 shared papers, 2020–2025 · strength 0.65
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Thomas L. Ekstrom · 3 shared papers, 2023–2026 · strength 0.56
- Paper KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. DOI 10.1126/sciadv.aea2106↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Shiv K. Singh · 4 shared papers, 2020–2026 · strength 0.51
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Paper New Insights Into Pancreatic Cancer: Notes from a Virtual Meeting. DOI 10.1053/j.gastro.2021.04.082↗
- Stephan A. Hahn · 4 shared papers, 2020–2024 · strength 0.49
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Günter Schneider · 3 shared papers, 2021–2026 · strength 0.43
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Paper New Insights Into Pancreatic Cancer: Notes from a Virtual Meeting. DOI 10.1053/j.gastro.2021.04.082↗
- Ulrich Sax · 2 shared papers, 2024–2026 · strength 0.34
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Albrecht Neeße · 3 shared papers, 2019–2024 · strength 0.29
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Paper EZH2 Regulates Pancreatic Cancer Subtype Identity and Tumor Progression via Transcriptional Repression of GATA6. DOI 10.1158/0008-5472.can-20-0672↗
- Paper Cytosolic 5'-nucleotidase 1A is overexpressed in pancreatic cancer and mediates gemcitabine resistance by reducing intr… DOI 10.1016/j.ebiom.2019.01.037↗
- Jennifer A. Yonkus · 2 shared papers, 2023 · strength 0.27
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Rondell Patrell Graham · 2 shared papers, 2023 · strength 0.27
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Roberto Alva‐Ruiz · 2 shared papers, 2023 · strength 0.27
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Marie C. Hasselluhn · 2 shared papers, 2019–2024 · strength 0.26
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Paper Aberrant NFATc1 signaling counteracts TGFβ-mediated growth arrest and apoptosis induction in pancreatic cancer progress… DOI 10.1038/s41419-019-1682-2↗
- Sven‐Thorsten Liffers · 2 shared papers, 2020–2024 · strength 0.26
- Marija Trajkovic‐Arsic · 2 shared papers, 2020–2024 · strength 0.26
- Thilo Hackert · 2 shared papers, 2016–2025 · strength 0.25
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.