ResearcherUniversitätsmedizin GöttingenActive in the last 2 years
Zeynab Najafova
Matched on ORCID iD 0000-0003-4403-5566; OpenAlex author ID A5026778626. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 0, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 36
2017: 1 paper · 2018: none · 2019: none · 2020: none · 2021: 2 papers · 2022: none · 2023: 2 papers · 2024: none · 2025: 1 paper · 2026: 2 papers
Papers 8
- KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. Co-author DOI 10.1126/sciadv.aea2106↗
- GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induction of homologous recombination repair. Co-author DOI 10.1136/gutjnl-2025-336227↗
- Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identity in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2024-334374↗
- Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2022-328154↗
- Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive Pancreatic Ductal Adenocarcinoma. Last author DOI 10.1158/1541-7786.mcr-22-0916↗
- STIM1 Mediates Calcium-Dependent Epigenetic Reprogramming in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-20-2874↗
- Telomerase and Pluripotency Factors Jointly Regulate Stemness in Pancreatic Cancer Stem Cells. Co-author DOI 10.3390/cancers13133145↗
- Histone deacetylase class-I inhibition promotes epithelial gene expression in pancreatic cancer cells in a BRD4- and MYC-dependent manner. Co-author DOI 10.1093/nar/gkx212↗
Closest collaborators
The 25 strongest of 36 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Steven A. Johnsen · 8 shared papers, 2017–2026 · strength 1.17
- Paper KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. DOI 10.1126/sciadv.aea2106↗
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Elisabeth Heßmann · 6 shared papers, 2017–2026 · strength 0.83
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Amro M. Abdelrahman · 4 shared papers, 2023–2026 · strength 0.69
- Paper KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. DOI 10.1126/sciadv.aea2106↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Mark Joseph Truty · 4 shared papers, 2023–2026 · strength 0.69
- Paper KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. DOI 10.1126/sciadv.aea2106↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Xin Wang · 4 shared papers, 2021–2025 · strength 0.57
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Jochen Gaedcke · 4 shared papers, 2021–2025 · strength 0.57
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Ana P. Kutschat · 4 shared papers, 2021–2025 · strength 0.57
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Thomas L. Ekstrom · 3 shared papers, 2023–2026 · strength 0.56
- Paper KLF5 controls subtype-independent highly interactive enhancers in pancreatic cancer to regulate cell survival. DOI 10.1126/sciadv.aea2106↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Volker Ellenrieder · 3 shared papers, 2021–2026 · strength 0.51
- Paper GSK3βhigh/NFATc1high subtype targeting overcomes therapy resistance in pancreatic cancer through transcriptional induct… DOI 10.1136/gutjnl-2025-336227↗
- Paper Glucocorticoid receptor suppresses GATA6-mediated RNA polymerase II pause release to modulate classical subtype identit… DOI 10.1136/gutjnl-2024-334374↗
- Paper STIM1 Mediates Calcium-Dependent Epigenetic Reprogramming in Pancreatic Cancer. DOI 10.1158/0008-5472.can-20-2874↗
- Feda H. Hamdan · 3 shared papers, 2021–2023 · strength 0.38
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Paper STIM1 Mediates Calcium-Dependent Epigenetic Reprogramming in Pancreatic Cancer. DOI 10.1158/0008-5472.can-20-2874↗
- Jens Thomas Siveke · 3 shared papers, 2017–2023 · strength 0.33
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Paper Histone deacetylase class-I inhibition promotes epithelial gene expression in pancreatic cancer cells in a BRD4- and MY… DOI 10.1093/nar/gkx212↗
- Jennifer A. Yonkus · 2 shared papers, 2023 · strength 0.27
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Joana E. Aggrey-Fynn · 2 shared papers, 2023 · strength 0.27
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Rondell Patrell Graham · 2 shared papers, 2023 · strength 0.27
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Roberto Alva‐Ruiz · 2 shared papers, 2023 · strength 0.27
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Stephan A. Hahn · 2 shared papers, 2023 · strength 0.27
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Patrick Christian Hermann · 2 shared papers, 2021–2023 · strength 0.23
- Shiv K. Singh · 1 shared paper, 2026 · strength 0.20
- Muhammad Umair Latif · 1 shared paper, 2026 · strength 0.20
- Ulrich Sax · 1 shared paper, 2026 · strength 0.20
- Lena‐Christin Conradi · 1 shared paper, 2026 · strength 0.20
- Lukas Klein · 1 shared paper, 2026 · strength 0.20
- Oliver Holger Krämer · 1 shared paper, 2026 · strength 0.20
- Günter Schneider · 1 shared paper, 2026 · strength 0.20
- Christina Parassiadis · 1 shared paper, 2025 · strength 0.19
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.