ResearcherUniversitätsklinikum Knappschaftskrankenhaus BochumActive in the last 2 years
Stephan A. Hahn
Matched on ORCID iD 0000-0003-0855-9741; OpenAlex author ID A5000633602. Sources: OpenAlex.
Their work
- Papers
- 28 (first author 2, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 138
2012: 2 papers · 2013: 3 papers · 2014: 4 papers · 2015: 2 papers · 2016: none · 2017: 1 paper · 2018: none · 2019: none · 2020: 2 papers · 2021: 3 papers · 2022: 2 papers · 2023: 4 papers · 2024: 3 papers · 2025: 1 paper · 2026: 1 paper
Papers 28
- Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarc… Co-author DOI 10.1038/s41591-026-04234-8↗
- PLK1 blockade enhances the anti-tumor effect of MAPK inhibition in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.celrep.2025.115541↗
- Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarc… Co-author DOI 10.1038/s41591-024-03054-y↗
- An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. Co-author DOI 10.1053/j.gastro.2023.10.026↗
- Maintenance Therapy for Pancreatic Cancer, a New Approach Based on the Synergy between the Novel Agent GP-2250 (Misetionamide) and Gemcitabine. Co-author DOI 10.3390/cancers16142612↗
- GP-2250, a novel anticancer agent, inhibits the energy metabolism, activates AMP-Kinase and impairs the NF-kB pathway in pancreatic cancer cells. Co-author DOI 10.1111/jcmm.17825↗
- Coinhibition of topoisomerase 1 and BRD4-mediated pause release selectively kills pancreatic cancer via readthrough transcription. Co-author DOI 10.1126/sciadv.adg5109↗
- Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2022-328154↗
- Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1158/1541-7786.mcr-22-0916↗
- De novo expression of gastrokines in pancreatic precursor lesions impede the development of pancreatic cancer. Co-author DOI 10.1038/s41388-022-02182-4↗
- Functional noninvasive detection of glycolytic pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s40170-022-00298-5↗
- TNF-α-producing macrophages determine subtype identity and prognosis via AP1 enhancer reprogramming in pancreatic cancer. Co-author DOI 10.1038/s43018-021-00258-w↗
- Functional metabolic phenotyping of human pancreatic ductal adenocarcinoma Co-author DOI 10.1101/2021.07.23.452145↗
- Synergistic targeting and resistance to PARP inhibition in DNA damage repair-deficient pancreatic cancer. Co-author DOI 10.1136/gutjnl-2019-319970↗
- Characterization of a dual BET/HDAC inhibitor for treatment of pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.33137↗
- TFEB-mediated lysosomal biogenesis and lysosomal drug sequestration confer resistance to MEK inhibition in pancreatic cancer. Co-author DOI 10.1038/s41420-020-0246-7↗
- A genetic roadmap of pancreatic cancer: still evolving. First author DOI 10.1136/gutjnl-2016-313317↗
- Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/nm.3952↗
- The miR-17-92 cluster counteracts quiescence and chemoresistance in a distinct subpopulation of pancreatic cancer stem cells. Co-author DOI 10.1136/gutjnl-2014-308470↗
- A microRNA-based test improves endoscopic ultrasound-guided cytologic diagnosis of pancreatic cancer. Co-author DOI 10.1016/j.cgh.2014.02.038↗
- Induction of pancreatic cancer cell migration by an autocrine epidermal growth factor receptor activation. First author DOI 10.1016/j.yexcr.2014.04.022↗
- New insights in the composition of extracellular vesicles from pancreatic cancer cells: implications for biomarkers and functions. Co-author DOI 10.1186/s12953-014-0050-5↗
- A soluble form of the giant cadherin Fat1 is released from pancreatic cancer cells by ADAM10 mediated ectodomain shedding. Co-author DOI 10.1371/journal.pone.0090461↗
- Norepinephrine inhibits the migratory activity of pancreatic cancer cells. Co-author DOI 10.1016/j.yexcr.2013.04.015↗
- Claudin-4-targeted optical imaging detects pancreatic cancer and its precursor lesions. Co-author DOI 10.1136/gutjnl-2012-302577↗
- Multimodal Treatment Eliminates Cancer Stem Cells and Leads to Long-Term Survival in Primary Human Pancreatic Cancer Tissue Xenografts. Co-author DOI 10.1371/journal.pone.0066371↗
- Global microRNA expression profiling of microdissected tissues identifies miR-135b as a novel biomarker for pancreatic ductal adenocarcinoma. Last author DOI 10.1002/ijc.26466↗
- Lack of CCR7 expression is rate limiting for lymphatic spread of pancreatic ductal adenocarcinoma. Co-author DOI 10.1002/ijc.26502↗
Closest collaborators
The 25 strongest of 138 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jens Thomas Siveke · 11 shared papers, 2015–2026 · strength 1.36
- Paper PLK1 blockade enhances the anti-tumor effect of MAPK inhibition in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.115541↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Marija Trajkovic‐Arsic · 8 shared papers, 2015–2026 · strength 0.96
- Paper PLK1 blockade enhances the anti-tumor effect of MAPK inhibition in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.115541↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Elisabeth Heßmann · 8 shared papers, 2015–2026 · strength 0.96
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Smiths Lueong · 5 shared papers, 2020–2025 · strength 0.63
- Paper PLK1 blockade enhances the anti-tumor effect of MAPK inhibition in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.115541↗
- Paper Coinhibition of topoisomerase 1 and BRD4-mediated pause release selectively kills pancreatic cancer via readthrough tra… DOI 10.1126/sciadv.adg5109↗
- Paper Functional noninvasive detection of glycolytic pancreatic ductal adenocarcinoma. DOI 10.1186/s40170-022-00298-5↗
- Volker Ellenrieder · 5 shared papers, 2015–2026 · strength 0.61
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Waldemar H. Uhl · 5 shared papers, 2012–2024 · strength 0.60
- Paper Maintenance Therapy for Pancreatic Cancer, a New Approach Based on the Synergy between the Novel Agent GP-2250 (Misetio… DOI 10.3390/cancers16142612↗
- Paper GP-2250, a novel anticancer agent, inhibits the energy metabolism, activates AMP-Kinase and impairs the NF-kB pathway i… DOI 10.1111/jcmm.17825↗
- Paper Functional noninvasive detection of glycolytic pancreatic ductal adenocarcinoma. DOI 10.1186/s40170-022-00298-5↗
- Irina Heid · 5 shared papers, 2015–2026 · strength 0.57
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Functional noninvasive detection of glycolytic pancreatic ductal adenocarcinoma. DOI 10.1186/s40170-022-00298-5↗
- Rickmer Braren · 5 shared papers, 2015–2026 · strength 0.57
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Functional noninvasive detection of glycolytic pancreatic ductal adenocarcinoma. DOI 10.1186/s40170-022-00298-5↗
- Steven A. Johnsen · 4 shared papers, 2020–2024 · strength 0.49
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Feda H. Hamdan · 4 shared papers, 2020–2024 · strength 0.49
- Paper Identification of a ΔNp63-Dependent Basal-Like A Subtype-Specific Transcribed Enhancer Program (B-STEP) in Aggressive P… DOI 10.1158/1541-7786.mcr-22-0916↗
- Paper An NFATc1/SMAD3/cJUN Complex Restricted to SMAD4-Deficient Pancreatic Cancer Guides Rational Therapies. DOI 10.1053/j.gastro.2023.10.026↗
- Paper Interactive enhancer hubs (iHUBs) mediate transcriptional reprogramming and adaptive resistance in pancreatic cancer. DOI 10.1136/gutjnl-2022-328154↗
- Bence Sipos · 5 shared papers, 2012–2026 · strength 0.43
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancre… DOI 10.1038/nm.3952↗
- Phyllis F. Cheung · 4 shared papers, 2020–2023 · strength 0.42
- Paper Coinhibition of topoisomerase 1 and BRD4-mediated pause release selectively kills pancreatic cancer via readthrough tra… DOI 10.1126/sciadv.adg5109↗
- Paper Functional noninvasive detection of glycolytic pancreatic ductal adenocarcinoma. DOI 10.1186/s40170-022-00298-5↗
- Paper Characterization of a dual BET/HDAC inhibitor for treatment of pancreatic ductal adenocarcinoma. DOI 10.1002/ijc.33137↗
- Laura Godfrey · 3 shared papers, 2021–2025 · strength 0.40
- Paper PLK1 blockade enhances the anti-tumor effect of MAPK inhibition in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.115541↗
- Paper Functional noninvasive detection of glycolytic pancreatic ductal adenocarcinoma. DOI 10.1186/s40170-022-00298-5↗
- Paper Functional metabolic phenotyping of human pancreatic ductal adenocarcinoma DOI 10.1101/2021.07.23.452145↗
- Andrea Tannapfel · 5 shared papers, 2012–2023 · strength 0.39
- Paper Coinhibition of topoisomerase 1 and BRD4-mediated pause release selectively kills pancreatic cancer via readthrough tra… DOI 10.1126/sciadv.adg5109↗
- Paper Functional noninvasive detection of glycolytic pancreatic ductal adenocarcinoma. DOI 10.1186/s40170-022-00298-5↗
- Paper Functional metabolic phenotyping of human pancreatic ductal adenocarcinoma DOI 10.1101/2021.07.23.452145↗
- Marie Buchholz · 2 shared papers, 2023–2024 · strength 0.38
- Paper Maintenance Therapy for Pancreatic Cancer, a New Approach Based on the Synergy between the Novel Agent GP-2250 (Misetio… DOI 10.3390/cancers16142612↗
- Paper GP-2250, a novel anticancer agent, inhibits the energy metabolism, activates AMP-Kinase and impairs the NF-kB pathway i… DOI 10.1111/jcmm.17825↗
- Britta Majchrzak‐Stiller · 2 shared papers, 2023–2024 · strength 0.38
- Paper Maintenance Therapy for Pancreatic Cancer, a New Approach Based on the Synergy between the Novel Agent GP-2250 (Misetio… DOI 10.3390/cancers16142612↗
- Paper GP-2250, a novel anticancer agent, inhibits the energy metabolism, activates AMP-Kinase and impairs the NF-kB pathway i… DOI 10.1111/jcmm.17825↗
- Chris Braumann · 2 shared papers, 2023–2024 · strength 0.38
- Paper Maintenance Therapy for Pancreatic Cancer, a New Approach Based on the Synergy between the Novel Agent GP-2250 (Misetio… DOI 10.3390/cancers16142612↗
- Paper GP-2250, a novel anticancer agent, inhibits the energy metabolism, activates AMP-Kinase and impairs the NF-kB pathway i… DOI 10.1111/jcmm.17825↗
- Philipp Höhn · 2 shared papers, 2023–2024 · strength 0.38
- Paper Maintenance Therapy for Pancreatic Cancer, a New Approach Based on the Synergy between the Novel Agent GP-2250 (Misetio… DOI 10.3390/cancers16142612↗
- Paper GP-2250, a novel anticancer agent, inhibits the energy metabolism, activates AMP-Kinase and impairs the NF-kB pathway i… DOI 10.1111/jcmm.17825↗
- Stephano S. Mello · 3 shared papers, 2015–2026 · strength 0.37
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancre… DOI 10.1038/nm.3952↗
- Mert Erkan · 3 shared papers, 2015–2026 · strength 0.37
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancre… DOI 10.1038/nm.3952↗
- Julien Sage · 3 shared papers, 2015–2026 · strength 0.37
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancre… DOI 10.1038/nm.3952↗
- Alexander Herner · 3 shared papers, 2015–2026 · strength 0.37
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancre… DOI 10.1038/nm.3952↗
- Roland Michael Schmid · 3 shared papers, 2015–2026 · strength 0.37
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancre… DOI 10.1038/nm.3952↗
- Pawel Karol Mazur · 3 shared papers, 2015–2026 · strength 0.37
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancre… DOI 10.1038/nm.3952↗
- Iréne Esposito · 3 shared papers, 2015–2026 · strength 0.37
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-026-04234-8↗
- Paper Author Correction: Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based… DOI 10.1038/s41591-024-03054-y↗
- Paper Combined inhibition of BET family proteins and histone deacetylases as a potential epigenetics-based therapy for pancre… DOI 10.1038/nm.3952↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.