ResearcherTUM KlinikumNo recent work listed
Zonera Hassan
Matched on ORCID iD 0000-0002-7981-8328; OpenAlex author ID A5072922624. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 5, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 50
2017: 2 papers · 2018: 2 papers · 2019: none · 2020: 2 papers · 2021: 1 paper · 2022: 5 papers
Papers 12
- Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. First author DOI 10.1002/cac2.12280↗
- AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. Co-author DOI 10.1007/s00018-022-04638-y↗
- HDAC2 Facilitates Pancreatic Cancer Metastasis. Co-author DOI 10.1158/0008-5472.can-20-3209↗
- Epigenetic drug screening defines a PRMT5 inhibitor-sensitive pancreatic cancer subtype. Co-author DOI 10.1172/jci.insight.151353↗
- RelA Is an Essential Target for Enhancing Cellular Responses to the DNA Repair/Ref-1 Redox Signaling Protein and Restoring Perturbated Cellular Redox Homeostas… Co-author DOI 10.3389/fonc.2022.826617↗
- The SUMO pathway in pancreatic cancer: insights and inhibition. First author DOI 10.1038/s41416-020-01119-6↗
- Targeting the ubiquitin-proteasome system in a pancreatic cancer subtype with hyperactive MYC. First author DOI 10.1002/1878-0261.12835↗
- SUMO pathway inhibition targets an aggressive pancreatic cancer subtype. First author DOI 10.1136/gutjnl-2018-317856↗
- MTOR inhibitor-based combination therapies for pancreatic cancer. Co-author DOI 10.1038/bjc.2017.421↗
- Evolutionary routes and KRAS dosage define pancreatic cancer phenotypes. Co-author DOI 10.1038/nature25459↗
- Role of CCL20 mediated immune cell recruitment in NF-κB mediated TRAIL resistance of pancreatic cancer. Co-author DOI 10.1016/j.bbamcr.2017.02.005↗
- HDAC1 and HDAC2 integrate the expression of p53 mutants in pancreatic cancer. First author DOI 10.1038/onc.2016.344↗
Closest collaborators
The 25 strongest of 50 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Günter Schneider · 11 shared papers, 2017–2022 · strength 1.05
- Paper The SUMO pathway in pancreatic cancer: insights and inhibition. DOI 10.1038/s41416-020-01119-6↗
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Christian Schneeweis · 8 shared papers, 2018–2022 · strength 0.93
- Paper The SUMO pathway in pancreatic cancer: insights and inhibition. DOI 10.1038/s41416-020-01119-6↗
- Paper RelA Is an Essential Target for Enhancing Cellular Responses to the DNA Repair/Ref-1 Redox Signaling Protein and Restor… DOI 10.3389/fonc.2022.826617↗
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Roland Rad · 9 shared papers, 2017–2022 · strength 0.82
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Dieter Saur · 9 shared papers, 2017–2022 · strength 0.82
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Maximilian Reichert · 8 shared papers, 2017–2022 · strength 0.77
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Matthias Wirth · 8 shared papers, 2017–2022 · strength 0.77
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Oliver Holger Krämer · 6 shared papers, 2017–2022 · strength 0.59
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Lukas Krauß · 4 shared papers, 2022 · strength 0.48
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Carolin Schneider · 4 shared papers, 2022 · strength 0.48
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Roland Michael Schmid · 5 shared papers, 2017–2022 · strength 0.39
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Paper Epigenetic drug screening defines a PRMT5 inhibitor-sensitive pancreatic cancer subtype. DOI 10.1172/jci.insight.151353↗
- Paper MTOR inhibitor-based combination therapies for pancreatic cancer. DOI 10.1038/bjc.2017.421↗
- Ulrich Bernd Keller · 3 shared papers, 2020–2021 · strength 0.35
- Paper The SUMO pathway in pancreatic cancer: insights and inhibition. DOI 10.1038/s41416-020-01119-6↗
- Paper Targeting the ubiquitin-proteasome system in a pancreatic cancer subtype with hyperactive MYC. DOI 10.1002/1878-0261.12835↗
- Paper SUMO pathway inhibition targets an aggressive pancreatic cancer subtype. DOI 10.1136/gutjnl-2018-317856↗
- Katharina Lankes · 3 shared papers, 2020–2022 · strength 0.32
- Paper HDAC2 Facilitates Pancreatic Cancer Metastasis. DOI 10.1158/0008-5472.can-20-3209↗
- Paper Epigenetic drug screening defines a PRMT5 inhibitor-sensitive pancreatic cancer subtype. DOI 10.1172/jci.insight.151353↗
- Paper Targeting the ubiquitin-proteasome system in a pancreatic cancer subtype with hyperactive MYC. DOI 10.1002/1878-0261.12835↗
- Felix Orben · 3 shared papers, 2020–2022 · strength 0.32
- Paper Indirect targeting of MYC sensitizes pancreatic cancer cells to mechanistic target of rapamycin (mTOR) inhibition. DOI 10.1002/cac2.12280↗
- Paper Epigenetic drug screening defines a PRMT5 inhibitor-sensitive pancreatic cancer subtype. DOI 10.1172/jci.insight.151353↗
- Paper SUMO pathway inhibition targets an aggressive pancreatic cancer subtype. DOI 10.1136/gutjnl-2018-317856↗
- Alexander Arlt · 4 shared papers, 2017–2022 · strength 0.30
- Paper AP1/Fra1 confers resistance to MAPK cascade inhibition in pancreatic cancer. DOI 10.1007/s00018-022-04638-y↗
- Paper MTOR inhibitor-based combination therapies for pancreatic cancer. DOI 10.1038/bjc.2017.421↗
- Paper Role of CCL20 mediated immune cell recruitment in NF-κB mediated TRAIL resistance of pancreatic cancer. DOI 10.1016/j.bbamcr.2017.02.005↗
- Wilko Weichert · 3 shared papers, 2018–2020 · strength 0.19
- Paper SUMO pathway inhibition targets an aggressive pancreatic cancer subtype. DOI 10.1136/gutjnl-2018-317856↗
- Paper MTOR inhibitor-based combination therapies for pancreatic cancer. DOI 10.1038/bjc.2017.421↗
- Paper Evolutionary routes and KRAS dosage define pancreatic cancer phenotypes. DOI 10.1038/nature25459↗
- Julia V. Mayerle · 2 shared papers, 2018–2022 · strength 0.16
- Melissa L. Fishel · 1 shared paper, 2022 · strength 0.15
- Mahmut Mijit · 1 shared paper, 2022 · strength 0.15
- Silpa Gampala · 1 shared paper, 2022 · strength 0.15
- Mark R. Kelley · 1 shared paper, 2022 · strength 0.15
- Zahra Dantes · 2 shared papers, 2018–2020 · strength 0.14
- Sandra Diersch · 1 shared paper, 2022 · strength 0.12
- Chiara Falcomatà · 1 shared paper, 2022 · strength 0.12
- Marian Grade · 1 shared paper, 2022 · strength 0.12
- Daniele Lucarelli · 1 shared paper, 2022 · strength 0.12
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.