ResearcherChinese Academy of Medical Sciences & Peking Union Medical CollegeNo recent work listed
Hong Shu
Matched on ORCID iD 0000-0002-2844-8347; OpenAlex author ID A5100769853. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 0, last author 6)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 19
2012: 1 paper · 2013: 2 papers · 2014: 5 papers · 2015: none · 2016: none · 2017: 1 paper
Papers 9
- Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14. Co-author DOI 10.1158/0008-5472.can-16-2339↗
- N-wasp in pancreatic ductal adenocarcinoma: associations with perineural invasion and poor prognosis. Last author DOI 10.1007/s00268-014-2500-8↗
- Alteration of the intrinsic apoptosis pathway is involved in Notch-induced chemoresistance to gemcitabine in pancreatic cancer. Last author DOI 10.1016/j.arcmed.2013.10.001↗
- Prognostic significance of epidermal growth factor-like domain 7 in pancreatic cancer. Last author DOI 10.1016/s1499-3872(14)60272-1↗
- Insulin-like growth factor 1 receptor (IGF-1R) as a target of MiR-497 and plasma IGF-1R levels associated with TNM stage of pancreatic cancer. Co-author DOI 10.1371/journal.pone.0092847↗
- CHIP is a novel tumor suppressor in pancreatic cancer through targeting EGFR. Co-author DOI 10.18632/oncotarget.1890↗
- Notch1 contributes to chemoresistance to gemcitabine and serves as an unfavorable prognostic indicator in pancreatic cancer. Last author DOI 10.1007/s00268-013-2010-0↗
- MLH1 as a direct target of MiR-155 and a potential predictor of favorable prognosis in pancreatic cancer. Last author DOI 10.1007/s11605-013-2230-5↗
- Induction, modulation and potential targets of miR-210 in pancreatic cancer cells. Last author DOI 10.1016/s1499-3872(12)60168-4↗
Closest collaborators
The 19 strongest of 19 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Lei You · 8 shared papers, 2013–2017 · strength 0.35
- Paper Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14. DOI 10.1158/0008-5472.can-16-2339↗
- Paper Insulin-like growth factor 1 receptor (IGF-1R) as a target of MiR-497 and plasma IGF-1R levels associated with TNM stag… DOI 10.1371/journal.pone.0092847↗
- Paper Alteration of the intrinsic apoptosis pathway is involved in Notch-induced chemoresistance to gemcitabine in pancreatic… DOI 10.1016/j.arcmed.2013.10.001↗
- Taiping Zhang · 7 shared papers, 2012–2014 · strength 0.28
- Paper Insulin-like growth factor 1 receptor (IGF-1R) as a target of MiR-497 and plasma IGF-1R levels associated with TNM stag… DOI 10.1371/journal.pone.0092847↗
- Paper Alteration of the intrinsic apoptosis pathway is involved in Notch-induced chemoresistance to gemcitabine in pancreatic… DOI 10.1016/j.arcmed.2013.10.001↗
- Paper N-wasp in pancreatic ductal adenocarcinoma: associations with perineural invasion and poor prognosis. DOI 10.1007/s00268-014-2500-8↗
- Yu-Pei Zhao · 7 shared papers, 2012–2014 · strength 0.28
- Paper Insulin-like growth factor 1 receptor (IGF-1R) as a target of MiR-497 and plasma IGF-1R levels associated with TNM stag… DOI 10.1371/journal.pone.0092847↗
- Paper Alteration of the intrinsic apoptosis pathway is involved in Notch-induced chemoresistance to gemcitabine in pancreatic… DOI 10.1016/j.arcmed.2013.10.001↗
- Paper N-wasp in pancreatic ductal adenocarcinoma: associations with perineural invasion and poor prognosis. DOI 10.1007/s00268-014-2500-8↗
- Li Zhou · 7 shared papers, 2012–2014 · strength 0.27
- Paper Alteration of the intrinsic apoptosis pathway is involved in Notch-induced chemoresistance to gemcitabine in pancreatic… DOI 10.1016/j.arcmed.2013.10.001↗
- Paper N-wasp in pancreatic ductal adenocarcinoma: associations with perineural invasion and poor prognosis. DOI 10.1007/s00268-014-2500-8↗
- Paper Prognostic significance of epidermal growth factor-like domain 7 in pancreatic cancer. DOI 10.1016/s1499-3872(14)60272-1↗
- Tianxiao Wang · 4 shared papers, 2013–2014 · strength 0.16
- Paper Insulin-like growth factor 1 receptor (IGF-1R) as a target of MiR-497 and plasma IGF-1R levels associated with TNM stag… DOI 10.1371/journal.pone.0092847↗
- Paper Alteration of the intrinsic apoptosis pathway is involved in Notch-induced chemoresistance to gemcitabine in pancreatic… DOI 10.1016/j.arcmed.2013.10.001↗
- Paper CHIP is a novel tumor suppressor in pancreatic cancer through targeting EGFR. DOI 10.18632/oncotarget.1890↗
- Ge Chen · 3 shared papers, 2012–2014 · strength 0.11
- Paper Alteration of the intrinsic apoptosis pathway is involved in Notch-induced chemoresistance to gemcitabine in pancreatic… DOI 10.1016/j.arcmed.2013.10.001↗
- Paper Notch1 contributes to chemoresistance to gemcitabine and serves as an unfavorable prognostic indicator in pancreatic ca… DOI 10.1007/s00268-013-2010-0↗
- Paper Induction, modulation and potential targets of miR-210 in pancreatic cancer cells. DOI 10.1016/s1499-3872(12)60168-4↗
- Yupei Zhao · 2 shared papers, 2014–2017 · strength 0.10
- Jianwei Xu · 2 shared papers, 2014 · strength 0.08
- Junchao Guo · 2 shared papers, 2014 · strength 0.08
- Xiao Hong Du · 2 shared papers, 2013–2014 · strength 0.08
- Paper Alteration of the intrinsic apoptosis pathway is involved in Notch-induced chemoresistance to gemcitabine in pancreatic… DOI 10.1016/j.arcmed.2013.10.001↗
- Paper Notch1 contributes to chemoresistance to gemcitabine and serves as an unfavorable prognostic indicator in pancreatic ca… DOI 10.1007/s00268-013-2010-0↗
- Ziwen Liu · 1 shared paper, 2017 · strength 0.06
- Yongxing Du · 1 shared paper, 2017 · strength 0.06
- Xiaoxia Ren · 1 shared paper, 2017 · strength 0.06
- Wenming Wu · 1 shared paper, 2014 · strength 0.04
- Min Li · 1 shared paper, 2014 · strength 0.04
- Zhe Cao · 1 shared paper, 2014 · strength 0.04
- Jingxuan Yang · 1 shared paper, 2014 · strength 0.04
- Wen-Jing Liu · 1 shared paper, 2013 · strength 0.04
- Weiyun Chen · 1 shared paper, 2012 · strength 0.03
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.