ResearcherPhilipps University of MarburgActive in the last 2 years
Corinna Ulrike Keber
Matched on ORCID iD 0000-0002-4312-1377; OpenAlex author ID A5017968976. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 39
2021: 1 paper · 2022: none · 2023: 2 papers · 2024: 2 papers · 2025: none · 2026: 3 papers
Papers 8
- Deciphering the regulatory role of ADAM8 in the PDAC tumor microenvironment. Co-author DOI 10.1007/s13402-026-01266-7↗
- Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. Co-author DOI 10.1016/j.bbcan.2026.189614↗
- Tumor slice cultures enable rapid ex vivo profiling of patient-specific drug sensitivity in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41698-026-01663-z↗
- BAG6 restricts pancreatic cancer progression by suppressing the release of IL33-presenting extracellular vesicles and the activation of mast cells. Co-author DOI 10.1038/s41423-024-01195-1↗
- DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2023-331854↗
- Dissecting the role of toll-like receptor 7 in pancreatic cancer. Co-author DOI 10.1002/cam4.5606↗
- IL18 Receptor Signaling Regulates Tumor-Reactive CD8+ T-cell Exhaustion via Activation of the IL2/STAT5/mTOR Pathway in a Pancreatic Cancer Model. Co-author DOI 10.1158/2326-6066.cir-22-0398↗
- PRMT1 promotes the tumor suppressor function of p14ARF and is indicative for pancreatic cancer prognosis. Co-author DOI 10.15252/embj.2020106777↗
Closest collaborators
The 25 strongest of 39 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Thomas Mathias Gress · 4 shared papers, 2023–2026 · strength 0.76
- Paper Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. DOI 10.1016/j.bbcan.2026.189614↗
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper Dissecting the role of toll-like receptor 7 in pancreatic cancer. DOI 10.1002/cam4.5606↗
- Uta‐Maria Bauer · 3 shared papers, 2021–2026 · strength 0.66
- Paper Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. DOI 10.1016/j.bbcan.2026.189614↗
- Paper Deciphering the regulatory role of ADAM8 in the PDAC tumor microenvironment. DOI 10.1007/s13402-026-01266-7↗
- Paper PRMT1 promotes the tumor suppressor function of p14ARF and is indicative for pancreatic cancer prognosis. DOI 10.15252/embj.2020106777↗
- Anna I. Brichkina · 3 shared papers, 2024–2026 · strength 0.60
- Paper Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. DOI 10.1016/j.bbcan.2026.189614↗
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper BAG6 restricts pancreatic cancer progression by suppressing the release of IL33-presenting extracellular vesicles and t… DOI 10.1038/s41423-024-01195-1↗
- Matthias Lauth · 3 shared papers, 2024–2026 · strength 0.60
- Paper Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. DOI 10.1016/j.bbcan.2026.189614↗
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper BAG6 restricts pancreatic cancer progression by suppressing the release of IL33-presenting extracellular vesicles and t… DOI 10.1038/s41423-024-01195-1↗
- Magdalena Huber · 3 shared papers, 2023–2026 · strength 0.60
- Paper Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. DOI 10.1016/j.bbcan.2026.189614↗
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper IL18 Receptor Signaling Regulates Tumor-Reactive CD8+ T-cell Exhaustion via Activation of the IL2/STAT5/mTOR Pathway in… DOI 10.1158/2326-6066.cir-22-0398↗
- Jörg Walter Bartsch · 2 shared papers, 2026 · strength 0.55
- Malte Buchholz · 3 shared papers, 2023–2024 · strength 0.45
- Paper Dissecting the role of toll-like receptor 7 in pancreatic cancer. DOI 10.1002/cam4.5606↗
- Paper BAG6 restricts pancreatic cancer progression by suppressing the release of IL33-presenting extracellular vesicles and t… DOI 10.1038/s41423-024-01195-1↗
- Paper IL18 Receptor Signaling Regulates Tumor-Reactive CD8+ T-cell Exhaustion via Activation of the IL2/STAT5/mTOR Pathway in… DOI 10.1158/2326-6066.cir-22-0398↗
- Elke Pogge von Strandmann · 2 shared papers, 2024–2026 · strength 0.44
- Detlef Klaus Bartsch · 3 shared papers, 2021–2024 · strength 0.43
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper Dissecting the role of toll-like receptor 7 in pancreatic cancer. DOI 10.1002/cam4.5606↗
- Paper PRMT1 promotes the tumor suppressor function of p14ARF and is indicative for pancreatic cancer prognosis. DOI 10.15252/embj.2020106777↗
- Carsten Denkert · 2 shared papers, 2024 · strength 0.31
- Felix S.R. Picard · 2 shared papers, 2023–2024 · strength 0.31
- Veronika Lutz · 2 shared papers, 2023–2024 · strength 0.31
- Emily P. Slater · 2 shared papers, 2021–2023 · strength 0.27
- Kimia Zandieh · 1 shared paper, 2026 · strength 0.26
- Yutong Gao · 1 shared paper, 2026 · strength 0.26
- Pietro Di Fazio · 1 shared paper, 2026 · strength 0.26
- Benjamin Heckelmann · 1 shared paper, 2026 · strength 0.22
- Tobias Keck · 1 shared paper, 2026 · strength 0.22
- Louisa Bolm · 1 shared paper, 2026 · strength 0.22
- Rüdiger Braun · 1 shared paper, 2026 · strength 0.22
- Steffen Deichmann · 1 shared paper, 2026 · strength 0.22
- Kim C. Honselmann · 1 shared paper, 2026 · strength 0.22
- Christoph Röcken · 1 shared paper, 2026 · strength 0.22
- Jens Uwe Marquardt · 1 shared paper, 2026 · strength 0.22
- Ulrich Friedrich Wellner · 1 shared paper, 2026 · strength 0.22
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.