ResearcherPhilipps University of MarburgActive in the last 2 years
Thomas Mathias Gress
Matched on ORCID iD 0000-0002-9333-5461; OpenAlex author ID A5080497221, A5142767908, A5158542081. Sources: OpenAlex.
Their work
- Papers
- 72 (first author 7, last author 7)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 239
2011: 3 papers · 2012: 5 papers · 2013: 10 papers · 2014: 3 papers · 2015: 5 papers · 2016: 6 papers · 2017: 4 papers · 2018: 3 papers · 2019: 3 papers · 2020: 6 papers · 2021: 7 papers · 2022: 1 paper · 2023: 5 papers · 2024: 4 papers · 2025: 4 papers · 2026: 3 papers
Papers 50 of 72
- Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. Co-author DOI 10.1016/j.bbcan.2026.189614↗
- Publisher Correction: Deciphering the role of complement system genes in pancreatic cancer susceptibility and prognosis. Co-author DOI 10.1038/s41467-026-69055-2↗
- A Comprehensive Assessment of the Causal Relationship between Atopic Conditions and Pancreatic Cancer Risk: A Two-Sample Mendelian Randomization Study. Co-author DOI 10.1158/1055-9965.epi-25-1601↗
- Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. Co-author DOI 10.1002/ijc.35397↗
- Combined analysis of a serum mRNA/miRNA marker signature and CA 19-9 for timely and accurate diagnosis of recurrence after resection of pancreatic ductal adeno… Last author DOI 10.1002/ueg2.12676↗
- Validation of two plasma multimetabolite signatures for patients at risk of or with suspected pancreatic ductal adenocarcinoma (METAPAC): a prospective, multic… Co-author DOI 10.1016/s2468-1253(25)00056-1↗
- Deciphering the role of complement system genes in pancreatic cancer susceptibility and prognosis. Co-author DOI 10.1038/s41467-025-65811-y↗
- Pancreatic cancer screening is effective in individuals at risk with predisposing germline gene variants, but not in gene variant-negative familial pancreatic … Co-author DOI 10.1002/ueg2.12631↗
- HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaustive analysis of theHLAvariation in PDAC Co-author DOI 10.1101/2024.08.29.24312704↗
- DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. Co-author DOI 10.1136/gutjnl-2023-331854↗
- Optimized Spheroid Model of Pancreatic Cancer Demonstrates Influence of Macrophage-T Cell Interaction for Intratumoral T Cell Motility. Co-author DOI 10.3390/cancers17010051↗
- Dissecting the role of toll-like receptor 7 in pancreatic cancer. Co-author DOI 10.1002/cam4.5606↗
- IL-17A-producing CD8+ T cells promote PDAC via induction of inflammatory cancer-associated fibroblasts. Co-author DOI 10.1136/gutjnl-2022-327855↗
- IL18 Receptor Signaling Regulates Tumor-Reactive CD8+ T-cell Exhaustion via Activation of the IL2/STAT5/mTOR Pathway in a Pancreatic Cancer Model. Co-author DOI 10.1158/2326-6066.cir-22-0398↗
- Corrigendum: Clinical impact of molecular subtyping of pancreatic cancer. Co-author DOI 10.3389/fcell.2023.1179559↗
- IL18 Receptor Signaling Inhibits Intratumoral CD8+ T-Cell Migration in a Murine Pancreatic Cancer Model. Co-author DOI 10.3390/cells12030456↗
- Timeline of Development of Pancreatic Cancer and Implications for Successful Early Detection in High-Risk Individuals. Co-author DOI 10.1053/j.gastro.2021.10.014↗
- New Insights Into Pancreatic Cancer: Notes from a Virtual Meeting. Co-author DOI 10.1053/j.gastro.2021.04.082↗
- Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. Co-author DOI 10.1097/cej.0000000000000588↗
- Deciphering the complex interplay between pancreatic cancer, diabetes mellitus subtypes and obesity/BMI through causal inference and mediation analyses. Co-author DOI 10.1136/gutjnl-2019-319990↗
- A multilayered post-GWAS assessment on genetic susceptibility to pancreatic cancer. Co-author DOI 10.1186/s13073-020-00816-4↗
- The German National Case Collection for Familial Pancreatic Carcinoma (FaPaCa)—Knowledge Gained in 20 Years. Co-author DOI 10.3238/arztebl.m2021.0004↗
- Clinical Impact of Molecular Subtyping of Pancreatic Cancer. Co-author DOI 10.3389/fcell.2021.743908↗
- Unraveling the Molecular Tumor-Promoting Regulation of Cofilin-1 in Pancreatic Cancer. Co-author DOI 10.3390/cancers13040725↗
- International consensus guidelines on surveillance for pancreatic cancer in chronic pancreatitis. Recommendations from the working group for the international … Co-author DOI 10.1016/j.pan.2020.05.011↗
- Management of patients with increased risk for familial pancreatic cancer: updated recommendations from the International Cancer of the Pancreas Screening (CAP… Co-author DOI 10.1136/gutjnl-2019-319352↗
- Correction: Management of patients with increased risk for familial pancreatic cancer: updated recommendations for the international cancer of the pancreas scr… Co-author DOI 10.1136/gutjnl-2019-319352corr1↗
- Microenvironmental Determinants of Pancreatic Cancer. Co-author DOI 10.1152/physrev.00042.2019↗
- Pancreatic Cancer Risk in Relation to Lifetime Smoking Patterns, Tobacco Type, and Dose-Response Relationships. Co-author DOI 10.1158/1055-9965.epi-19-1027↗
- The Immune Microenvironment in Pancreatic Cancer. Co-author DOI 10.3390/ijms21197307↗
- Pancreatic cancer and autoimmune diseases: An association sustained by computational and epidemiological case-control approaches. Co-author DOI 10.1002/ijc.31866↗
- Author Correction: TGF-β induces miR-100 and miR-125b but blocks let-7a through LIN28B controlling PDAC progression. Co-author DOI 10.1038/s41467-019-11752-2↗
- Stromal biology and therapy in pancreatic cancer: ready for clinical translation? Last author DOI 10.1136/gutjnl-2018-316451↗
- [Pancreatic ductal adenocarcinoma]. First author DOI 10.1007/s00108-018-0460-z↗
- TGF-β induces miR-100 and miR-125b but blocks let-7a through LIN28B controlling PDAC progression. Co-author DOI 10.1038/s41467-018-03962-x↗
- Risk of pancreatic cancer associated with family history of cancer and other medical conditions by accounting for smoking among relatives. Co-author DOI 10.1093/ije/dyx269↗
- A systems approach identifies time-dependent associations of multimorbidities with pancreatic cancer risk. Co-author DOI 10.1093/annonc/mdx167↗
- Pharmacological macrophage inhibition decreases metastasis formation in a genetic model of pancreatic cancer. Co-author DOI 10.1136/gutjnl-2015-310049↗
- Reduced risk of pancreatic cancer associated with asthma and nasal allergies. Co-author DOI 10.1136/gutjnl-2015-310442↗
- The deubiquitinating enzyme USP5 promotes pancreatic cancer via modulating cell cycle regulators. Co-author DOI 10.18632/oncotarget.19882↗
- Prevailing over T cell exhaustion: New developments in the immunotherapy of pancreatic cancer. Co-author DOI 10.1016/j.canlet.2016.02.057↗
- miRNA dynamics in tumor-infiltrating myeloid cells modulating tumor progression in pancreatic cancer. Co-author DOI 10.1080/2162402x.2016.1160181↗
- Refinement of screening for familial pancreatic cancer. Last author DOI 10.1136/gutjnl-2015-311098↗
- PLAC8 Localizes to the Inner Plasma Membrane of Pancreatic Cancer Cells and Regulates Cell Growth and Disease Progression through Critical Cell-Cycle Regulator… Co-author DOI 10.1158/0008-5472.can-15-0216↗
- GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. Co-author DOI 10.1158/1535-7163.mct-15-0309↗
- DocOx (AIO-PK0106): a phase II trial of docetaxel and oxaliplatin as a second line systemic therapy in patients with advanced pancreatic ductal adenocarcinoma. Co-author DOI 10.1186/s12885-016-2052-4↗
- Kif20a inhibition reduces migration and invasion of pancreatic cancer cells. Co-author DOI 10.1016/j.jss.2015.03.070↗
- Controversy on the time to progression of pancreatic ductal adenocarcinoma. Last author DOI 10.1136/gutjnl-2014-309066↗
- Stromal biology and therapy in pancreatic cancer: a changing paradigm. Last author DOI 10.1136/gutjnl-2015-309304↗
- Emerging role of microRNAs to tackle drug resistance in pancreatic cancer. First author DOI 10.1136/gutjnl-2015-309503↗
Closest collaborators
The 25 strongest of 239 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Malte Buchholz · 22 shared papers, 2011–2025 · strength 2.09
- Paper Optimized Spheroid Model of Pancreatic Cancer Demonstrates Influence of Macrophage-T Cell Interaction for Intratumoral … DOI 10.3390/cancers17010051↗
- Paper Combined analysis of a serum mRNA/miRNA marker signature and CA 19-9 for timely and accurate diagnosis of recurrence af… DOI 10.1002/ueg2.12676↗
- Paper IL18 Receptor Signaling Inhibits Intratumoral CD8+ T-Cell Migration in a Murine Pancreatic Cancer Model. DOI 10.3390/cells12030456↗
- Magdalena Huber · 9 shared papers, 2016–2026 · strength 1.39
- Paper Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. DOI 10.1016/j.bbcan.2026.189614↗
- Paper Optimized Spheroid Model of Pancreatic Cancer Demonstrates Influence of Macrophage-T Cell Interaction for Intratumoral … DOI 10.3390/cancers17010051↗
- Paper IL18 Receptor Signaling Inhibits Intratumoral CD8+ T-Cell Migration in a Murine Pancreatic Cancer Model. DOI 10.3390/cells12030456↗
- Alfredo Carrato · 14 shared papers, 2016–2025 · strength 1.28
- Paper Combined analysis of a serum mRNA/miRNA marker signature and CA 19-9 for timely and accurate diagnosis of recurrence af… DOI 10.1002/ueg2.12676↗
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Detlef Klaus Bartsch · 14 shared papers, 2012–2024 · strength 1.28
- Paper Pancreatic cancer screening is effective in individuals at risk with predisposing germline gene variants, but not in ge… DOI 10.1002/ueg2.12631↗
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper Dissecting the role of toll-like receptor 7 in pancreatic cancer. DOI 10.1002/cam4.5606↗
- Christian Alexander Bauer · 8 shared papers, 2016–2024 · strength 1.10
- Paper Optimized Spheroid Model of Pancreatic Cancer Demonstrates Influence of Macrophage-T Cell Interaction for Intratumoral … DOI 10.3390/cancers17010051↗
- Paper IL18 Receptor Signaling Inhibits Intratumoral CD8+ T-Cell Migration in a Murine Pancreatic Cancer Model. DOI 10.3390/cells12030456↗
- Paper IL18 Receptor Signaling Regulates Tumor-Reactive CD8+ T-cell Exhaustion via Activation of the IL2/STAT5/mTOR Pathway in… DOI 10.1158/2326-6066.cir-22-0398↗
- Rita Teresa Lawlor · 9 shared papers, 2017–2025 · strength 0.99
- Paper Combined analysis of a serum mRNA/miRNA marker signature and CA 19-9 for timely and accurate diagnosis of recurrence af… DOI 10.1002/ueg2.12676↗
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper Dissecting the role of toll-like receptor 7 in pancreatic cancer. DOI 10.1002/cam4.5606↗
- Aldo Scarpa · 11 shared papers, 2014–2025 · strength 0.99
- Paper Combined analysis of a serum mRNA/miRNA marker signature and CA 19-9 for timely and accurate diagnosis of recurrence af… DOI 10.1002/ueg2.12676↗
- Paper Dissecting the role of toll-like receptor 7 in pancreatic cancer. DOI 10.1002/cam4.5606↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Núria Malats · 10 shared papers, 2017–2025 · strength 0.97
- Paper Combined analysis of a serum mRNA/miRNA marker signature and CA 19-9 for timely and accurate diagnosis of recurrence af… DOI 10.1002/ueg2.12676↗
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Jorg H. Kleeff · 11 shared papers, 2015–2025 · strength 0.89
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- William Greenhalf · 10 shared papers, 2017–2025 · strength 0.84
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Patrick Michl · 15 shared papers, 2011–2021 · strength 0.80
- Paper Stromal biology and therapy in pancreatic cancer: ready for clinical translation? DOI 10.1136/gutjnl-2018-316451↗
- Paper New Insights Into Pancreatic Cancer: Notes from a Virtual Meeting. DOI 10.1053/j.gastro.2021.04.082↗
- Paper Pharmacological macrophage inhibition decreases metastasis formation in a genetic model of pancreatic cancer. DOI 10.1136/gutjnl-2015-310049↗
- Francisco X. Real · 10 shared papers, 2014–2025 · strength 0.79
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Matthias Lauth · 5 shared papers, 2019–2026 · strength 0.79
- Paper Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. DOI 10.1016/j.bbcan.2026.189614↗
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper IL-17A-producing CD8+ T cells promote PDAC via induction of inflammatory cancer-associated fibroblasts. DOI 10.1136/gutjnl-2022-327855↗
- Luis Muñoz‐Bellvís · 9 shared papers, 2017–2025 · strength 0.76
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- J. Enrique Domínguez‐Muñoz · 9 shared papers, 2017–2025 · strength 0.76
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Tatjana Crnogorac‐Jurcevic · 9 shared papers, 2017–2025 · strength 0.76
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Corinna Ulrike Keber · 4 shared papers, 2023–2026 · strength 0.76
- Paper Emerging concepts, hot topics, and open questions in today's pancreatic cancer research. DOI 10.1016/j.bbcan.2026.189614↗
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper Dissecting the role of toll-like receptor 7 in pancreatic cancer. DOI 10.1002/cam4.5606↗
- Emily P. Slater · 8 shared papers, 2013–2024 · strength 0.75
- Paper Pancreatic cancer screening is effective in individuals at risk with predisposing germline gene variants, but not in ge… DOI 10.1002/ueg2.12631↗
- Paper Dissecting the role of toll-like receptor 7 in pancreatic cancer. DOI 10.1002/cam4.5606↗
- Paper The German National Case Collection for Familial Pancreatic Carcinoma (FaPaCa)—Knowledge Gained in 20 Years. DOI 10.3238/arztebl.m2021.0004↗
- Veronika Lutz · 4 shared papers, 2023–2024 · strength 0.65
- Paper Optimized Spheroid Model of Pancreatic Cancer Demonstrates Influence of Macrophage-T Cell Interaction for Intratumoral … DOI 10.3390/cancers17010051↗
- Paper DYRK1B blockade promotes tumoricidal macrophage activity in pancreatic cancer. DOI 10.1136/gutjnl-2023-331854↗
- Paper IL18 Receptor Signaling Regulates Tumor-Reactive CD8+ T-cell Exhaustion via Activation of the IL2/STAT5/mTOR Pathway in… DOI 10.1158/2326-6066.cir-22-0398↗
- Albrecht Neeße · 10 shared papers, 2011–2020 · strength 0.64
- Paper Microenvironmental Determinants of Pancreatic Cancer. DOI 10.1152/physrev.00042.2019↗
- Paper Emerging role of microRNAs to tackle drug resistance in pancreatic cancer. DOI 10.1136/gutjnl-2015-309503↗
- Paper Stromal biology and therapy in pancreatic cancer: ready for clinical translation? DOI 10.1136/gutjnl-2018-316451↗
- Eithne Costello · 9 shared papers, 2016–2024 · strength 0.63
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Paper Deciphering the complex interplay between pancreatic cancer, diabetes mellitus subtypes and obesity/BMI through causal … DOI 10.1136/gutjnl-2019-319990↗
- Matthias J. Löhr · 8 shared papers, 2017–2025 · strength 0.62
- Paper Low type-2 immune effectors modulate atopic diseases' protective role in pancreatic cancer risk. DOI 10.1002/ijc.35397↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Paper Deciphering the complex interplay between pancreatic cancer, diabetes mellitus subtypes and obesity/BMI through causal … DOI 10.1136/gutjnl-2019-319990↗
- Christoph W. Michalski · 8 shared papers, 2015–2024 · strength 0.59
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Paper Deciphering the complex interplay between pancreatic cancer, diabetes mellitus subtypes and obesity/BMI through causal … DOI 10.1136/gutjnl-2019-319990↗
- Linda Sharp · 8 shared papers, 2017–2024 · strength 0.58
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Paper Deciphering the complex interplay between pancreatic cancer, diabetes mellitus subtypes and obesity/BMI through causal … DOI 10.1136/gutjnl-2019-319990↗
- Bo Kong · 8 shared papers, 2017–2024 · strength 0.58
- Paper HLA-A*02:01allele is associated with decreased risk and a longer survival in pancreatic cancer: Results from an exhaust… DOI 10.1101/2024.08.29.24312704↗
- Paper Gallbladder disease and pancreatic cancer risk: a multicentric case-control European study. DOI 10.1097/cej.0000000000000588↗
- Paper Deciphering the complex interplay between pancreatic cancer, diabetes mellitus subtypes and obesity/BMI through causal … DOI 10.1136/gutjnl-2019-319990↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.