ResearcherLyon 1 UniversitéNo recent work listed
Laurent Bartholin
Matched on ORCID iD 0000-0002-5637-3223; OpenAlex author ID A5075937328. Sources: OpenAlex.
Their work
- Papers
- 7 (first author 1, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 33
2012: 1 paper · 2013: none · 2014: none · 2015: none · 2016: 2 papers · 2017: none · 2018: 2 papers · 2019: 1 paper · 2020: 1 paper
Papers 7
- Correction: Schwann cells support oncogenic potential of pancreatic cancer cells through TGFβ signaling. Last author DOI 10.1038/s41419-020-2262-1↗
- Schwann cells support oncogenic potential of pancreatic cancer cells through TGFβ signaling. Last author DOI 10.1038/s41419-019-2116-x↗
- Prognostic stratification of resected pancreatic ductal adenocarcinoma: Past, present, and future. Co-author DOI 10.1016/j.dld.2018.08.009↗
- Immune therapies in pancreatic ductal adenocarcinoma: Where are we now? First author DOI 10.3748/wjg.v24.i20.2137↗
- TAp73 loss favors Smad-independent TGF-β signaling that drives EMT in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/cdd.2016.18↗
- Lysyl oxidase family activity promotes resistance of pancreatic ductal adenocarcinoma to chemotherapy by limiting the intratumoral anticancer drug distribution. Co-author DOI 10.18632/oncotarget.8527↗
- Homotypic cell cannibalism, a cell-death process regulated by the nuclear protein 1, opposes to metastasis in pancreatic cancer. Co-author DOI 10.1002/emmm.201201255↗
Closest collaborators
The 25 strongest of 33 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Cindy Neuzillet · 4 shared papers, 2018–2020 · strength 0.41
- Paper Immune therapies in pancreatic ductal adenocarcinoma: Where are we now? DOI 10.3748/wjg.v24.i20.2137↗
- Paper Prognostic stratification of resected pancreatic ductal adenocarcinoma: Past, present, and future. DOI 10.1016/j.dld.2018.08.009↗
- Paper Correction: Schwann cells support oncogenic potential of pancreatic cancer cells through TGFβ signaling. DOI 10.1038/s41419-020-2262-1↗
- Richard Tomasini · 3 shared papers, 2016–2020 · strength 0.20
- Paper Correction: Schwann cells support oncogenic potential of pancreatic cancer cells through TGFβ signaling. DOI 10.1038/s41419-020-2262-1↗
- Paper Schwann cells support oncogenic potential of pancreatic cancer cells through TGFβ signaling. DOI 10.1038/s41419-019-2116-x↗
- Paper TAp73 loss favors Smad-independent TGF-β signaling that drives EMT in pancreatic ductal adenocarcinoma. DOI 10.1038/cdd.2016.18↗
- Jerome F. Cros · 2 shared papers, 2016–2018 · strength 0.17
- Valérie Hervieu · 2 shared papers, 2019–2020 · strength 0.15
- Tanguy Fenouil · 2 shared papers, 2019–2020 · strength 0.15
- Mustapha Adham · 2 shared papers, 2019–2020 · strength 0.15
- Élodie Roger · 2 shared papers, 2019–2020 · strength 0.15
- Victoire Cardot‐Ruffino · 2 shared papers, 2019–2020 · strength 0.15
- Philippe Alexandre Cassier · 2 shared papers, 2019–2020 · strength 0.15
- Marc Hilmi · 1 shared paper, 2018 · strength 0.14
- Aline Barhli · 1 shared paper, 2018 · strength 0.12
- Juan Iovanna · 2 shared papers, 2012–2016 · strength 0.08
- Christian Bressy · 1 shared paper, 2016 · strength 0.05
- Fabienne Guillaumond · 1 shared paper, 2016 · strength 0.05
- Sophie Vasseur · 1 shared paper, 2016 · strength 0.05
- Julie Leca · 1 shared paper, 2016 · strength 0.05
- Jérémy Nigri · 1 shared paper, 2016 · strength 0.05
- A K Thakur · 1 shared paper, 2016 · strength 0.05
- Raphaël Maréchal · 1 shared paper, 2016 · strength 0.05
- Magali Svrcek · 1 shared paper, 2016 · strength 0.05
- David Bernard · 1 shared paper, 2016 · strength 0.05
- Johann Gout · 1 shared paper, 2016 · strength 0.05
- Christelle de la Fouchardière · 1 shared paper, 2016 · strength 0.05
- Benjamin Le Calvé · 1 shared paper, 2016 · strength 0.05
- Jean‐Baptiste Bachet · 1 shared paper, 2016 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.