ResearcherCentre National de la Recherche ScientifiqueActive in the last 2 years
Fabienne Guillaumond
Matched on ORCID iD 0000-0001-7456-7315; OpenAlex author ID A5047258963. Sources: OpenAlex.
Their work
- Papers
- 16 (first author 1, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 58
2013: 2 papers · 2014: none · 2015: 3 papers · 2016: 1 paper · 2017: 2 papers · 2018: 1 paper · 2019: none · 2020: none · 2021: 1 paper · 2022: 3 papers · 2023: 1 paper · 2024: none · 2025: 1 paper · 2026: 1 paper
Papers 16
- Co-translational O-glycosylation driven by GALNTs spatial reprogramming fosters pancreatic cancer growth Co-author DOI 10.64898/2026.09.04.749345↗
- The extracellular matrix drives guanylate production and protects pancreatic cancer cells from oxaliplatin-induced DNA damage. Co-author DOI 10.1126/sciadv.adu2276↗
- Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. Co-author DOI 10.1016/j.isci.2023.106899↗
- Sympathetic axonal sprouting induces changes in macrophage populations and protects against pancreatic cancer. Co-author DOI 10.1038/s41467-022-29659-w↗
- CD9 mediates the uptake of extracellular vesicles from cancer-associated fibroblasts that promote pancreatic cancer cell aggressiveness. Co-author DOI 10.1126/scisignal.abg8191↗
- Ketogenic HMG-CoA lyase and its product β-hydroxybutyrate promote pancreatic cancer progression. Co-author DOI 10.15252/embj.2021110466↗
- LDL receptor-peptide conjugate as in vivo tool for specific targeting of pancreatic ductal adenocarcinoma. Last author DOI 10.1038/s42003-021-02508-0↗
- LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. Co-author DOI 10.1158/0008-5472.can-15-2790↗
- Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenografts. Co-author DOI 10.1016/j.celrep.2017.11.003↗
- Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. Co-author DOI 10.1038/ncomms16031↗
- TAp73 loss favors Smad-independent TGF-β signaling that drives EMT in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/cdd.2016.18↗
- Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. Co-author DOI 10.1038/cddis.2014.557↗
- [Pancreatic adenocarcinoma: a tumor highly dependent on lipoproteins]. First author DOI 10.1051/medsci/20153108009↗
- Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreatic adenocarcinoma. Co-author DOI 10.1073/pnas.1421601112↗
- Strengthened glycolysis under hypoxia supports tumor symbiosis and hexosamine biosynthesis in pancreatic adenocarcinoma. Co-author DOI 10.1073/pnas.1219555110↗
- The metabolic facet of pancreatic cancer: how hypoxia shapes fatal cancer cells. Co-author DOI 10.4161/cc.24489↗
Closest collaborators
The 25 strongest of 58 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Sophie Vasseur · 14 shared papers, 2013–2026 · strength 1.30
- Paper Co-translational O-glycosylation driven by GALNTs spatial reprogramming fosters pancreatic cancer growth DOI 10.64898/2026.09.04.749345↗
- Paper The extracellular matrix drives guanylate production and protects pancreatic cancer cells from oxaliplatin-induced DNA … DOI 10.1126/sciadv.adu2276↗
- Paper CD9 mediates the uptake of extracellular vesicles from cancer-associated fibroblasts that promote pancreatic cancer cel… DOI 10.1126/scisignal.abg8191↗
- Richard Tomasini · 12 shared papers, 2013–2026 · strength 1.19
- Paper Co-translational O-glycosylation driven by GALNTs spatial reprogramming fosters pancreatic cancer growth DOI 10.64898/2026.09.04.749345↗
- Paper The extracellular matrix drives guanylate production and protects pancreatic cancer cells from oxaliplatin-induced DNA … DOI 10.1126/sciadv.adu2276↗
- Paper CD9 mediates the uptake of extracellular vesicles from cancer-associated fibroblasts that promote pancreatic cancer cel… DOI 10.1126/scisignal.abg8191↗
- Juan Iovanna · 11 shared papers, 2013–2023 · strength 0.83
- Paper Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. DOI 10.1016/j.isci.2023.106899↗
- Paper CD9 mediates the uptake of extracellular vesicles from cancer-associated fibroblasts that promote pancreatic cancer cel… DOI 10.1126/scisignal.abg8191↗
- Paper Ketogenic HMG-CoA lyase and its product β-hydroxybutyrate promote pancreatic cancer progression. DOI 10.15252/embj.2021110466↗
- Nelson Dusetti · 7 shared papers, 2013–2026 · strength 0.73
- Paper Co-translational O-glycosylation driven by GALNTs spatial reprogramming fosters pancreatic cancer growth DOI 10.64898/2026.09.04.749345↗
- Paper Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. DOI 10.1016/j.isci.2023.106899↗
- Paper Sympathetic axonal sprouting induces changes in macrophage populations and protects against pancreatic cancer. DOI 10.1038/s41467-022-29659-w↗
- François Bertucci · 4 shared papers, 2021–2025 · strength 0.56
- Paper The extracellular matrix drives guanylate production and protects pancreatic cancer cells from oxaliplatin-induced DNA … DOI 10.1126/sciadv.adu2276↗
- Paper CD9 mediates the uptake of extracellular vesicles from cancer-associated fibroblasts that promote pancreatic cancer cel… DOI 10.1126/scisignal.abg8191↗
- Paper Ketogenic HMG-CoA lyase and its product β-hydroxybutyrate promote pancreatic cancer progression. DOI 10.15252/embj.2021110466↗
- Jérémy Nigri · 7 shared papers, 2015–2022 · strength 0.56
- Paper CD9 mediates the uptake of extracellular vesicles from cancer-associated fibroblasts that promote pancreatic cancer cel… DOI 10.1126/scisignal.abg8191↗
- Paper Sympathetic axonal sprouting induces changes in macrophage populations and protects against pancreatic cancer. DOI 10.1038/s41467-022-29659-w↗
- Paper Ketogenic HMG-CoA lyase and its product β-hydroxybutyrate promote pancreatic cancer progression. DOI 10.15252/embj.2021110466↗
- Julie Leca · 6 shared papers, 2013–2022 · strength 0.44
- Paper CD9 mediates the uptake of extracellular vesicles from cancer-associated fibroblasts that promote pancreatic cancer cel… DOI 10.1126/scisignal.abg8191↗
- Paper Ketogenic HMG-CoA lyase and its product β-hydroxybutyrate promote pancreatic cancer progression. DOI 10.15252/embj.2021110466↗
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Tristan Gicquel · 3 shared papers, 2017–2023 · strength 0.32
- Paper Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. DOI 10.1016/j.isci.2023.106899↗
- Paper Ketogenic HMG-CoA lyase and its product β-hydroxybutyrate promote pancreatic cancer progression. DOI 10.15252/embj.2021110466↗
- Paper Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. DOI 10.1038/ncomms16031↗
- Rebecca Bennion · 1 shared paper, 2026 · strength 0.24
- Frédéric Bard · 1 shared paper, 2026 · strength 0.24
- Eric Mas · 1 shared paper, 2026 · strength 0.24
- Brice Chanez · 1 shared paper, 2026 · strength 0.24
- Martin James Humphries · 1 shared paper, 2026 · strength 0.24
- Odile Gayet · 3 shared papers, 2015–2023 · strength 0.23
- Paper Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. DOI 10.1016/j.isci.2023.106899↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreat… DOI 10.1073/pnas.1421601112↗
- Victoire Gouirand · 3 shared papers, 2015–2022 · strength 0.20
- Paper Ketogenic HMG-CoA lyase and its product β-hydroxybutyrate promote pancreatic cancer progression. DOI 10.15252/embj.2021110466↗
- Paper Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. DOI 10.1038/ncomms16031↗
- Paper Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreat… DOI 10.1073/pnas.1421601112↗
- Zainab Hussain · 1 shared paper, 2025 · strength 0.20
- Georgios Efthymiou · 1 shared paper, 2025 · strength 0.20
- Julie Roques · 4 shared papers, 2015–2018 · strength 0.20
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. DOI 10.1038/ncomms16031↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Véronique Secq · 4 shared papers, 2015–2018 · strength 0.20
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. DOI 10.1038/ncomms16031↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Mehdi Ouaïssi · 4 shared papers, 2015–2018 · strength 0.18
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreat… DOI 10.1073/pnas.1421601112↗
- Jared R. Mayers · 2 shared papers, 2017–2022 · strength 0.17
- Matthew G. Vander Heiden · 2 shared papers, 2017–2022 · strength 0.17
- Christian Bressy · 3 shared papers, 2015–2018 · strength 0.15
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper TAp73 loss favors Smad-independent TGF-β signaling that drives EMT in pancreatic ductal adenocarcinoma. DOI 10.1038/cdd.2016.18↗
- Paper Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. DOI 10.1038/cddis.2014.557↗
- Nadine Abdel Hadi · 1 shared paper, 2023 · strength 0.15
- Rawand Masoud · 1 shared paper, 2023 · strength 0.15
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.