ResearcherInsermNo recent work listed
Mehdi Ouaïssi
Matched on ORCID iD 0000-0002-0906-4428; OpenAlex author ID A5016731047. Sources: OpenAlex.
Their work
- Papers
- 16
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 49
2011: 1 paper · 2012: none · 2013: none · 2014: 1 paper · 2015: 7 papers · 2016: 2 papers · 2017: 3 papers · 2018: 1 paper · 2019: none · 2020: 1 paper
Papers 16
- Primary Thromboprophylaxis in Pancreatic Cancer Patients: Why Clinical Practice Guidelines Should Be Implemented. Co-author DOI 10.3390/cancers12030618↗
- LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. Co-author DOI 10.1158/0008-5472.can-15-2790↗
- Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenografts. Co-author DOI 10.1016/j.celrep.2017.11.003↗
- Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain inhibitor JQ1: implications for individua… Co-author DOI 10.15252/emmm.201606975↗
- Venous Resection in Pancreatic Ductal Adenocarcinoma: Impact of Surgical Experience on Early Postoperative Courses. Co-author DOI 10.21873/anticanres.11811↗
- Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. Co-author DOI 10.1172/jci87734↗
- A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. Co-author DOI 10.18632/oncotarget.10776↗
- Transcriptomic analysis predicts survival and sensitivity to anticancer drugs of patients with a pancreatic adenocarcinoma. Co-author DOI 10.1016/j.ajpath.2014.11.029↗
- Impact of venous thromboembolism on the natural history of pancreatic adenocarcinoma. Co-author DOI 10.1016/s1499-3872(15)60397-6↗
- Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. Co-author DOI 10.1038/cddis.2014.557↗
- Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreatic adenocarcinoma. Co-author DOI 10.1073/pnas.1421601112↗
- Targeting CD44 as a novel therapeutic approach for treating pancreatic cancer recurrence. Co-author DOI 10.18632/oncoscience.172↗
- A subgroup of pancreatic adenocarcinoma is sensitive to the 5-aza-dC DNA methyltransferase inhibitor. Co-author DOI 10.18632/oncotarget.2685↗
- Deciphering the cellular source of tumor relapse identifies CD44 as a major therapeutic target in pancreatic adenocarcinoma. Co-author DOI 10.18632/oncotarget.3510↗
- Further characterization of HDAC and SIRT gene expression patterns in pancreatic cancer and their relation to disease outcome. Co-author DOI 10.1371/journal.pone.0108520↗
- Rationale for possible targeting of histone deacetylase signaling in cancer diseases with a special reference to pancreatic cancer. Co-author DOI 10.1155/2011/315939↗
Closest collaborators
The 25 strongest of 49 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Juan Iovanna · 12 shared papers, 2014–2018 · strength 0.52
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Targeting CD44 as a novel therapeutic approach for treating pancreatic cancer recurrence. DOI 10.18632/oncoscience.172↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Nelson Dusetti · 9 shared papers, 2015–2017 · strength 0.38
- Paper Targeting CD44 as a novel therapeutic approach for treating pancreatic cancer recurrence. DOI 10.18632/oncoscience.172↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Olivıer Turrini · 8 shared papers, 2015–2017 · strength 0.38
- Paper Venous Resection in Pancreatic Ductal Adenocarcinoma: Impact of Surgical Experience on Early Postoperative Courses. DOI 10.21873/anticanres.11811↗
- Paper Targeting CD44 as a novel therapeutic approach for treating pancreatic cancer recurrence. DOI 10.18632/oncoscience.172↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Céline Loncle · 9 shared papers, 2014–2017 · strength 0.36
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Véronique Secq · 8 shared papers, 2015–2018 · strength 0.35
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Odile Gayet · 7 shared papers, 2015–2017 · strength 0.29
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. DOI 10.18632/oncotarget.10776↗
- Sophie Vasseur · 6 shared papers, 2015–2018 · strength 0.27
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Jean‐Robert Delpéro · 5 shared papers, 2015–2017 · strength 0.26
- Paper Venous Resection in Pancreatic Ductal Adenocarcinoma: Impact of Surgical Experience on Early Postoperative Courses. DOI 10.21873/anticanres.11811↗
- Paper Targeting CD44 as a novel therapeutic approach for treating pancreatic cancer recurrence. DOI 10.18632/oncoscience.172↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Flora Poizat · 6 shared papers, 2015–2017 · strength 0.25
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. DOI 10.18632/oncotarget.10776↗
- Marine Gilabert · 6 shared papers, 2015–2017 · strength 0.25
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. DOI 10.18632/oncotarget.10776↗
- Marc F. Giovannini · 5 shared papers, 2015–2017 · strength 0.21
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. DOI 10.18632/oncotarget.10776↗
- Pauline Duconseil · 5 shared papers, 2015–2017 · strength 0.20
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. DOI 10.18632/oncotarget.10776↗
- Paper Deciphering the cellular source of tumor relapse identifies CD44 as a major therapeutic target in pancreatic adenocarci… DOI 10.18632/oncotarget.3510↗
- Julie Roques · 4 shared papers, 2015–2018 · strength 0.19
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Fabienne Guillaumond · 4 shared papers, 2015–2018 · strength 0.18
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreat… DOI 10.1073/pnas.1421601112↗
- Richard Tomasini · 4 shared papers, 2015–2018 · strength 0.18
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Paper Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreat… DOI 10.1073/pnas.1421601112↗
- Jérémy Nigri · 4 shared papers, 2015–2018 · strength 0.18
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Paper Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreat… DOI 10.1073/pnas.1421601112↗
- Jean‐Luc Raoul · 4 shared papers, 2015–2017 · strength 0.17
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Transcriptomic analysis predicts survival and sensitivity to anticancer drugs of patients with a pancreatic adenocarcin… DOI 10.1016/j.ajpath.2014.11.029↗
- Dominique Farge Bancel · 2 shared papers, 2015–2020 · strength 0.16
- Christian Bressy · 3 shared papers, 2015–2018 · strength 0.15
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Paper Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. DOI 10.1038/cddis.2014.557↗
- Julie Leca · 3 shared papers, 2015–2018 · strength 0.15
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Paper Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. DOI 10.1038/cddis.2014.557↗
- Benjamin Bian · 3 shared papers, 2016–2017 · strength 0.13
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. DOI 10.18632/oncotarget.10776↗
- Anthony Gonçalvès · 3 shared papers, 2015–2017 · strength 0.12
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Transcriptomic analysis predicts survival and sensitivity to anticancer drugs of patients with a pancreatic adenocarcin… DOI 10.1016/j.ajpath.2014.11.029↗
- Paper A subgroup of pancreatic adenocarcinoma is sensitive to the 5-aza-dC DNA methyltransferase inhibitor. DOI 10.18632/oncotarget.2685↗
- Igor Sielezneff · 3 shared papers, 2011–2015 · strength 0.11
- Paper Impact of venous thromboembolism on the natural history of pancreatic adenocarcinoma. DOI 10.1016/s1499-3872(15)60397-6↗
- Paper Further characterization of HDAC and SIRT gene expression patterns in pancreatic cancer and their relation to disease o… DOI 10.1371/journal.pone.0108520↗
- Paper Rationale for possible targeting of histone deacetylase signaling in cancer diseases with a special reference to pancre… DOI 10.1155/2011/315939↗
- Daniel Pietrasz · 2 shared papers, 2016–2018 · strength 0.11
- Jean‐Baptiste Bachet · 2 shared papers, 2016–2018 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.