ResearcherCentre National de la Recherche ScientifiqueActive in the last 2 years
Odile Gayet
Matched on ORCID iD 0000-0001-8635-3648; OpenAlex author ID A5004923572. Sources: OpenAlex.
Their work
- Papers
- 29 (first author 3, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 69
2015: 4 papers · 2016: 1 paper · 2017: 2 papers · 2018: 3 papers · 2019: 2 papers · 2020: 4 papers · 2021: 6 papers · 2022: 3 papers · 2023: 2 papers · 2024: none · 2025: 1 paper · 2026: 1 paper
Papers 29
- A comprehensive proteomic dataset of secretomes from primary pancreatic cancer cell cultures. Co-author DOI 10.1038/s41597-026-07902-z↗
- The T-Box Transcription Factors TBX2 and TBX3 Are Molecular Targets of Piroctone Olamine in the Treatment of Pancreatic Cancer. Co-author DOI 10.1111/jcmm.70736↗
- Pancreatic ductal adenocarcinoma ubiquitination profiling reveals specific prognostic and theranostic markers. Co-author DOI 10.1016/j.ebiom.2023.104634↗
- Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. Co-author DOI 10.1016/j.isci.2023.106899↗
- Inhibition of glucuronidation in pancreatic cancer improves gemcitabine anticancer activity. Co-author DOI 10.1002/cac2.12365↗
- Relevance of biopsy-derived pancreatic organoids in the development of efficient transcriptomic signatures to predict adjuvant chemosensitivity in pancreatic c… First author DOI 10.1016/j.tranon.2021.101315↗
- Multi-omics data integration and modeling unravels new mechanisms for pancreatic cancer and improves prognostic prediction. Co-author DOI 10.1038/s41698-022-00299-z↗
- Dendrimeric nanosystem consistently circumvents heterogeneous drug response and resistance in pancreatic cancer. Co-author DOI 10.1002/exp.20210003↗
- A transcriptomic signature to predict adjuvant gemcitabine sensitivity in pancreatic adenocarcinoma. First author DOI 10.1016/j.annonc.2020.10.601↗
- Metabolomic profiling of pancreatic adenocarcinoma reveals key features driving clinical outcome and drug resistance. Co-author DOI 10.1016/j.ebiom.2021.103332↗
- Squamousness gain defines pancreatic ductal adenocarcinoma hepatic metastases phenotype, and gemcitabine response. Co-author DOI 10.1016/j.ejca.2021.06.038↗
- Exploring the Complementarity of Pancreatic Ductal Adenocarcinoma Preclinical Models. Co-author DOI 10.3390/cancers13102473↗
- Induction of Apoptosis in Human Pancreatic Cancer Stem Cells by the Endoplasmic Reticulum-Targeted Alkylphospholipid Analog Edelfosine and Potentiation by Auto… First author DOI 10.3390/cancers13236124↗
- Establishment of a pancreatic adenocarcinoma molecular gradient (PAMG) that predicts the clinical outcome of pancreatic cancer. Co-author DOI 10.1016/j.ebiom.2020.102858↗
- Targeting Mitochondrial Complex I Overcomes Chemoresistance in High OXPHOS Pancreatic Cancer. Co-author DOI 10.1016/j.xcrm.2020.100143↗
- Basal-like and classical cells coexist in pancreatic cancer revealed by single-cell analysis on biopsy-derived pancreatic cancer organoids from the classical s… Co-author DOI 10.1096/fj.202000363rr↗
- Evidencing a Pancreatic Ductal Adenocarcinoma Subpopulation Sensitive to the Proteasome Inhibitor Carfilzomib. Co-author DOI 10.1158/1078-0432.ccr-20-1232↗
- PML hyposumoylation is responsible for the resistance of pancreatic cancer. Co-author DOI 10.1096/fj.201901091r↗
- Pancreatic Cancer Organoids for Determining Sensitivity to Bromodomain and Extra-Terminal Inhibitors (BETi). Co-author DOI 10.3389/fonc.2019.00475↗
- Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. Co-author DOI 10.1038/s41467-018-04383-6↗
- E2F signature is predictive for the pancreatic adenocarcinoma clinical outcome and sensitivity to E2F inhibitors, but not for the response to cytotoxic-based t… Co-author DOI 10.1038/s41598-018-26613-z↗
- Chloroquine plays a cell-dependent role in the response to treatment of pancreatic adenocarcinoma. Co-author DOI 10.18632/oncotarget.25745↗
- Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenografts. Co-author DOI 10.1016/j.celrep.2017.11.003↗
- Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain inhibitor JQ1: implications for individua… Co-author DOI 10.15252/emmm.201606975↗
- A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. Co-author DOI 10.18632/oncotarget.10776↗
- Transcriptomic analysis predicts survival and sensitivity to anticancer drugs of patients with a pancreatic adenocarcinoma. Co-author DOI 10.1016/j.ajpath.2014.11.029↗
- Cholesterol uptake disruption, in association with chemotherapy, is a promising combined metabolic therapy for pancreatic adenocarcinoma. Co-author DOI 10.1073/pnas.1421601112↗
- A subgroup of pancreatic adenocarcinoma is sensitive to the 5-aza-dC DNA methyltransferase inhibitor. Co-author DOI 10.18632/oncotarget.2685↗
- Deciphering the cellular source of tumor relapse identifies CD44 as a major therapeutic target in pancreatic adenocarcinoma. Co-author DOI 10.18632/oncotarget.3510↗
Closest collaborators
The 25 strongest of 69 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Nelson Dusetti · 27 shared papers, 2015–2026 · strength 2.93
- Paper A comprehensive proteomic dataset of secretomes from primary pancreatic cancer cell cultures. DOI 10.1038/s41597-026-07902-z↗
- Paper The T-Box Transcription Factors TBX2 and TBX3 Are Molecular Targets of Piroctone Olamine in the Treatment of Pancreatic… DOI 10.1111/jcmm.70736↗
- Paper Pancreatic ductal adenocarcinoma ubiquitination profiling reveals specific prognostic and theranostic markers. DOI 10.1016/j.ebiom.2023.104634↗
- Juan Iovanna · 28 shared papers, 2015–2026 · strength 2.79
- Paper A comprehensive proteomic dataset of secretomes from primary pancreatic cancer cell cultures. DOI 10.1038/s41597-026-07902-z↗
- Paper Pancreatic ductal adenocarcinoma ubiquitination profiling reveals specific prognostic and theranostic markers. DOI 10.1016/j.ebiom.2023.104634↗
- Paper Induction of Apoptosis in Human Pancreatic Cancer Stem Cells by the Endoplasmic Reticulum-Targeted Alkylphospholipid An… DOI 10.3390/cancers13236124↗
- Julie Roques · 15 shared papers, 2015–2026 · strength 1.74
- Paper A comprehensive proteomic dataset of secretomes from primary pancreatic cancer cell cultures. DOI 10.1038/s41597-026-07902-z↗
- Paper Pancreatic ductal adenocarcinoma ubiquitination profiling reveals specific prognostic and theranostic markers. DOI 10.1016/j.ebiom.2023.104634↗
- Paper Inhibition of glucuronidation in pancreatic cancer improves gemcitabine anticancer activity. DOI 10.1002/cac2.12365↗
- Nicolas Alejandro Fraunhoffer · 9 shared papers, 2020–2026 · strength 1.36
- Paper A comprehensive proteomic dataset of secretomes from primary pancreatic cancer cell cultures. DOI 10.1038/s41597-026-07902-z↗
- Paper Pancreatic ductal adenocarcinoma ubiquitination profiling reveals specific prognostic and theranostic markers. DOI 10.1016/j.ebiom.2023.104634↗
- Paper Induction of Apoptosis in Human Pancreatic Cancer Stem Cells by the Endoplasmic Reticulum-Targeted Alkylphospholipid An… DOI 10.3390/cancers13236124↗
- Rémy Nicolle · 10 shared papers, 2017–2022 · strength 0.93
- Paper Multi-omics data integration and modeling unravels new mechanisms for pancreatic cancer and improves prognostic predict… DOI 10.1038/s41698-022-00299-z↗
- Paper Squamousness gain defines pancreatic ductal adenocarcinoma hepatic metastases phenotype, and gemcitabine response. DOI 10.1016/j.ejca.2021.06.038↗
- Paper Basal-like and classical cells coexist in pancreatic cancer revealed by single-cell analysis on biopsy-derived pancreat… DOI 10.1096/fj.202000363rr↗
- Abdessamad El Kaoutari · 6 shared papers, 2020–2023 · strength 0.74
- Paper Pancreatic ductal adenocarcinoma ubiquitination profiling reveals specific prognostic and theranostic markers. DOI 10.1016/j.ebiom.2023.104634↗
- Paper Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. DOI 10.1016/j.isci.2023.106899↗
- Paper Metabolomic profiling of pancreatic adenocarcinoma reveals key features driving clinical outcome and drug resistance. DOI 10.1016/j.ebiom.2021.103332↗
- Philippe Soubeyran · 5 shared papers, 2018–2026 · strength 0.73
- Paper A comprehensive proteomic dataset of secretomes from primary pancreatic cancer cell cultures. DOI 10.1038/s41597-026-07902-z↗
- Paper Pancreatic ductal adenocarcinoma ubiquitination profiling reveals specific prognostic and theranostic markers. DOI 10.1016/j.ebiom.2023.104634↗
- Paper Metabolomic profiling of pancreatic adenocarcinoma reveals key features driving clinical outcome and drug resistance. DOI 10.1016/j.ebiom.2021.103332↗
- Brice Chanez · 3 shared papers, 2022–2026 · strength 0.55
- Paper A comprehensive proteomic dataset of secretomes from primary pancreatic cancer cell cultures. DOI 10.1038/s41597-026-07902-z↗
- Paper Inhibition of glucuronidation in pancreatic cancer improves gemcitabine anticancer activity. DOI 10.1002/cac2.12365↗
- Paper Relevance of biopsy-derived pancreatic organoids in the development of efficient transcriptomic signatures to predict a… DOI 10.1016/j.tranon.2021.101315↗
- Marc F. Giovannini · 9 shared papers, 2015–2022 · strength 0.55
- Paper Relevance of biopsy-derived pancreatic organoids in the development of efficient transcriptomic signatures to predict a… DOI 10.1016/j.tranon.2021.101315↗
- Paper A transcriptomic signature to predict adjuvant gemcitabine sensitivity in pancreatic adenocarcinoma. DOI 10.1016/j.annonc.2020.10.601↗
- Paper Establishment of a pancreatic adenocarcinoma molecular gradient (PAMG) that predicts the clinical outcome of pancreatic… DOI 10.1016/j.ebiom.2020.102858↗
- Analía Meilerman Abuelafia · 4 shared papers, 2020–2022 · strength 0.54
- Paper Inhibition of glucuronidation in pancreatic cancer improves gemcitabine anticancer activity. DOI 10.1002/cac2.12365↗
- Paper Multi-omics data integration and modeling unravels new mechanisms for pancreatic cancer and improves prognostic predict… DOI 10.1038/s41698-022-00299-z↗
- Paper Squamousness gain defines pancreatic ductal adenocarcinoma hepatic metastases phenotype, and gemcitabine response. DOI 10.1016/j.ejca.2021.06.038↗
- Jerome F. Cros · 5 shared papers, 2019–2022 · strength 0.50
- Paper Squamousness gain defines pancreatic ductal adenocarcinoma hepatic metastases phenotype, and gemcitabine response. DOI 10.1016/j.ejca.2021.06.038↗
- Paper Relevance of biopsy-derived pancreatic organoids in the development of efficient transcriptomic signatures to predict a… DOI 10.1016/j.tranon.2021.101315↗
- Paper Pancreatic Cancer Organoids for Determining Sensitivity to Bromodomain and Extra-Terminal Inhibitors (BETi). DOI 10.3389/fonc.2019.00475↗
- Marine Gilabert · 9 shared papers, 2015–2021 · strength 0.47
- Paper A transcriptomic signature to predict adjuvant gemcitabine sensitivity in pancreatic adenocarcinoma. DOI 10.1016/j.annonc.2020.10.601↗
- Paper Establishment of a pancreatic adenocarcinoma molecular gradient (PAMG) that predicts the clinical outcome of pancreatic… DOI 10.1016/j.ebiom.2020.102858↗
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Pauline Duconseil · 8 shared papers, 2015–2021 · strength 0.43
- Paper A transcriptomic signature to predict adjuvant gemcitabine sensitivity in pancreatic adenocarcinoma. DOI 10.1016/j.annonc.2020.10.601↗
- Paper Establishment of a pancreatic adenocarcinoma molecular gradient (PAMG) that predicts the clinical outcome of pancreatic… DOI 10.1016/j.ebiom.2020.102858↗
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Raul A. Urrutia · 5 shared papers, 2017–2022 · strength 0.42
- Paper Multi-omics data integration and modeling unravels new mechanisms for pancreatic cancer and improves prognostic predict… DOI 10.1038/s41698-022-00299-z↗
- Paper Metabolomic profiling of pancreatic adenocarcinoma reveals key features driving clinical outcome and drug resistance. DOI 10.1016/j.ebiom.2021.103332↗
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Gwen Alyce Lomberk · 5 shared papers, 2017–2022 · strength 0.42
- Paper Multi-omics data integration and modeling unravels new mechanisms for pancreatic cancer and improves prognostic predict… DOI 10.1038/s41698-022-00299-z↗
- Paper Metabolomic profiling of pancreatic adenocarcinoma reveals key features driving clinical outcome and drug resistance. DOI 10.1016/j.ebiom.2021.103332↗
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Céline Loncle · 8 shared papers, 2015–2021 · strength 0.40
- Paper Dendrimeric nanosystem consistently circumvents heterogeneous drug response and resistance in pancreatic cancer. DOI 10.1002/exp.20210003↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Eduardo Chuluyán · 3 shared papers, 2020–2022 · strength 0.39
- Paper Inhibition of glucuronidation in pancreatic cancer improves gemcitabine anticancer activity. DOI 10.1002/cac2.12365↗
- Paper Squamousness gain defines pancreatic ductal adenocarcinoma hepatic metastases phenotype, and gemcitabine response. DOI 10.1016/j.ejca.2021.06.038↗
- Paper Evidencing a Pancreatic Ductal Adenocarcinoma Subpopulation Sensitive to the Proteasome Inhibitor Carfilzomib. DOI 10.1158/1078-0432.ccr-20-1232↗
- Benjamin Bian · 6 shared papers, 2016–2019 · strength 0.36
- Paper Pancreatic Cancer Organoids for Determining Sensitivity to Bromodomain and Extra-Terminal Inhibitors (BETi). DOI 10.3389/fonc.2019.00475↗
- Paper E2F signature is predictive for the pancreatic adenocarcinoma clinical outcome and sensitivity to E2F inhibitors, but n… DOI 10.1038/s41598-018-26613-z↗
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Rawand Masoud · 3 shared papers, 2019–2023 · strength 0.32
- Paper Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. DOI 10.1016/j.isci.2023.106899↗
- Paper Targeting Mitochondrial Complex I Overcomes Chemoresistance in High OXPHOS Pancreatic Cancer. DOI 10.1016/j.xcrm.2020.100143↗
- Paper PML hyposumoylation is responsible for the resistance of pancreatic cancer. DOI 10.1096/fj.201901091r↗
- Alice Carrier · 3 shared papers, 2019–2023 · strength 0.32
- Paper Combining the antianginal drug perhexiline with chemotherapy induces complete pancreatic cancer regression in vivo. DOI 10.1016/j.isci.2023.106899↗
- Paper Targeting Mitochondrial Complex I Overcomes Chemoresistance in High OXPHOS Pancreatic Cancer. DOI 10.1016/j.xcrm.2020.100143↗
- Paper PML hyposumoylation is responsible for the resistance of pancreatic cancer. DOI 10.1096/fj.201901091r↗
- Olivıer Turrini · 7 shared papers, 2015–2018 · strength 0.30
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Mehdi Ouaïssi · 7 shared papers, 2015–2017 · strength 0.29
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. DOI 10.18632/oncotarget.10776↗
- Sharon Prince · 1 shared paper, 2025 · strength 0.27
- Karabo Serala · 1 shared paper, 2025 · strength 0.27
- Véronique Secq · 6 shared papers, 2015–2018 · strength 0.26
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.