ResearcherCentre National de la Recherche ScientifiqueNo recent work listed
Véronique Secq
Matched on OpenAlex author ID A5011899830. Sources: OpenAlex.
Their work
- Papers
- 12
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 48
2012: 1 paper · 2013: none · 2014: none · 2015: 3 papers · 2016: 2 papers · 2017: 4 papers · 2018: 2 papers
Papers 12
- Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. Co-author DOI 10.1038/s41467-018-04383-6↗
- LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. Co-author DOI 10.1158/0008-5472.can-15-2790↗
- Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenografts. Co-author DOI 10.1016/j.celrep.2017.11.003↗
- Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. Co-author DOI 10.1038/ncomms16031↗
- BTN3A is a prognosis marker and a promising target for Vγ9Vδ2 T cells based-immunotherapy in pancreatic ductal adenocarcinoma (PDAC). Co-author DOI 10.1080/2162402x.2017.1372080↗
- Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain inhibitor JQ1: implications for individua… Co-author DOI 10.15252/emmm.201606975↗
- Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. Co-author DOI 10.1172/jci87734↗
- A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. Co-author DOI 10.18632/oncotarget.10776↗
- Transcriptomic analysis predicts survival and sensitivity to anticancer drugs of patients with a pancreatic adenocarcinoma. Co-author DOI 10.1016/j.ajpath.2014.11.029↗
- Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. Co-author DOI 10.1038/cddis.2014.557↗
- A subgroup of pancreatic adenocarcinoma is sensitive to the 5-aza-dC DNA methyltransferase inhibitor. Co-author DOI 10.18632/oncotarget.2685↗
- Homotypic cell cannibalism, a cell-death process regulated by the nuclear protein 1, opposes to metastasis in pancreatic cancer. Co-author DOI 10.1002/emmm.201201255↗
Closest collaborators
The 25 strongest of 48 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Juan Iovanna · 12 shared papers, 2012–2018 · strength 0.54
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper BTN3A is a prognosis marker and a promising target for Vγ9Vδ2 T cells based-immunotherapy in pancreatic ductal adenocar… DOI 10.1080/2162402x.2017.1372080↗
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Nelson Dusetti · 9 shared papers, 2015–2018 · strength 0.40
- Paper BTN3A is a prognosis marker and a promising target for Vγ9Vδ2 T cells based-immunotherapy in pancreatic ductal adenocar… DOI 10.1080/2162402x.2017.1372080↗
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Mehdi Ouaïssi · 8 shared papers, 2015–2018 · strength 0.35
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Céline Loncle · 8 shared papers, 2012–2017 · strength 0.33
- Paper BTN3A is a prognosis marker and a promising target for Vγ9Vδ2 T cells based-immunotherapy in pancreatic ductal adenocar… DOI 10.1080/2162402x.2017.1372080↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Olivıer Turrini · 7 shared papers, 2012–2018 · strength 0.29
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Sophie Vasseur · 6 shared papers, 2015–2018 · strength 0.28
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. DOI 10.1038/ncomms16031↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Odile Gayet · 6 shared papers, 2015–2018 · strength 0.26
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Marc F. Giovannini · 6 shared papers, 2015–2018 · strength 0.26
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Marine Gilabert · 6 shared papers, 2015–2018 · strength 0.26
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Pauline Duconseil · 5 shared papers, 2015–2018 · strength 0.21
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper A pancreatic ductal adenocarcinoma subpopulation is sensitive to FK866, an inhibitor of NAMPT. DOI 10.18632/oncotarget.10776↗
- Julie Roques · 4 shared papers, 2016–2018 · strength 0.21
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. DOI 10.1038/ncomms16031↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Fabienne Guillaumond · 4 shared papers, 2015–2018 · strength 0.20
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. DOI 10.1038/ncomms16031↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Richard Tomasini · 4 shared papers, 2015–2018 · strength 0.20
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Collagen-derived proline promotes pancreatic ductal adenocarcinoma cell survival under nutrient limited conditions. DOI 10.1038/ncomms16031↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Benjamin Bian · 4 shared papers, 2016–2018 · strength 0.19
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Jean‐Robert Delpéro · 4 shared papers, 2016–2018 · strength 0.19
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Raul A. Urrutia · 4 shared papers, 2012–2018 · strength 0.17
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Gwen Alyce Lomberk · 4 shared papers, 2012–2018 · strength 0.17
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Jean‐Luc Raoul · 4 shared papers, 2015–2017 · strength 0.17
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Transcriptomic analysis predicts survival and sensitivity to anticancer drugs of patients with a pancreatic adenocarcin… DOI 10.1016/j.ajpath.2014.11.029↗
- Christian Bressy · 3 shared papers, 2015–2018 · strength 0.15
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Paper Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. DOI 10.1038/cddis.2014.557↗
- Julie Leca · 3 shared papers, 2015–2018 · strength 0.15
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Paper Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. DOI 10.1038/cddis.2014.557↗
- Jérémy Nigri · 3 shared papers, 2015–2018 · strength 0.15
- Paper LIF Drives Neural Remodeling in Pancreatic Cancer and Offers a New Candidate Biomarker. DOI 10.1158/0008-5472.can-15-2790↗
- Paper Cancer-associated fibroblast-derived annexin A6+ extracellular vesicles support pancreatic cancer aggressiveness. DOI 10.1172/jci87734↗
- Paper Stromal SLIT2 impacts on pancreatic cancer-associated neural remodeling. DOI 10.1038/cddis.2014.557↗
- Rémy Nicolle · 3 shared papers, 2017–2018 · strength 0.14
- Paper Distinct epigenetic landscapes underlie the pathobiology of pancreatic cancer subtypes. DOI 10.1038/s41467-018-04383-6↗
- Paper Gene expression profiling of patient-derived pancreatic cancer xenografts predicts sensitivity to the BET bromodomain i… DOI 10.15252/emmm.201606975↗
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Anthony Gonçalvès · 3 shared papers, 2015–2017 · strength 0.12
- Paper Pancreatic Adenocarcinoma Therapeutic Targets Revealed by Tumor-Stroma Cross-Talk Analyses in Patient-Derived Xenograft… DOI 10.1016/j.celrep.2017.11.003↗
- Paper Transcriptomic analysis predicts survival and sensitivity to anticancer drugs of patients with a pancreatic adenocarcin… DOI 10.1016/j.ajpath.2014.11.029↗
- Paper A subgroup of pancreatic adenocarcinoma is sensitive to the 5-aza-dC DNA methyltransferase inhibitor. DOI 10.18632/oncotarget.2685↗
- Daniel Pietrasz · 2 shared papers, 2016–2018 · strength 0.11
- Jean‐Baptiste Bachet · 2 shared papers, 2016–2018 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.