ResearcherOntario Institute for Cancer ResearchActive in the last 2 years
Ashton Anthony Connor
Matched on ORCID iD 0000-0002-7596-3771; OpenAlex author ID A5012881959, A5122775193. Sources: OpenAlex.
Their work
- Papers
- 18 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 74
2015: 4 papers · 2016: 2 papers · 2017: 3 papers · 2018: 3 papers · 2019: 1 paper · 2020: 3 papers · 2021: none · 2022: 1 paper · 2023: none · 2024: none · 2025: none · 2026: 1 paper
Papers 18
- Neoadjuvant Therapy and Pancreatic Cancer Resection Outcomes: A Retrospective Analysis at a Quaternary Care Center. First author DOI 10.1016/j.jss.2026.02.012↗
- Pancreatic cancer evolution and heterogeneity: integrating omics and clinical data. Co-author DOI 10.1038/s41568-021-00418-1↗
- Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. Co-author DOI 10.1038/s41588-019-0566-9↗
- Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. Co-author DOI 10.1038/s41588-020-0588-3↗
- A Four-Chemokine Signature Is Associated with a T-cell-Inflamed Phenotype in Primary and Metastatic Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-19-2803↗
- Integration of Genomic and Transcriptional Features in Pancreatic Cancer Reveals Increased Cell Cycle Progression in Metastases. Co-author DOI 10.1016/j.ccell.2018.12.010↗
- Exome-Wide Association Study of Pancreatic Cancer Risk. Co-author DOI 10.1053/j.gastro.2017.10.015↗
- Genomics-Driven Precision Medicine for Advanced Pancreatic Cancer: Early Results from the COMPASS Trial. Co-author DOI 10.1158/1078-0432.ccr-17-2994↗
- Reflex Testing for Germline BRCA1, BRCA2, PALB2, and ATM Mutations in Pancreatic Cancer: Mutation Prevalence and Clinical Outcomes From Two Canadian Research R… Co-author DOI 10.1200/po.17.00098↗
- Association of Distinct Mutational Signatures With Correlates of Increased Immune Activity in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1001/jamaoncol.2016.3916↗
- Molecular Events in the Natural History of Pancreatic Cancer. First author DOI 10.1016/j.trecan.2017.04.005↗
- Next generation sequencing of pancreatic ductal adenocarcinoma: right or wrong? Co-author DOI 10.1080/17474124.2017.1324296↗
- Candidate DNA repair susceptibility genes identified by exome sequencing in high-risk pancreatic cancer. Co-author DOI 10.1016/j.canlet.2015.10.030↗
- A renewed model of pancreatic cancer evolution based on genomic rearrangement patterns. Co-author DOI 10.1038/nature19823↗
- Increased in vitro and in vivo sensitivity of BRCA2-associated pancreatic cancer to the poly(ADP-ribose) polymerase-1/2 inhibitor BMN 673. Co-author DOI 10.1016/j.canlet.2015.04.003↗
- Hereditary Pancreatic Cancer Syndromes. Co-author DOI 10.1016/j.soc.2015.06.007↗
- Ductal pancreatic cancer modeling and drug screening using human pluripotent stem cell- and patient-derived tumor organoids. Co-author DOI 10.1038/nm.3973↗
- Prevalence of germline mutations in cancer predisposition genes in patients with pancreatic cancer. Co-author DOI 10.1053/j.gastro.2014.11.042↗
Closest collaborators
The 25 strongest of 74 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Steve Gallinger · 17 shared papers, 2015–2022 · strength 1.39
- Paper Pancreatic cancer evolution and heterogeneity: integrating omics and clinical data. DOI 10.1038/s41568-021-00418-1↗
- Paper Next generation sequencing of pancreatic ductal adenocarcinoma: right or wrong? DOI 10.1080/17474124.2017.1324296↗
- Paper Molecular Events in the Natural History of Pancreatic Cancer. DOI 10.1016/j.trecan.2017.04.005↗
- George Zogopoulos · 9 shared papers, 2015–2020 · strength 0.51
- Paper A Four-Chemokine Signature Is Associated with a T-cell-Inflamed Phenotype in Primary and Metastatic Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-19-2803↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Robert C. Grant · 8 shared papers, 2015–2020 · strength 0.45
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Paper Exome-Wide Association Study of Pancreatic Cancer Risk. DOI 10.1053/j.gastro.2017.10.015↗
- Ayelet Borgida · 8 shared papers, 2015–2020 · strength 0.44
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Paper Exome-Wide Association Study of Pancreatic Cancer Risk. DOI 10.1053/j.gastro.2017.10.015↗
- Faiyaz Notta · 7 shared papers, 2016–2020 · strength 0.41
- Paper A Four-Chemokine Signature Is Associated with a T-cell-Inflamed Phenotype in Primary and Metastatic Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-19-2803↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Sandra Elisabeth Fischer · 5 shared papers, 2018–2020 · strength 0.33
- Paper A Four-Chemokine Signature Is Associated with a T-cell-Inflamed Phenotype in Primary and Metastatic Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-19-2803↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Grainne Mary O'Kane · 5 shared papers, 2018–2020 · strength 0.33
- Paper A Four-Chemokine Signature Is Associated with a T-cell-Inflamed Phenotype in Primary and Metastatic Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-19-2803↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Jennifer Jane Knox · 5 shared papers, 2018–2020 · strength 0.33
- Paper A Four-Chemokine Signature Is Associated with a T-cell-Inflamed Phenotype in Primary and Metastatic Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-19-2803↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Michelle Chan‐Seng‐Yue · 6 shared papers, 2016–2020 · strength 0.32
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Paper Integration of Genomic and Transcriptional Features in Pancreatic Cancer Reveals Increased Cell Cycle Progression in Me… DOI 10.1016/j.ccell.2018.12.010↗
- Sean Patrick Cleary · 6 shared papers, 2015–2019 · strength 0.28
- Paper Exome-Wide Association Study of Pancreatic Cancer Risk. DOI 10.1053/j.gastro.2017.10.015↗
- Paper Integration of Genomic and Transcriptional Features in Pancreatic Cancer Reveals Increased Cell Cycle Progression in Me… DOI 10.1016/j.ccell.2018.12.010↗
- Paper Candidate DNA repair susceptibility genes identified by exome sequencing in high-risk pancreatic cancer. DOI 10.1016/j.canlet.2015.10.030↗
- Alyssa L. Smith · 5 shared papers, 2015–2019 · strength 0.26
- Paper Exome-Wide Association Study of Pancreatic Cancer Risk. DOI 10.1053/j.gastro.2017.10.015↗
- Paper Integration of Genomic and Transcriptional Features in Pancreatic Cancer Reveals Increased Cell Cycle Progression in Me… DOI 10.1016/j.ccell.2018.12.010↗
- Paper Reflex Testing for Germline BRCA1, BRCA2, PALB2, and ATM Mutations in Pancreatic Cancer: Mutation Prevalence and Clinic… DOI 10.1200/po.17.00098↗
- Gloria M. Petersen · 5 shared papers, 2015–2019 · strength 0.23
- Paper Integration of Genomic and Transcriptional Features in Pancreatic Cancer Reveals Increased Cell Cycle Progression in Me… DOI 10.1016/j.ccell.2018.12.010↗
- Paper Prevalence of germline mutations in cancer predisposition genes in patients with pancreatic cancer. DOI 10.1053/j.gastro.2014.11.042↗
- Paper Candidate DNA repair susceptibility genes identified by exome sequencing in high-risk pancreatic cancer. DOI 10.1016/j.canlet.2015.10.030↗
- Kelvin Allenson · 1 shared paper, 2026 · strength 0.23
- Nestor F. Esnaola · 1 shared paper, 2026 · strength 0.23
- Ashish Saharia · 1 shared paper, 2026 · strength 0.23
- Ahmed Elaileh · 1 shared paper, 2026 · strength 0.23
- Ahmed Osama Gaber · 1 shared paper, 2026 · strength 0.23
- Maen Abdelrahim · 1 shared paper, 2026 · strength 0.23
- Kirk Heyne · 1 shared paper, 2026 · strength 0.23
- Youssef E. Dib · 1 shared paper, 2026 · strength 0.23
- Spring Holter · 4 shared papers, 2015–2018 · strength 0.20
- Paper Reflex Testing for Germline BRCA1, BRCA2, PALB2, and ATM Mutations in Pancreatic Cancer: Mutation Prevalence and Clinic… DOI 10.1200/po.17.00098↗
- Paper Genomics-Driven Precision Medicine for Advanced Pancreatic Cancer: Early Results from the COMPASS Trial. DOI 10.1158/1078-0432.ccr-17-2994↗
- Paper Prevalence of germline mutations in cancer predisposition genes in patients with pancreatic cancer. DOI 10.1053/j.gastro.2014.11.042↗
- Sangeetha N. Kalimuthu · 3 shared papers, 2017–2020 · strength 0.18
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Paper Association of Distinct Mutational Signatures With Correlates of Increased Immune Activity in Pancreatic Ductal Adenoca… DOI 10.1001/jamaoncol.2016.3916↗
- David F. Schaeffer · 3 shared papers, 2015–2020 · strength 0.18
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Paper Ductal pancreatic cancer modeling and drug screening using human pluripotent stem cell- and patient-derived tumor organ… DOI 10.1038/nm.3973↗
- Daniel John Renouf · 3 shared papers, 2015–2020 · strength 0.18
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Paper Ductal pancreatic cancer modeling and drug screening using human pluripotent stem cell- and patient-derived tumor organ… DOI 10.1038/nm.3973↗
- Michael H. A. Roehrl · 4 shared papers, 2015–2019 · strength 0.17
- Paper Integration of Genomic and Transcriptional Features in Pancreatic Cancer Reveals Increased Cell Cycle Progression in Me… DOI 10.1016/j.ccell.2018.12.010↗
- Paper Association of Distinct Mutational Signatures With Correlates of Increased Immune Activity in Pancreatic Ductal Adenoca… DOI 10.1001/jamaoncol.2016.3916↗
- Paper Ductal pancreatic cancer modeling and drug screening using human pluripotent stem cell- and patient-derived tumor organ… DOI 10.1038/nm.3973↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.