ResearcherOntario Institute for Cancer ResearchActive in the last 2 years
Michelle Chan‐Seng‐Yue
Matched on ORCID iD 0000-0003-3773-9354; OpenAlex author ID A5031785087. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 66
2016: 2 papers · 2017: 1 paper · 2018: none · 2019: 1 paper · 2020: 2 papers · 2021: none · 2022: none · 2023: none · 2024: none · 2025: 4 papers · 2026: 2 papers
Papers 12
- Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic Cancer Co-author DOI 10.64898/2026.04.07.717011↗
- Extra-lineage tissue programs define the transcription states of human pancreatic cancer Co-author DOI 10.64898/2026.04.29.721655↗
- Whole genome and transcriptome profiling in advanced pancreatic cancer patients on the COMPASS trial. Co-author DOI 10.1038/s41467-025-60808-z↗
- The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. Co-author DOI 10.1038/s41698-025-00888-8↗
- Integrated spatial proteomics of human PDAC uncovers an expanded tumour-immune-stroma spectrum with genomic associations Co-author DOI 10.1101/2025.08.15.670565↗
- The subclonal footprint of pervasive early dissemination in pancreatic cancer First author DOI 10.1101/2025.11.27.690823↗
- Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. Co-author DOI 10.1038/s41588-019-0566-9↗
- Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. Co-author DOI 10.1038/s41588-020-0588-3↗
- Integration of Genomic and Transcriptional Features in Pancreatic Cancer Reveals Increased Cell Cycle Progression in Metastases. Co-author DOI 10.1016/j.ccell.2018.12.010↗
- Association of Distinct Mutational Signatures With Correlates of Increased Immune Activity in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1001/jamaoncol.2016.3916↗
- Candidate DNA repair susceptibility genes identified by exome sequencing in high-risk pancreatic cancer. Co-author DOI 10.1016/j.canlet.2015.10.030↗
- A renewed model of pancreatic cancer evolution based on genomic rearrangement patterns. First author DOI 10.1038/nature19823↗
Closest collaborators
The 25 strongest of 66 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Steve Gallinger · 12 shared papers, 2016–2026 · strength 1.38
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Robert C. Grant · 11 shared papers, 2016–2026 · strength 1.35
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Faiyaz Notta · 11 shared papers, 2016–2026 · strength 1.34
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Grainne Mary O'Kane · 9 shared papers, 2019–2026 · strength 1.26
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Jennifer Jane Knox · 9 shared papers, 2019–2026 · strength 1.26
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Erica S. Tsang · 5 shared papers, 2025–2026 · strength 0.90
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Ayelet Borgida · 8 shared papers, 2016–2026 · strength 0.80
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Paper Integrated spatial proteomics of human PDAC uncovers an expanded tumour-immune-stroma spectrum with genomic associations DOI 10.1101/2025.08.15.670565↗
- George Zogopoulos · 8 shared papers, 2016–2025 · strength 0.78
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Whole genome and transcriptome profiling in advanced pancreatic cancer patients on the COMPASS trial. DOI 10.1038/s41467-025-60808-z↗
- Spring Holter · 5 shared papers, 2016–2026 · strength 0.73
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Eugenia Flores Figueroa · 5 shared papers, 2020–2026 · strength 0.71
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Barbara T. Grünwald · 4 shared papers, 2025–2026 · strength 0.70
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Paper Integrated spatial proteomics of human PDAC uncovers an expanded tumour-immune-stroma spectrum with genomic associations DOI 10.1101/2025.08.15.670565↗
- Yuanchang Fang · 3 shared papers, 2025–2026 · strength 0.57
- Paper Transcriptomic, Genomic, and Clinical Characterization of Morphological Classes in Localized and Metastatic Pancreatic … DOI 10.64898/2026.04.07.717011↗
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper The subclonal footprint of pervasive early dissemination in pancreatic cancer DOI 10.1101/2025.11.27.690823↗
- Sandra Elisabeth Fischer · 5 shared papers, 2019–2025 · strength 0.52
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Whole genome and transcriptome profiling in advanced pancreatic cancer patients on the COMPASS trial. DOI 10.1038/s41467-025-60808-z↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Malcolm J. Moore · 3 shared papers, 2017–2025 · strength 0.37
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Whole genome and transcriptome profiling in advanced pancreatic cancer patients on the COMPASS trial. DOI 10.1038/s41467-025-60808-z↗
- Paper Association of Distinct Mutational Signatures With Correlates of Increased Immune Activity in Pancreatic Ductal Adenoca… DOI 10.1001/jamaoncol.2016.3916↗
- Hartland Warren Jackson · 2 shared papers, 2025–2026 · strength 0.35
- Sangeetha N. Kalimuthu · 4 shared papers, 2017–2025 · strength 0.34
- Paper Whole genome and transcriptome profiling in advanced pancreatic cancer patients on the COMPASS trial. DOI 10.1038/s41467-025-60808-z↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Yifan Wang · 2 shared papers, 2025 · strength 0.33
- David Arthur Tuveson · 3 shared papers, 2020–2026 · strength 0.33
- Paper Extra-lineage tissue programs define the transcription states of human pancreatic cancer DOI 10.64898/2026.04.29.721655↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Raymond Jang · 2 shared papers, 2025 · strength 0.33
- Kyaw Lwin Aung · 2 shared papers, 2025 · strength 0.33
- Ashton Anthony Connor · 6 shared papers, 2016–2020 · strength 0.32
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Paper Integration of Genomic and Transcriptional Features in Pancreatic Cancer Reveals Increased Cell Cycle Progression in Me… DOI 10.1016/j.ccell.2018.12.010↗
- Joanna M. Karasinska · 3 shared papers, 2020–2025 · strength 0.31
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- Janessa J. Laskin · 3 shared papers, 2020–2025 · strength 0.31
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- James Thomas Topham · 3 shared papers, 2020–2025 · strength 0.31
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
- David F. Schaeffer · 3 shared papers, 2020–2025 · strength 0.31
- Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
- Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
- Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.