Janessa J. Laskin

Closest collaborators

The 25 strongest of 44 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

1. David F. Schaeffer · 13 shared papers, 2018–2026 · strength 1.34
   • Paper Parent-of-Origin-Aware Genomic Analysis to Enhance Cascade Testing in Pancreatic Cancer: A Population-Based Study DOI 10.64898/2026.10.30.26366356↗
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
2. Daniel John Renouf · 13 shared papers, 2018–2026 · strength 1.34
   • Paper Parent-of-Origin-Aware Genomic Analysis to Enhance Cascade Testing in Pancreatic Cancer: A Population-Based Study DOI 10.64898/2026.10.30.26366356↗
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
3. Joanna M. Karasinska · 12 shared papers, 2018–2025 · strength 1.16
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
   • Paper Tumor infiltrating neutrophils and gland formation predict overall survival and molecular subgroups in pancreatic ducta… DOI 10.1002/cam4.3695↗
4. James Thomas Topham · 10 shared papers, 2019–2025 · strength 1.04
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
   • Paper Tumor infiltrating neutrophils and gland formation predict overall survival and molecular subgroups in pancreatic ducta… DOI 10.1002/cam4.3695↗
5. Jennifer Jane Knox · 9 shared papers, 2019–2025 · strength 0.93
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
   • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
6. Steve Gallinger · 9 shared papers, 2019–2025 · strength 0.93
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
   • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
7. Grainne Mary O'Kane · 8 shared papers, 2020–2025 · strength 0.86
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
   • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
8. Faiyaz Notta · 8 shared papers, 2020–2025 · strength 0.86
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
   • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
9. Steve E. Kalloger · 8 shared papers, 2018–2025 · strength 0.79
   • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
   • Paper Tumor infiltrating neutrophils and gland formation predict overall survival and molecular subgroups in pancreatic ducta… DOI 10.1002/cam4.3695↗
   • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
10. Rachel Anne Goodwin · 5 shared papers, 2021–2025 · strength 0.64
    • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
    • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
11. Oliver F. Bathe · 5 shared papers, 2021–2025 · strength 0.64
    • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
    • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
12. Hui‐Li Wong · 7 shared papers, 2018–2022 · strength 0.58
    • Paper Tumor infiltrating neutrophils and gland formation predict overall survival and molecular subgroups in pancreatic ducta… DOI 10.1002/cam4.3695↗
    • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
    • Paper Subtype-Discordant Pancreatic Ductal Adenocarcinoma Tumors Show Intermediate Clinical and Molecular Characteristics. DOI 10.1158/1078-0432.ccr-20-2831↗
13. Erica S. Tsang · 4 shared papers, 2021–2025 · strength 0.46
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
    • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
    • Paper Delving into Early-onset Pancreatic Ductal Adenocarcinoma: How Does Age Fit In? DOI 10.1158/1078-0432.ccr-20-1042↗
14. Michael K.C. Lee · 5 shared papers, 2019–2021 · strength 0.45
    • Paper Tumor infiltrating neutrophils and gland formation predict overall survival and molecular subgroups in pancreatic ducta… DOI 10.1002/cam4.3695↗
    • Paper Delving into Early-onset Pancreatic Ductal Adenocarcinoma: How Does Age Fit In? DOI 10.1158/1078-0432.ccr-20-1042↗
    • Paper Subtype-Discordant Pancreatic Ductal Adenocarcinoma Tumors Show Intermediate Clinical and Molecular Characteristics. DOI 10.1158/1078-0432.ccr-20-2831↗
15. Malcolm J. Moore · 3 shared papers, 2019–2025 · strength 0.32
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
    • Paper Altered Gene Expression along the Glycolysis-Cholesterol Synthesis Axis Is Associated with Outcome in Pancreatic Cancer. DOI 10.1158/1078-0432.ccr-19-1543↗
    • Paper NRG1 Gene Fusions Are Recurrent, Clinically Actionable Gene Rearrangements in KRAS Wild-Type Pancreatic Ductal Adenocar… DOI 10.1158/1078-0432.ccr-19-0191↗
16. Sandra Elisabeth Fischer · 3 shared papers, 2020–2025 · strength 0.31
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
    • Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
    • Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
17. Robert C. Grant · 3 shared papers, 2020–2025 · strength 0.31
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
    • Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
    • Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
18. Michelle Chan‐Seng‐Yue · 3 shared papers, 2020–2025 · strength 0.31
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
    • Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
    • Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
19. George Zogopoulos · 3 shared papers, 2020–2025 · strength 0.31
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗
    • Paper Author Correction: Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-020-0588-3↗
    • Paper Transcription phenotypes of pancreatic cancer are driven by genomic events during tumor evolution. DOI 10.1038/s41588-019-0566-9↗
20. Kasmintan A. Schrader · 3 shared papers, 2018–2026 · strength 0.30
    • Paper Parent-of-Origin-Aware Genomic Analysis to Enhance Cascade Testing in Pancreatic Cancer: A Population-Based Study DOI 10.64898/2026.10.30.26366356↗
    • Paper Temporal Dynamics of Genomic Alterations in a BRCA1 Germline-Mutated Pancreatic Cancer With Low Genomic Instability Bur… DOI 10.1200/po.18.00057↗
    • Paper Base excision repair deficiency signatures implicate germline and somatic MUTYH aberrations in pancreatic ductal adenoc… DOI 10.1101/mcs.a003681↗
21. Hassan A. Ali · 2 shared papers, 2022–2025 · strength 0.28
    • Paper Multi-omics whole-genome characterization of the copy number landscape of metastatic pancreatic ductal adenocarcinoma. DOI 10.1016/j.isci.2025.113176↗
    • Paper Integrative analysis of KRAS wildtype metastatic pancreatic ductal adenocarcinoma reveals mutation and expression-based… DOI 10.1038/s41467-022-33718-7↗
22. Carol Cremin · 2 shared papers, 2019–2026 · strength 0.24
    • Paper Parent-of-Origin-Aware Genomic Analysis to Enhance Cascade Testing in Pancreatic Cancer: A Population-Based Study DOI 10.64898/2026.10.30.26366356↗
    • Paper Base excision repair deficiency signatures implicate germline and somatic MUTYH aberrations in pancreatic ductal adenoc… DOI 10.1101/mcs.a003681↗
23. Martin R. Jones · 3 shared papers, 2018–2019 · strength 0.19
    • Paper NRG1 Gene Fusions Are Recurrent, Clinically Actionable Gene Rearrangements in KRAS Wild-Type Pancreatic Ductal Adenocar… DOI 10.1158/1078-0432.ccr-19-0191↗
    • Paper Temporal Dynamics of Genomic Alterations in a BRCA1 Germline-Mutated Pancreatic Cancer With Low Genomic Instability Bur… DOI 10.1200/po.18.00057↗
    • Paper Base excision repair deficiency signatures implicate germline and somatic MUTYH aberrations in pancreatic ductal adenoc… DOI 10.1101/mcs.a003681↗
24. Pheobe TM Cheng · 1 shared paper, 2026 · strength 0.18
    • Paper Parent-of-Origin-Aware Genomic Analysis to Enhance Cascade Testing in Pancreatic Cancer: A Population-Based Study DOI 10.64898/2026.10.30.26366356↗
25. Spring Holter · 1 shared paper, 2025 · strength 0.17
    • Paper The tandem duplicator phenotype may be a novel targetable subgroup in pancreatic cancer. DOI 10.1038/s41698-025-00888-8↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.