ResearcherHospital Universitario Fundación Jiménez DíazActive in the last 2 years
Carlos Lacalle-González
Matched on ORCID iD 0000-0002-7690-399X; OpenAlex author ID A5045805543. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 7
2023: 2 papers · 2024: 1 paper · 2025: 1 paper · 2026: 1 paper
Papers 5
- Targeting pancreatic cancer with natural compounds: molecular mechanisms, preclinical evidence, clinical perspectives, and limitations. Co-author DOI 10.3389/fphar.2026.1868595↗
- FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreatic Cancer Co-author DOI 10.1101/2025.09.12.675778↗
- Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. First author DOI 10.3390/antiox13111405↗
- DLL3 Is a Prognostic and Potentially Predictive Biomarker for Immunotherapy Linked to PD/PD-L Axis and NOTCH1 in Pancreatic Cancer. Co-author DOI 10.3390/biomedicines11102812↗
- Iron-Dependent Cell Death: A New Treatment Approach against Pancreatic Ductal Adenocarcinoma. First author DOI 10.3390/ijms241914979↗
Closest collaborators
The 7 strongest of 7 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jesús García‐Foncillas López · 5 shared papers, 2023–2026 · strength 1.25
- Paper Targeting pancreatic cancer with natural compounds: molecular mechanisms, preclinical evidence, clinical perspectives, … DOI 10.3389/fphar.2026.1868595↗
- Paper Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. DOI 10.3390/antiox13111405↗
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- Javier Martínez‐Useros · 5 shared papers, 2023–2026 · strength 1.25
- Paper Targeting pancreatic cancer with natural compounds: molecular mechanisms, preclinical evidence, clinical perspectives, … DOI 10.3389/fphar.2026.1868595↗
- Paper Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. DOI 10.3390/antiox13111405↗
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- Michael Ochieng’ Otieno · 4 shared papers, 2023–2025 · strength 0.92
- Paper Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. DOI 10.3390/antiox13111405↗
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- Paper Iron-Dependent Cell Death: A New Treatment Approach against Pancreatic Ductal Adenocarcinoma. DOI 10.3390/ijms241914979↗
- Carlos Lopez-Blazquez · 3 shared papers, 2023–2025 · strength 0.73
- Paper Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. DOI 10.3390/antiox13111405↗
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- Paper Iron-Dependent Cell Death: A New Treatment Approach against Pancreatic Ductal Adenocarcinoma. DOI 10.3390/ijms241914979↗
- María Jesús Fernández‐Aceñero · 2 shared papers, 2023–2025 · strength 0.44
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- Paper DLL3 Is a Prognostic and Potentially Predictive Biomarker for Immunotherapy Linked to PD/PD-L Axis and NOTCH1 in Pancre… DOI 10.3390/biomedicines11102812↗
- Alberto Orta · 1 shared paper, 2026 · strength 0.33
- Paula Iglesias-Matesanz · 1 shared paper, 2024 · strength 0.26
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.