ResearcherHospital Universitario Fundación Jiménez DíazActive in the last 2 years
Jesús García‐Foncillas
Matched on ORCID iD 0000-0002-7591-8006; OpenAlex author ID A5028200237. Sources: OpenAlex.
Their work
- Papers
- 18 (first author 2, last author 7)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 21
2014: 1 paper · 2015: none · 2016: 2 papers · 2017: 2 papers · 2018: 1 paper · 2019: 2 papers · 2020: 1 paper · 2021: 1 paper · 2022: 1 paper · 2023: 2 papers · 2024: 2 papers · 2025: 2 papers · 2026: 1 paper
Papers 18
- Targeting pancreatic cancer with natural compounds: molecular mechanisms, preclinical evidence, clinical perspectives, and limitations. Co-author DOI 10.3389/fphar.2026.1868595↗
- The crosstalk within tumor microenvironment and exosomes in pancreatic cancer. Co-author DOI 10.1016/j.bbcan.2025.189308↗
- FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreatic Cancer Co-author DOI 10.1101/2025.09.12.675778↗
- Vitamin-C-dependent downregulation of the citrate metabolism pathway potentiates pancreatic ductal adenocarcinoma growth arrest. Co-author DOI 10.1002/1878-0261.13616↗
- Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. Co-author DOI 10.3390/antiox13111405↗
- DLL3 Is a Prognostic and Potentially Predictive Biomarker for Immunotherapy Linked to PD/PD-L Axis and NOTCH1 in Pancreatic Cancer. Co-author DOI 10.3390/biomedicines11102812↗
- Iron-Dependent Cell Death: A New Treatment Approach against Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3390/ijms241914979↗
- A Novel PiRNA Enhances CA19-9 Sensitivity for Pancreatic Cancer Identification by Liquid Biopsy. Co-author DOI 10.3390/jcm11247310↗
- The Match between Molecular Subtypes, Histology and Microenvironment of Pancreatic Cancer and Its Relevance for Chemoresistance. Last author DOI 10.3390/cancers13020322↗
- The Clinical Significance of PIWIL3 and PIWIL4 Expression in Pancreatic Cancer. Last author DOI 10.3390/jcm9051252↗
- Clinical Implications of NRAS Overexpression in Resectable Pancreatic Adenocarcinoma Patients. Last author DOI 10.1007/s12253-017-0341-0↗
- The Prognosis Value of PIWIL1 and PIWIL2 Expression in Pancreatic Cancer. Last author DOI 10.3390/jcm8091275↗
- New Hope for Pancreatic Ductal Adenocarcinoma Treatment Targeting Endoplasmic Reticulum Stress Response: A Systematic Review. Last author DOI 10.3390/ijms19092468↗
- UNR/CDSE1 expression as prognosis biomarker in resectable pancreatic ductal adenocarcinoma patients: A proof-of-concept. Last author DOI 10.1371/journal.pone.0182044↗
- Oxidative Stress: A New Target for Pancreatic Cancer Prognosis and Treatment. Last author DOI 10.3390/jcm6030029↗
- The Role of BRCA2 Mutation Status as Diagnostic, Predictive, and Prognosis Biomarker for Pancreatic Cancer. First author DOI 10.1155/2016/1869304↗
- Can Molecular Biomarkers Change the Paradigm of Pancreatic Cancer Prognosis? First author DOI 10.1155/2016/4873089↗
- TTD consensus document on the diagnosis and management of exocrine pancreatic cancer. Co-author DOI 10.1007/s12094-014-1177-7↗
Closest collaborators
The 21 strongest of 21 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Javier Martínez‐Useros · 16 shared papers, 2016–2026 · strength 2.79
- Paper The crosstalk within tumor microenvironment and exosomes in pancreatic cancer. DOI 10.1016/j.bbcan.2025.189308↗
- Paper Targeting pancreatic cancer with natural compounds: molecular mechanisms, preclinical evidence, clinical perspectives, … DOI 10.3389/fphar.2026.1868595↗
- Paper Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. DOI 10.3390/antiox13111405↗
- Michael Ochieng’ Otieno · 5 shared papers, 2023–2025 · strength 1.28
- Paper The crosstalk within tumor microenvironment and exosomes in pancreatic cancer. DOI 10.1016/j.bbcan.2025.189308↗
- Paper Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. DOI 10.3390/antiox13111405↗
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- Carlos Lacalle-González · 5 shared papers, 2023–2026 · strength 1.25
- Paper Targeting pancreatic cancer with natural compounds: molecular mechanisms, preclinical evidence, clinical perspectives, … DOI 10.3389/fphar.2026.1868595↗
- Paper Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. DOI 10.3390/antiox13111405↗
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- María Jesús Fernández‐Aceñero · 7 shared papers, 2017–2025 · strength 0.97
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- Paper DLL3 Is a Prognostic and Potentially Predictive Biomarker for Immunotherapy Linked to PD/PD-L Axis and NOTCH1 in Pancre… DOI 10.3390/biomedicines11102812↗
- Paper Vitamin-C-dependent downregulation of the citrate metabolism pathway potentiates pancreatic ductal adenocarcinoma growt… DOI 10.1002/1878-0261.13616↗
- Carlos Lopez-Blazquez · 3 shared papers, 2023–2025 · strength 0.73
- Paper Glutathione Peroxidases: An Emerging and Promising Therapeutic Target for Pancreatic Cancer Treatment. DOI 10.3390/antiox13111405↗
- Paper FOLFIRINOX Combined with GPX4 Inhibition Induces Ferroptosis and Defines Redox-Based Therapeutic Subgroups in Pancreati… DOI 10.1101/2025.09.12.675778↗
- Paper Iron-Dependent Cell Death: A New Treatment Approach against Pancreatic Ductal Adenocarcinoma. DOI 10.3390/ijms241914979↗
- Weiyao Li · 7 shared papers, 2017–2022 · strength 0.71
- Paper A Novel PiRNA Enhances CA19-9 Sensitivity for Pancreatic Cancer Identification by Liquid Biopsy. DOI 10.3390/jcm11247310↗
- Paper New Hope for Pancreatic Ductal Adenocarcinoma Treatment Targeting Endoplasmic Reticulum Stress Response: A Systematic R… DOI 10.3390/ijms19092468↗
- Paper The Clinical Significance of PIWIL3 and PIWIL4 Expression in Pancreatic Cancer. DOI 10.3390/jcm9051252↗
- Alberto Orta · 3 shared papers, 2020–2026 · strength 0.58
- Paper Targeting pancreatic cancer with natural compounds: molecular mechanisms, preclinical evidence, clinical perspectives, … DOI 10.3389/fphar.2026.1868595↗
- Paper A Novel PiRNA Enhances CA19-9 Sensitivity for Pancreatic Cancer Identification by Liquid Biopsy. DOI 10.3390/jcm11247310↗
- Paper The Clinical Significance of PIWIL3 and PIWIL4 Expression in Pancreatic Cancer. DOI 10.3390/jcm9051252↗
- Nuria Garcia-Carbonero · 4 shared papers, 2018–2022 · strength 0.45
- Paper A Novel PiRNA Enhances CA19-9 Sensitivity for Pancreatic Cancer Identification by Liquid Biopsy. DOI 10.3390/jcm11247310↗
- Paper New Hope for Pancreatic Ductal Adenocarcinoma Treatment Targeting Endoplasmic Reticulum Stress Response: A Systematic R… DOI 10.3390/ijms19092468↗
- Paper The Clinical Significance of PIWIL3 and PIWIL4 Expression in Pancreatic Cancer. DOI 10.3390/jcm9051252↗
- Tomasz Powrózek · 1 shared paper, 2025 · strength 0.37
- Paula Iglesias-Matesanz · 1 shared paper, 2024 · strength 0.26
- Aiora Cenigaonandia‐Campillo · 1 shared paper, 2024 · strength 0.17
- Óscar Aguilera · 1 shared paper, 2024 · strength 0.17
- Roberto Pazo-Cid · 1 shared paper, 2022 · strength 0.14
- Fernando Rivera · 1 shared paper, 2014 · strength 0.04
- Josep M. Tabernero · 1 shared paper, 2014 · strength 0.04
- Carles Pericay · 1 shared paper, 2014 · strength 0.04
- Berta Laquente · 1 shared paper, 2014 · strength 0.04
- Manuel Benavides · 1 shared paper, 2014 · strength 0.04
- Alfredo Carrato · 1 shared paper, 2014 · strength 0.04
- Javier Gallego-Plazas · 1 shared paper, 2014 · strength 0.04
- Enrique Aranda · 1 shared paper, 2014 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.