ResearcherGerman Cancer Research CenterActive in the last 2 years
Barbara M. Grüner
Matched on ORCID iD 0000-0003-0974-4826; OpenAlex author ID A5025856946. Sources: funder_awards.
Their work
- Papers
- 8 (first author 0, last author 3)
- Trials
- 0
- Grants
- 1
- Labs led
- 1
- Collaborators
- 36
2012: 1 paper · 2013: none · 2014: 1 grant · 2015: 1 paper · 2016: 1 paper · 2017: 1 paper · 2018: 1 paper · 2019: none · 2020: none · 2021: 1 paper · 2022: none · 2023: none · 2024: none · 2025: 1 paper · 2026: 1 paper
Papers 8
- Loss of the Y chromosome drives basal identity and metastatic potential via epigenetic reprogramming in pancreatic cancer Last author DOI 10.64898/2026.10.06.756936↗
- PLK1 blockade enhances the anti-tumor effect of MAPK inhibition in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.celrep.2025.115541↗
- Unraveling Tumor Heterogeneity by Using DNA Barcoding Technologies to Develop Personalized Treatment Strategies in Advanced-Stage PDAC. Last author DOI 10.3390/cancers13164187↗
- Hmga2 is dispensable for pancreatic cancer development, metastasis, and therapy resistance. Last author DOI 10.1038/s41598-018-32159-x↗
- BLIMP1 Induces Transient Metastatic Heterogeneity in Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-17-0250↗
- Modeling Therapy Response and Spatial Tissue Distribution of Erlotinib in Pancreatic Cancer. Co-author DOI 10.1158/1535-7163.mct-15-0165↗
- Pancreatic cancer modeling using retrograde viral vector delivery and in vivo CRISPR/Cas9-mediated somatic genome editing. Co-author DOI 10.1101/gad.264861.115↗
- MALDI imaging mass spectrometry for in situ proteomic analysis of preneoplastic lesions in pancreatic cancer. Co-author DOI 10.1371/journal.pone.0039424↗
Grants 1
Closest collaborators
The 25 strongest of 36 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Madeleine Dorsch · 3 shared papers, 2018–2026 · strength 0.48
- Paper PLK1 blockade enhances the anti-tumor effect of MAPK inhibition in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.115541↗
- Paper Loss of the Y chromosome drives basal identity and metastatic potential via epigenetic reprogramming in pancreatic canc… DOI 10.64898/2026.10.06.756936↗
- Paper Hmga2 is dispensable for pancreatic cancer development, metastasis, and therapy resistance. DOI 10.1038/s41598-018-32159-x↗
- Jens Thomas Siveke · 4 shared papers, 2012–2026 · strength 0.47
- Paper PLK1 blockade enhances the anti-tumor effect of MAPK inhibition in pancreatic ductal adenocarcinoma. DOI 10.1016/j.celrep.2025.115541↗
- Paper Loss of the Y chromosome drives basal identity and metastatic potential via epigenetic reprogramming in pancreatic canc… DOI 10.64898/2026.10.06.756936↗
- Paper Modeling Therapy Response and Spatial Tissue Distribution of Erlotinib in Pancreatic Cancer. DOI 10.1158/1535-7163.mct-15-0165↗
- Laura Godfrey · 2 shared papers, 2025–2026 · strength 0.40
- Marija Trajkovic‐Arsic · 2 shared papers, 2012–2025 · strength 0.23
- Rui Fang · 1 shared paper, 2025 · strength 0.20
- Hendrik Schürmann · 1 shared paper, 2025 · strength 0.20
- Stephan A. Hahn · 1 shared paper, 2025 · strength 0.20
- Ben Zhao · 1 shared paper, 2025 · strength 0.20
- Smiths Lueong · 1 shared paper, 2025 · strength 0.20
- Barbara T. Grünwald · 1 shared paper, 2026 · strength 0.20
- Kazeera Aliar · 1 shared paper, 2026 · strength 0.20
- Günter Schneider · 1 shared paper, 2026 · strength 0.20
- John P. Neoptolemos · 1 shared paper, 2026 · strength 0.20
- Elisabeth Heßmann · 1 shared paper, 2026 · strength 0.20
- Faiyaz Notta · 1 shared paper, 2026 · strength 0.20
- Philip Dujardin · 1 shared paper, 2021 · strength 0.19
- Shin‐Heng Chiou · 3 shared papers, 2015–2018 · strength 0.18
- Paper Hmga2 is dispensable for pancreatic cancer development, metastasis, and therapy resistance. DOI 10.1038/s41598-018-32159-x↗
- Paper BLIMP1 Induces Transient Metastatic Heterogeneity in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-17-0250↗
- Paper Pancreatic cancer modeling using retrograde viral vector delivery and in vivo CRISPR/Cas9-mediated somatic genome editi… DOI 10.1101/gad.264861.115↗
- Monte Meier Winslow · 3 shared papers, 2015–2018 · strength 0.18
- Paper Hmga2 is dispensable for pancreatic cancer development, metastasis, and therapy resistance. DOI 10.1038/s41598-018-32159-x↗
- Paper BLIMP1 Induces Transient Metastatic Heterogeneity in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-17-0250↗
- Paper Pancreatic cancer modeling using retrograde viral vector delivery and in vivo CRISPR/Cas9-mediated somatic genome editi… DOI 10.1101/gad.264861.115↗
- Albert C. Koong · 2 shared papers, 2017–2018 · strength 0.14
- Margaret M. Kozak · 2 shared papers, 2017–2018 · strength 0.14
- Grace Kim · 2 shared papers, 2015–2017 · strength 0.10
- Axel Karl Walch · 2 shared papers, 2012–2016 · strength 0.08
- Roland Michael Schmid · 2 shared papers, 2012–2016 · strength 0.08
- Iréne Esposito · 2 shared papers, 2012–2016 · strength 0.08
- Amato J. Giaccia · 1 shared paper, 2017 · strength 0.06
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.