ResearcherNo recent work listed

Wei Lv

ORCID-verified (high confidence, 0.95), identity_conf@1As of 8 November 2026

Matched on ORCID iD 0009-0004-0820-8620; OpenAlex author ID A5111995792. Sources: OpenAlex.

Their work

Papers: 2 (first author 1, last author 1)
Trials: 0
Grants: 0
Labs led: 0
Collaborators: 4

Papers, trials and grants by year

2014: 1 paper · 2015: 1 paper

Papers 2

miR-15b promotes epithelial-mesenchymal transition by inhibiting SMURF2 in pancreatic cancer. Last author DOI 10.3892/ijo.2015.3076 ↗
miR‑96 functions as a tumor suppressor gene by targeting NUAK1 in pancreatic cancer. First author DOI 10.3892/ijmm.2014.1940 ↗

Closest collaborators

The 4 strongest of 4 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

Jianhua Zhang · 1 shared paper, 2015 · strength 0.06
- Paper miR-15b promotes epithelial-mesenchymal transition by inhibiting SMURF2 in pancreatic cancer. DOI 10.3892/ijo.2015.3076 ↗
Wenlong Zhang · 1 shared paper, 2015 · strength 0.06
- Paper miR-15b promotes epithelial-mesenchymal transition by inhibiting SMURF2 in pancreatic cancer. DOI 10.3892/ijo.2015.3076 ↗
Da‐Lin Lu · 1 shared paper, 2014 · strength 0.06
- Paper miR‑96 functions as a tumor suppressor gene by targeting NUAK1 in pancreatic cancer. DOI 10.3892/ijmm.2014.1940 ↗
Xuan Huang · 1 shared paper, 2014 · strength 0.06
- Paper miR‑96 functions as a tumor suppressor gene by targeting NUAK1 in pancreatic cancer. DOI 10.3892/ijmm.2014.1940 ↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.