ResearcherThe University of Texas MD Anderson Cancer CenterActive in the last 2 years
Jianhua Zhang
Matched on ORCID iD 0000-0001-5412-9860; OpenAlex author ID A5100434755. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 2, last author 1)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 55
2014: 2 papers · 2015: 1 paper · 2016: none · 2017: 3 papers · 2018: none · 2019: none · 2020: 3 papers · 2021: 1 paper · 2022: 1 paper · 2023: none · 2024: 1 paper
Papers 12
- Clonal dominance defines metastatic dissemination in pancreatic cancer. Co-author DOI 10.1126/sciadv.add9342↗
- Prognostic signature construction of energy metabolism-related genes in pancreatic cancer. First author DOI 10.3389/fonc.2022.917897↗
- USP21 deubiquitinase elevates macropinocytosis to enable oncogenic KRAS bypass in pancreatic cancer. Co-author DOI 10.1101/gad.348787.121↗
- Combination of PD-1 Inhibitor and OX40 Agonist Induces Tumor Rejection and Immune Memory in Mouse Models of Pancreatic Cancer. Co-author DOI 10.1053/j.gastro.2020.03.018↗
- Oncogenic KRAS-Driven Metabolic Reprogramming in Pancreatic Cancer Cells Utilizes Cytokines from the Tumor Microenvironment. Co-author DOI 10.1158/2159-8290.cd-19-0297↗
- Tumor Microenvironment Remodeling Enables Bypass of Oncogenic KRAS Dependency in Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-19-0597↗
- Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. Co-author DOI 10.1038/nature21064↗
- 4-1BB Agonist Focuses CD8+ Tumor-Infiltrating T-Cell Growth into a Distinct Repertoire Capable of Tumor Recognition in Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-17-0831↗
- Sclareolide enhances gemcitabine‑induced cell death through mediating the NICD and Gli1 pathways in gemcitabine‑resistant human pancreatic cancer. Last author DOI 10.3892/mmr.2017.6182↗
- miR-15b promotes epithelial-mesenchymal transition by inhibiting SMURF2 in pancreatic cancer. First author DOI 10.3892/ijo.2015.3076↗
- Yap1 activation enables bypass of oncogenic Kras addiction in pancreatic cancer. Co-author DOI 10.1016/j.cell.2014.06.003↗
- Identification of double-stranded genomic DNA spanning all chromosomes with mutated KRAS and p53 DNA in the serum exosomes of patients with pancreatic cancer. Co-author DOI 10.1074/jbc.c113.532267↗
Closest collaborators
The 25 strongest of 55 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Anirban Maitra · 5 shared papers, 2017–2024 · strength 0.41
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Paper Combination of PD-1 Inhibitor and OX40 Agonist Induces Tumor Rejection and Immune Memory in Mouse Models of Pancreatic … DOI 10.1053/j.gastro.2020.03.018↗
- Paper Oncogenic KRAS-Driven Metabolic Reprogramming in Pancreatic Cancer Cells Utilizes Cytokines from the Tumor Microenviron… DOI 10.1158/2159-8290.cd-19-0297↗
- Ronald Anthony Depinho · 5 shared papers, 2014–2021 · strength 0.35
- Paper USP21 deubiquitinase elevates macropinocytosis to enable oncogenic KRAS bypass in pancreatic cancer. DOI 10.1101/gad.348787.121↗
- Paper Tumor Microenvironment Remodeling Enables Bypass of Oncogenic KRAS Dependency in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-0597↗
- Paper Oncogenic KRAS-Driven Metabolic Reprogramming in Pancreatic Cancer Cells Utilizes Cytokines from the Tumor Microenviron… DOI 10.1158/2159-8290.cd-19-0297↗
- Haoqiang Ying · 4 shared papers, 2014–2024 · strength 0.28
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Paper Oncogenic KRAS-Driven Metabolic Reprogramming in Pancreatic Cancer Cells Utilizes Cytokines from the Tumor Microenviron… DOI 10.1158/2159-8290.cd-19-0297↗
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Timothy Paul Heffernan · 4 shared papers, 2014–2024 · strength 0.26
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Paper 4-1BB Agonist Focuses CD8+ Tumor-Infiltrating T-Cell Growth into a Distinct Repertoire Capable of Tumor Recognition in … DOI 10.1158/1078-0432.ccr-17-0831↗
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Giannicola Genovese · 3 shared papers, 2017–2024 · strength 0.25
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Paper Oncogenic KRAS-Driven Metabolic Reprogramming in Pancreatic Cancer Cells Utilizes Cytokines from the Tumor Microenviron… DOI 10.1158/2159-8290.cd-19-0297↗
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Huamin Wang · 4 shared papers, 2014–2020 · strength 0.25
- Paper Combination of PD-1 Inhibitor and OX40 Agonist Induces Tumor Rejection and Immune Memory in Mouse Models of Pancreatic … DOI 10.1053/j.gastro.2020.03.018↗
- Paper Oncogenic KRAS-Driven Metabolic Reprogramming in Pancreatic Cancer Cells Utilizes Cytokines from the Tumor Microenviron… DOI 10.1158/2159-8290.cd-19-0297↗
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Andrea Viale · 3 shared papers, 2014–2024 · strength 0.21
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Paper Yap1 activation enables bypass of oncogenic Kras addiction in pancreatic cancer. DOI 10.1016/j.cell.2014.06.003↗
- Giulio Francesco Draetta · 3 shared papers, 2014–2024 · strength 0.21
- Paper Clonal dominance defines metastatic dissemination in pancreatic cancer. DOI 10.1126/sciadv.add9342↗
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Paper Yap1 activation enables bypass of oncogenic Kras addiction in pancreatic cancer. DOI 10.1016/j.cell.2014.06.003↗
- Pingping Hou · 2 shared papers, 2020–2021 · strength 0.20
- Alessandro Carugo · 2 shared papers, 2017–2024 · strength 0.18
- Virginia Giuliani · 2 shared papers, 2017–2024 · strength 0.18
- Phillip Andrew Futreal · 2 shared papers, 2014–2024 · strength 0.17
- Hao Liu · 1 shared paper, 2022 · strength 0.16
- Yuxiang Zhao · 1 shared paper, 2022 · strength 0.16
- Wantong Yao · 2 shared papers, 2014–2021 · strength 0.14
- Francesca Citron · 1 shared paper, 2024 · strength 0.13
- I-Lin Ho · 1 shared paper, 2024 · strength 0.13
- Ziheng Chen · 1 shared paper, 2024 · strength 0.13
- Michael P. Kim · 1 shared paper, 2024 · strength 0.13
- Raghu Kalluri · 2 shared papers, 2014–2020 · strength 0.13
- Vincent Bernard · 2 shared papers, 2017–2020 · strength 0.13
- Paper Oncogenic KRAS-Driven Metabolic Reprogramming in Pancreatic Cancer Cells Utilizes Cytokines from the Tumor Microenviron… DOI 10.1158/2159-8290.cd-19-0297↗
- Paper 4-1BB Agonist Focuses CD8+ Tumor-Infiltrating T-Cell Growth into a Distinct Repertoire Capable of Tumor Recognition in … DOI 10.1158/1078-0432.ccr-17-0831↗
- Avnish Kapoor · 2 shared papers, 2014–2020 · strength 0.12
- Prasenjit Dey · 2 shared papers, 2017–2020 · strength 0.12
- Lynda Chin · 3 shared papers, 2014–2017 · strength 0.11
- Paper Synthetic vulnerabilities of mesenchymal subpopulations in pancreatic cancer. DOI 10.1038/nature21064↗
- Paper Identification of double-stranded genomic DNA spanning all chromosomes with mutated KRAS and p53 DNA in the serum exoso… DOI 10.1074/jbc.c113.532267↗
- Paper Yap1 activation enables bypass of oncogenic Kras addiction in pancreatic cancer. DOI 10.1016/j.cell.2014.06.003↗
- Yan Liang · 1 shared paper, 2021 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.