ResearcherThe University of Texas MD Anderson Cancer CenterNo recent work listed
Yan Liang
Matched on ORCID iD 0000-0002-3002-5431; OpenAlex author ID A5082895932. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 52
2013: 1 paper · 2014: none · 2015: none · 2016: none · 2017: none · 2018: none · 2019: 4 papers · 2020: 1 paper · 2021: 2 papers
Papers 8
- USP21 deubiquitinase elevates macropinocytosis to enable oncogenic KRAS bypass in pancreatic cancer. Co-author DOI 10.1101/gad.348787.121↗
- Targeting Glucose Metabolism Sensitizes Pancreatic Cancer to MEK Inhibition. Co-author DOI 10.1158/0008-5472.can-20-3792↗
- Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. Co-author DOI 10.1038/s41591-020-0947-8↗
- Syndecan 1 is a critical mediator of macropinocytosis in pancreatic cancer. Co-author DOI 10.1038/s41586-019-1062-1↗
- Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. Co-author DOI 10.1038/s41591-019-0368-8↗
- YAP1 oncogene is a context-specific driver for pancreatic ductal adenocarcinoma. Co-author DOI 10.1172/jci.insight.130811↗
- Glucose Metabolism in Pancreatic Cancer. Co-author DOI 10.3390/cancers11101460↗
- Analyzing S100A6 expression in endoscopic ultrasonography-guided fine-needle aspiration specimens: a promising diagnostic method of pancreatic cancer. Co-author DOI 10.1097/mcg.0b013e3182601752↗
Closest collaborators
The 25 strongest of 52 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Haoqiang Ying · 6 shared papers, 2019–2021 · strength 0.51
- Paper Glucose Metabolism in Pancreatic Cancer. DOI 10.3390/cancers11101460↗
- Paper Targeting Glucose Metabolism Sensitizes Pancreatic Cancer to MEK Inhibition. DOI 10.1158/0008-5472.can-20-3792↗
- Paper Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. DOI 10.1038/s41591-020-0947-8↗
- Wantong Yao · 5 shared papers, 2019–2021 · strength 0.48
- Paper Glucose Metabolism in Pancreatic Cancer. DOI 10.3390/cancers11101460↗
- Paper USP21 deubiquitinase elevates macropinocytosis to enable oncogenic KRAS bypass in pancreatic cancer. DOI 10.1101/gad.348787.121↗
- Paper Targeting Glucose Metabolism Sensitizes Pancreatic Cancer to MEK Inhibition. DOI 10.1158/0008-5472.can-20-3792↗
- Alec C. Kimmelman · 3 shared papers, 2019–2021 · strength 0.25
- Paper USP21 deubiquitinase elevates macropinocytosis to enable oncogenic KRAS bypass in pancreatic cancer. DOI 10.1101/gad.348787.121↗
- Paper Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. DOI 10.1038/s41591-020-0947-8↗
- Paper Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. DOI 10.1038/s41591-019-0368-8↗
- Timothy Paul Heffernan · 3 shared papers, 2019–2021 · strength 0.24
- Paper Targeting Glucose Metabolism Sensitizes Pancreatic Cancer to MEK Inhibition. DOI 10.1158/0008-5472.can-20-3792↗
- Paper YAP1 oncogene is a context-specific driver for pancreatic ductal adenocarcinoma. DOI 10.1172/jci.insight.130811↗
- Paper Syndecan 1 is a critical mediator of macropinocytosis in pancreatic cancer. DOI 10.1038/s41586-019-1062-1↗
- Jun Yao · 3 shared papers, 2019–2021 · strength 0.24
- Paper Targeting Glucose Metabolism Sensitizes Pancreatic Cancer to MEK Inhibition. DOI 10.1158/0008-5472.can-20-3792↗
- Paper YAP1 oncogene is a context-specific driver for pancreatic ductal adenocarcinoma. DOI 10.1172/jci.insight.130811↗
- Paper Syndecan 1 is a critical mediator of macropinocytosis in pancreatic cancer. DOI 10.1038/s41586-019-1062-1↗
- Jason B. Fleming · 3 shared papers, 2019–2021 · strength 0.24
- Paper Targeting Glucose Metabolism Sensitizes Pancreatic Cancer to MEK Inhibition. DOI 10.1158/0008-5472.can-20-3792↗
- Paper YAP1 oncogene is a context-specific driver for pancreatic ductal adenocarcinoma. DOI 10.1172/jci.insight.130811↗
- Paper Syndecan 1 is a critical mediator of macropinocytosis in pancreatic cancer. DOI 10.1038/s41586-019-1062-1↗
- Ya’an Kang · 2 shared papers, 2019–2021 · strength 0.18
- Bo Tu · 2 shared papers, 2019–2021 · strength 0.18
- Alessandro Carugo · 2 shared papers, 2019–2021 · strength 0.17
- Ronald Anthony Depinho · 2 shared papers, 2019–2021 · strength 0.17
- Pingping Hou · 2 shared papers, 2019–2021 · strength 0.17
- Bjoern Papke · 2 shared papers, 2019–2020 · strength 0.14
- Emanuel F. Petricoin · 2 shared papers, 2019–2020 · strength 0.14
- Nina V. Chaika · 2 shared papers, 2019–2020 · strength 0.14
- J. Nathaniel Diehl · 2 shared papers, 2019–2020 · strength 0.14
- Venugopal Gunda · 2 shared papers, 2019–2020 · strength 0.14
- Mariaelena Pierobon · 2 shared papers, 2019–2020 · strength 0.14
- Daniel Zeitouni · 2 shared papers, 2019–2020 · strength 0.14
- Yingxue Wang · 2 shared papers, 2019–2020 · strength 0.14
- Kirsten L. Bryant · 2 shared papers, 2019–2020 · strength 0.14
- Adrienne D. Cox · 2 shared papers, 2019–2020 · strength 0.14
- Erik S. Knudsen · 2 shared papers, 2019–2020 · strength 0.14
- Pankaj Kumar Singh · 2 shared papers, 2019–2020 · strength 0.14
- Jennifer E. Klomp · 2 shared papers, 2019–2020 · strength 0.14
- Agnieszka Katarzyna Witkiewicz · 2 shared papers, 2019–2020 · strength 0.14
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.