ResearcherUniversity of PisaActive in the last 2 years
Emanuel F. Petricoin
Matched on ORCID iD 0000-0001-9146-5572; OpenAlex author ID A5023762376. Sources: OpenAlex.
Their work
- Papers
- 25 (first author 1, last author 6)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 115
2011: 2 papers · 2012: 1 paper · 2013: 1 paper · 2014: none · 2015: none · 2016: 2 papers · 2017: none · 2018: 2 papers · 2019: 2 papers · 2020: 3 papers · 2021: 4 papers · 2022: 1 paper · 2023: 1 paper · 2024: 3 papers · 2025: 2 papers · 2026: 1 paper
Papers 25
- Targeting of MEK and Autophagy in Pancreatic Adenocarcinoma and Analysis of Treatment Sensitivity in Preclinical and Clinical Models: MEKiAUTO. Co-author DOI 10.1200/po-26-00138↗
- Evaluating the Effect of KRAS Variants on Survival Outcomes and Therapy Response in Pancreatic Cancer. Co-author DOI 10.1200/po-24-00684↗
- Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer and Overcomes RAS Inhibitor Resistance Co-author DOI 10.64898/2025.12.01.691471↗
- Keratin 17 is a prognostic and predictive biomarker in pancreatic ductal adenocarcinoma. Co-author DOI 10.1093/ajcp/aqae038↗
- Quantitative proteomic analysis of HER2 protein expression in PDAC tumors. Co-author DOI 10.1186/s12014-024-09476-7↗
- Keratin 17 modulates the immune topography of pancreatic cancer. Co-author DOI 10.1186/s12967-024-05252-1↗
- Association of Pancreatic Adenocarcinoma Location With DNA Damage Response Status and Response to Platinum-Based Therapy. Co-author DOI 10.1200/po.22.00648↗
- Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. Co-author DOI 10.1158/0008-5472.can-21-1443↗
- CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. Co-author DOI 10.1016/j.celrep.2021.110060↗
- The KRAS-regulated kinome identifies WEE1 and ERK coinhibition as a potential therapeutic strategy in KRAS-mutant pancreatic cancer. Co-author DOI 10.1016/j.jbc.2021.101335↗
- The FDA-Approved Anthelmintic Pyrvinium Pamoate Inhibits Pancreatic Cancer Cells in Nutrient-Depleted Conditions by Targeting the Mitochondria. Co-author DOI 10.1158/1535-7163.mct-20-0652↗
- Retrospective Case Series Analysis of RAF Family Alterations in Pancreatic Cancer: Real-World Outcomes From Targeted and Standard Therapies. Co-author DOI 10.1200/po.20.00494↗
- Overall survival in patients with pancreatic cancer receiving matched therapies following molecular profiling: a retrospective analysis of the Know Your Tumor … Last author DOI 10.1016/s1470-2045(20)30074-7↗
- Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. Co-author DOI 10.1038/s41591-020-0947-8↗
- Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-19-1006↗
- Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. Co-author DOI 10.1038/s41591-019-0368-8↗
- Outcomes in Patients With Pancreatic Adenocarcinoma With Genetic Mutations in DNA Damage Response Pathways: Results From the Know Your Tumor Program. Last author DOI 10.1200/po.19.00115↗
- Molecular Profiling of Patients with Pancreatic Cancer: Initial Results from the Know Your Tumor Initiative. Last author DOI 10.1158/1078-0432.ccr-18-0531↗
- Molecular Profiling of Pancreatic Cancer Patients-Response. First author DOI 10.1158/1078-0432.ccr-18-2645↗
- Long-Term ERK Inhibition in KRAS-Mutant Pancreatic Cancer Is Associated with MYC Degradation and Senescence-like Growth Suppression. Co-author DOI 10.1016/j.ccell.2015.11.011↗
- The mRNA-binding protein HuR promotes hypoxia-induced chemoresistance through posttranscriptional regulation of the proto-oncogene PIM1 in pancreatic cancer ce… Co-author DOI 10.1038/onc.2015.325↗
- Mass spectrometric analysis reveals O-methylation of pyruvate kinase from pancreatic cancer cells. Last author DOI 10.1007/s00216-013-6880-7↗
- MS analysis reveals O-methylation of L-lactate dehydrogenase from pancreatic ductal adenocarcinoma cells. Last author DOI 10.1002/elps.201200017↗
- Circulating autoantibodies to phosphorylated α-enolase are a hallmark of pancreatic cancer. Co-author DOI 10.1021/pr100213b↗
- Proteomic analysis of pancreatic ductal adenocarcinoma cells reveals metabolic alterations. Last author DOI 10.1021/pr101179t↗
Closest collaborators
The 25 strongest of 115 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Michael Jon Pishvaian · 8 shared papers, 2018–2025 · strength 1.02
- Paper Evaluating the Effect of KRAS Variants on Survival Outcomes and Therapy Response in Pancreatic Cancer. DOI 10.1200/po-24-00684↗
- Paper Molecular Profiling of Pancreatic Cancer Patients-Response. DOI 10.1158/1078-0432.ccr-18-2645↗
- Paper Association of Pancreatic Adenocarcinoma Location With DNA Damage Response Status and Response to Platinum-Based Therap… DOI 10.1200/po.22.00648↗
- Andrew Eugene Hendifar · 7 shared papers, 2018–2025 · strength 0.93
- Paper Evaluating the Effect of KRAS Variants on Survival Outcomes and Therapy Response in Pancreatic Cancer. DOI 10.1200/po-24-00684↗
- Paper Keratin 17 is a prognostic and predictive biomarker in pancreatic ductal adenocarcinoma. DOI 10.1093/ajcp/aqae038↗
- Paper Keratin 17 modulates the immune topography of pancreatic cancer. DOI 10.1186/s12967-024-05252-1↗
- Adrienne D. Cox · 8 shared papers, 2016–2025 · strength 0.79
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper The KRAS-regulated kinome identifies WEE1 and ERK coinhibition as a potential therapeutic strategy in KRAS-mutant pancr… DOI 10.1016/j.jbc.2021.101335↗
- Channing J. Der · 8 shared papers, 2016–2025 · strength 0.79
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper The KRAS-regulated kinome identifies WEE1 and ERK coinhibition as a potential therapeutic strategy in KRAS-mutant pancr… DOI 10.1016/j.jbc.2021.101335↗
- Lynn McCormick Matrisian · 6 shared papers, 2018–2024 · strength 0.72
- Paper Keratin 17 is a prognostic and predictive biomarker in pancreatic ductal adenocarcinoma. DOI 10.1093/ajcp/aqae038↗
- Paper Keratin 17 modulates the immune topography of pancreatic cancer. DOI 10.1186/s12967-024-05252-1↗
- Paper Association of Pancreatic Adenocarcinoma Location With DNA Damage Response Status and Response to Platinum-Based Therap… DOI 10.1200/po.22.00648↗
- Kirsten L. Bryant · 7 shared papers, 2016–2025 · strength 0.68
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Mariaelena Pierobon · 7 shared papers, 2016–2022 · strength 0.59
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper The KRAS-regulated kinome identifies WEE1 and ERK coinhibition as a potential therapeutic strategy in KRAS-mutant pancr… DOI 10.1016/j.jbc.2021.101335↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Clint A. Stalnecker · 5 shared papers, 2019–2025 · strength 0.56
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Bjoern Papke · 6 shared papers, 2019–2022 · strength 0.54
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper The KRAS-regulated kinome identifies WEE1 and ERK coinhibition as a potential therapeutic strategy in KRAS-mutant pancr… DOI 10.1016/j.jbc.2021.101335↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- J. Nathaniel Diehl · 5 shared papers, 2019–2021 · strength 0.41
- Paper The KRAS-regulated kinome identifies WEE1 and ERK coinhibition as a potential therapeutic strategy in KRAS-mutant pancr… DOI 10.1016/j.jbc.2021.101335↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Paper Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1006↗
- Jennifer E. Klomp · 5 shared papers, 2019–2021 · strength 0.41
- Paper The KRAS-regulated kinome identifies WEE1 and ERK coinhibition as a potential therapeutic strategy in KRAS-mutant pancr… DOI 10.1016/j.jbc.2021.101335↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Paper Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1006↗
- Jonathan Robert Brody · 5 shared papers, 2016–2021 · strength 0.37
- Paper Overall survival in patients with pancreatic cancer receiving matched therapies following molecular profiling: a retros… DOI 10.1016/s1470-2045(20)30074-7↗
- Paper The FDA-Approved Anthelmintic Pyrvinium Pamoate Inhibits Pancreatic Cancer Cells in Nutrient-Depleted Conditions by Tar… DOI 10.1158/1535-7163.mct-20-0652↗
- Paper Outcomes in Patients With Pancreatic Adenocarcinoma With Genetic Mutations in DNA Damage Response Pathways: Results Fro… DOI 10.1200/po.19.00115↗
- Wei Jiang · 3 shared papers, 2016–2024 · strength 0.37
- Paper Keratin 17 is a prognostic and predictive biomarker in pancreatic ductal adenocarcinoma. DOI 10.1093/ajcp/aqae038↗
- Paper Keratin 17 modulates the immune topography of pancreatic cancer. DOI 10.1186/s12967-024-05252-1↗
- Paper The mRNA-binding protein HuR promotes hypoxia-induced chemoresistance through posttranscriptional regulation of the pro… DOI 10.1038/onc.2015.325↗
- Lucia Roa‐Peña · 2 shared papers, 2024 · strength 0.33
- Brent K. Larson · 2 shared papers, 2024 · strength 0.33
- Lyanne Delgado Coka · 2 shared papers, 2024 · strength 0.33
- Luisa F. Escobar‐Hoyos · 2 shared papers, 2024 · strength 0.33
- Kenneth Reed Shroyer · 2 shared papers, 2024 · strength 0.33
- Andrew M. Waters · 4 shared papers, 2019–2021 · strength 0.31
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Paper Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1006↗
- Paper Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. DOI 10.1038/s41591-020-0947-8↗
- Lee M. Graves · 2 shared papers, 2021–2025 · strength 0.31
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper The KRAS-regulated kinome identifies WEE1 and ERK coinhibition as a potential therapeutic strategy in KRAS-mutant pancr… DOI 10.1016/j.jbc.2021.101335↗
- Jonathan M. DeLiberty · 3 shared papers, 2020–2022 · strength 0.30
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Paper Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1006↗
- Craig M. Goodwin · 3 shared papers, 2020–2022 · strength 0.30
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Paper Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1006↗
- Kristina Drizyte‐Miller · 2 shared papers, 2021–2025 · strength 0.30
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Charles J. Yeo · 3 shared papers, 2016–2023 · strength 0.29
- Paper Association of Pancreatic Adenocarcinoma Location With DNA Damage Response Status and Response to Platinum-Based Therap… DOI 10.1200/po.22.00648↗
- Paper The FDA-Approved Anthelmintic Pyrvinium Pamoate Inhibits Pancreatic Cancer Cells in Nutrient-Depleted Conditions by Tar… DOI 10.1158/1535-7163.mct-20-0652↗
- Paper The mRNA-binding protein HuR promotes hypoxia-induced chemoresistance through posttranscriptional regulation of the pro… DOI 10.1038/onc.2015.325↗
- Timothy Lewis Cannon · 2 shared papers, 2021–2024 · strength 0.29
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.