ResearcherUniversity of North Carolina at Chapel HillActive in the last 2 years
Kristina Drizyte‐Miller
Matched on ORCID iD 0000-0001-7049-2934; OpenAlex author ID A5012787124. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 26
2021: 1 paper · 2022: none · 2023: 1 paper · 2024: 2 papers · 2025: 4 papers
Papers 8
- Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. Co-author DOI 10.1126/scisignal.adu7145↗
- Concurrent Inhibition of the RAS-MAPK Pathway and PIKfyve Is a Therapeutic Strategy for Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-24-1757↗
- Combination of the MTA-Cooperative PRMT5 Inhibitor BMS-986504 and KRAS Inhibitors Is an Effective Treatment Strategy for MTAP-Deleted KRAS-Mutant Pancreatic Ca… Co-author DOI 10.1158/0008-5472.can-25-1507↗
- Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer and Overcomes RAS Inhibitor Resistance Co-author DOI 10.64898/2025.12.01.691471↗
- Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer. Co-author DOI 10.1038/s41586-024-07379-z↗
- Author Correction: Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer. Co-author DOI 10.1038/s41586-024-08084-7↗
- Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-22-0391↗
- CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. Co-author DOI 10.1016/j.celrep.2021.110060↗
Closest collaborators
The 25 strongest of 26 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Kirsten L. Bryant · 6 shared papers, 2021–2025 · strength 0.99
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Concurrent Inhibition of the RAS-MAPK Pathway and PIKfyve Is a Therapeutic Strategy for Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-1757↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Adrienne D. Cox · 6 shared papers, 2021–2025 · strength 0.99
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Concurrent Inhibition of the RAS-MAPK Pathway and PIKfyve Is a Therapeutic Strategy for Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-1757↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Clint A. Stalnecker · 6 shared papers, 2021–2025 · strength 0.99
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Concurrent Inhibition of the RAS-MAPK Pathway and PIKfyve Is a Therapeutic Strategy for Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-1757↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Channing J. Der · 6 shared papers, 2021–2025 · strength 0.99
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Concurrent Inhibition of the RAS-MAPK Pathway and PIKfyve Is a Therapeutic Strategy for Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-1757↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Emanuel F. Petricoin · 4 shared papers, 2023–2025 · strength 0.69
- Paper Concurrent Inhibition of the RAS-MAPK Pathway and PIKfyve Is a Therapeutic Strategy for Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-1757↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Combination of the MTA-Cooperative PRMT5 Inhibitor BMS-986504 and KRAS Inhibitors Is an Effective Treatment Strategy fo… DOI 10.1158/0008-5472.can-25-1507↗
- Mariaelena Pierobon · 4 shared papers, 2021–2025 · strength 0.59
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Combination of the MTA-Cooperative PRMT5 Inhibitor BMS-986504 and KRAS Inhibitors Is an Effective Treatment Strategy fo… DOI 10.1158/0008-5472.can-25-1507↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- Jennifer E. Klomp · 3 shared papers, 2021–2025 · strength 0.41
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Andrew M. Waters · 3 shared papers, 2021–2025 · strength 0.41
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Lee M. Graves · 2 shared papers, 2025 · strength 0.39
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Ryan N. Robb · 2 shared papers, 2025 · strength 0.37
- Paper Concurrent Inhibition of the RAS-MAPK Pathway and PIKfyve Is a Therapeutic Strategy for Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-1757↗
- Paper Combination of the MTA-Cooperative PRMT5 Inhibitor BMS-986504 and KRAS Inhibitors Is an Effective Treatment Strategy fo… DOI 10.1158/0008-5472.can-25-1507↗
- Mallory K. Roach · 2 shared papers, 2025 · strength 0.37
- Paper Concurrent Inhibition of the RAS-MAPK Pathway and PIKfyve Is a Therapeutic Strategy for Pancreatic Cancer. DOI 10.1158/0008-5472.can-24-1757↗
- Paper Combination of the MTA-Cooperative PRMT5 Inhibitor BMS-986504 and KRAS Inhibitors Is an Effective Treatment Strategy fo… DOI 10.1158/0008-5472.can-25-1507↗
- Emanuel F. Petricoin · 2 shared papers, 2021–2025 · strength 0.30
- Paper Dordaviprone/ONC201 Activation of the ClpP Mitochondrial Protease Inhibits the Growth of KRAS-Mutant Pancreatic Cancer … DOI 10.64898/2025.12.01.691471↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Jonathan M. DeLiberty · 2 shared papers, 2021–2025 · strength 0.28
- J. Nathaniel Diehl · 2 shared papers, 2021–2025 · strength 0.28
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Bjoern Papke · 2 shared papers, 2021–2023 · strength 0.23
- Craig M. Goodwin · 2 shared papers, 2021–2023 · strength 0.23
- John P. Morris · 1 shared paper, 2025 · strength 0.19
- Sarah E. Ackermann · 1 shared paper, 2025 · strength 0.19
- Naim U. Rashid · 1 shared paper, 2025 · strength 0.18
- A. Cole Edwards · 1 shared paper, 2025 · strength 0.18
- Priya S. Hibshman · 1 shared paper, 2025 · strength 0.18
- Autumn J. McRee · 1 shared paper, 2023 · strength 0.13
- Ye S. Lee · 1 shared paper, 2021 · strength 0.09
- Calvin Jay Kuo · 1 shared paper, 2021 · strength 0.09
- Christopher M Counter · 1 shared paper, 2021 · strength 0.09
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.