ResearcherBroad InstituteActive in the last 2 years
A. Cole Edwards
Matched on ORCID iD 0000-0001-9220-9642; OpenAlex author ID A5004033177, A5128909592; NIH RePORTER profile ID 16052067. Sources: OpenAlex.
Their work
- Papers
- 9
- Trials
- 0
- Grants
- 1
- Labs led
- 0
- Collaborators
- 34
2021: 1 paper · 2022: 1 paper · 2023: 1 grant · 2024: 2 papers · 2025: 2 papers · 2026: 3 papers
Papers 9
- Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. Co-author DOI 10.1016/j.celrep.2026.117627↗
- Cancer interception with KRAS inhibitors in preclinical models of pancreatic ductal adenocarcinoma. Co-author DOI 10.1126/science.aec7929↗
- CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. Co-author DOI 10.1158/0008-5472.can-25-1835↗
- Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. Co-author DOI 10.1126/scisignal.adu7145↗
- p53 and YAP/TAZ-TEAD activities determine metaplastic heterogeneity in pancreatic cancer Co-author DOI 10.64898/2025.12.11.693520↗
- Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer. Co-author DOI 10.1038/s41586-024-07379-z↗
- Author Correction: Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer. Co-author DOI 10.1038/s41586-024-08084-7↗
- Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. Co-author DOI 10.1158/0008-5472.can-21-1443↗
- Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. Co-author DOI 10.1016/j.celrep.2021.109291↗
Grants 1
Closest collaborators
The 25 strongest of 34 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Channing J. Der · 6 shared papers, 2021–2026 · strength 1.05
- Paper p53 and YAP/TAZ-TEAD activities determine metaplastic heterogeneity in pancreatic cancer DOI 10.64898/2025.12.11.693520↗
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Adrienne D. Cox · 5 shared papers, 2021–2026 · strength 0.80
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Clint A. Stalnecker · 5 shared papers, 2021–2026 · strength 0.80
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Andrew M. Waters · 4 shared papers, 2021–2026 · strength 0.67
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Craig M. Goodwin · 4 shared papers, 2021–2026 · strength 0.62
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Kirsten L. Bryant · 3 shared papers, 2022–2026 · strength 0.52
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Andrew J. Aguirre · 3 shared papers, 2021–2026 · strength 0.49
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- G. Aaron Hobbs · 3 shared papers, 2021–2026 · strength 0.49
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- J. Nathaniel Diehl · 3 shared papers, 2021–2026 · strength 0.46
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Priya S. Hibshman · 3 shared papers, 2021–2026 · strength 0.46
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Lee M. Graves · 3 shared papers, 2021–2026 · strength 0.46
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Jennifer E. Klomp · 3 shared papers, 2021–2026 · strength 0.46
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Bjoern Papke · 3 shared papers, 2021–2026 · strength 0.40
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Concurrent Inhibition of IGF1R and ERK Increases Pancreatic Cancer Sensitivity to Autophagy Inhibitors. DOI 10.1158/0008-5472.can-21-1443↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Mariaelena Pierobon · 2 shared papers, 2022–2025 · strength 0.31
- Devon R. Blake · 2 shared papers, 2021–2026 · strength 0.28
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- William C. Hahn · 2 shared papers, 2021–2026 · strength 0.28
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Tikvah K. Hayes · 2 shared papers, 2021–2026 · strength 0.28
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- John P. Morris · 1 shared paper, 2025 · strength 0.26
- Cole P Martin · 1 shared paper, 2025 · strength 0.26
- Minh T. Than · 1 shared paper, 2026 · strength 0.23
- Ben Z. Stanger · 1 shared paper, 2026 · strength 0.23
- Robert H. Vonderheide · 1 shared paper, 2026 · strength 0.23
- Ashley L. Kiemen · 1 shared paper, 2026 · strength 0.23
- Szu-Aun Lim · 1 shared paper, 2026 · strength 0.21
- Julien Dilly · 1 shared paper, 2026 · strength 0.21
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.