ResearcherUniversity of North Carolina at Chapel HillActive in the last 2 years
Andrew M. Waters
Matched on ORCID iD 0000-0002-6058-5878; OpenAlex author ID A5031911025; NIH RePORTER profile ID 14892518. Sources: funder_awards.
Their work
- Papers
- 12 (first author 1, last author 2)
- Trials
- 0
- Grants
- 2
- Labs led
- 1
- Collaborators
- 61
2018: 1 paper · 2019: 1 paper · 2020: 2 papers · 2021: 2 papers · 2022: none · 2023: 1 paper · 2024: 1 paper · 2025: 2 papers, 1 grant · 2026: 2 papers, 1 grant
Papers 12
- Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. Co-author DOI 10.1016/j.celrep.2026.117627↗
- CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. Last author DOI 10.1158/0008-5472.can-25-1835↗
- Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. Co-author DOI 10.1126/scisignal.adu7145↗
- Human Pancreatic Cancer Single-Cell Atlas Reveals Association of CXCL10+ Fibroblasts and Basal Subtype Tumor Cells. Co-author DOI 10.1158/1078-0432.ccr-24-2183↗
- Evaluation of KRAS inhibitor-directed therapies for pancreatic cancer treatment. Last author DOI 10.3389/fonc.2024.1402128↗
- Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. First author DOI 10.1158/0008-5472.can-22-0391↗
- Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. Co-author DOI 10.1016/j.celrep.2021.109291↗
- CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. Co-author DOI 10.1016/j.celrep.2021.110060↗
- Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. Co-author DOI 10.1038/s41591-020-0947-8↗
- Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-19-1006↗
- Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. Co-author DOI 10.1038/s41591-019-0368-8↗
- KRAS: The Critical Driver and Therapeutic Target for Pancreatic Cancer. Co-author DOI 10.1101/cshperspect.a031435↗
Grants 2
Closest collaborators
The 25 strongest of 61 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Channing J. Der · 10 shared papers, 2018–2026 · strength 1.29
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Adrienne D. Cox · 9 shared papers, 2019–2026 · strength 1.11
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Clint A. Stalnecker · 8 shared papers, 2019–2026 · strength 1.04
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Jennifer E. Klomp · 8 shared papers, 2019–2026 · strength 0.90
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- Kirsten L. Bryant · 7 shared papers, 2019–2026 · strength 0.84
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- Craig M. Goodwin · 6 shared papers, 2020–2026 · strength 0.79
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- J. Nathaniel Diehl · 7 shared papers, 2019–2026 · strength 0.77
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Bjoern Papke · 7 shared papers, 2019–2026 · strength 0.72
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- G. Aaron Hobbs · 6 shared papers, 2019–2026 · strength 0.70
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- A. Cole Edwards · 4 shared papers, 2021–2026 · strength 0.67
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Mariaelena Pierobon · 6 shared papers, 2019–2025 · strength 0.63
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Andrew J. Aguirre · 4 shared papers, 2020–2026 · strength 0.57
- Paper CRISPR-Cas9 Screening Identifies Resistance Mechanisms to KRAS Inhibition in Pancreatic Cancer. DOI 10.1158/0008-5472.can-25-1835↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Szu-Aun Lim · 2 shared papers, 2024–2026 · strength 0.49
- Priya S. Hibshman · 3 shared papers, 2021–2026 · strength 0.46
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Lee M. Graves · 3 shared papers, 2021–2026 · strength 0.46
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Tikvah K. Hayes · 4 shared papers, 2019–2026 · strength 0.41
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Paper Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. DOI 10.1038/s41591-020-0947-8↗
- Kristina Drizyte‐Miller · 3 shared papers, 2021–2025 · strength 0.41
- Paper Defining the MYC-regulated transcriptome and kinome that support KRAS- and ERK-dependent growth of pancreatic cancer. DOI 10.1126/scisignal.adu7145↗
- Paper Combination Therapies with CDK4/6 Inhibitors to Treat KRAS-Mutant Pancreatic Cancer. DOI 10.1158/0008-5472.can-22-0391↗
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Emanuel F. Petricoin · 2 shared papers, 2023–2025 · strength 0.32
- Emanuel F. Petricoin · 4 shared papers, 2019–2021 · strength 0.31
- Paper CHK1 protects oncogenic KRAS-expressing cells from DNA damage and is a target for pancreatic cancer treatment. DOI 10.1016/j.celrep.2021.110060↗
- Paper Atypical KRAS G12R Mutant Is Impaired in PI3K Signaling and Macropinocytosis in Pancreatic Cancer. DOI 10.1158/2159-8290.cd-19-1006↗
- Paper Author Correction: Combination of ERK and autophagy inhibition as a treatment approach for pancreatic cancer. DOI 10.1038/s41591-020-0947-8↗
- Syed Ameen Ahmad · 1 shared paper, 2024 · strength 0.28
- Devon R. Blake · 2 shared papers, 2021–2026 · strength 0.28
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- William C. Hahn · 2 shared papers, 2021–2026 · strength 0.28
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2026.117627↗
- Paper Targeting p130Cas- and microtubule-dependent MYC regulation sensitizes pancreatic cancer to ERK MAPK inhibition. DOI 10.1016/j.celrep.2021.109291↗
- Julien Dilly · 1 shared paper, 2026 · strength 0.21
- Syed Arif Ahmad · 1 shared paper, 2026 · strength 0.21
- Sarah E. Ackermann · 1 shared paper, 2026 · strength 0.21
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.