ResearcherUniversity of Massachusetts Chan Medical SchoolActive in the last 2 years
Nikita Bhalerao
Matched on ORCID iD 0009-0007-1633-4389; OpenAlex author ID A5035854387. Sources: OpenAlex.
Their work
- Papers
- 6 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 20
2021: 1 paper · 2022: none · 2023: 2 papers · 2024: none · 2025: 2 papers · 2026: 1 paper
Papers 6
- Multiplexed cytokine and antigen mRNA administration generates durable anti-tumor immunity against pancreatic cancer. Co-author DOI 10.1038/s41467-026-74574-z↗
- Mitochondrial Ca2+ controls pancreatic cancer growth and metastasis by regulating epithelial cell plasticity. Co-author DOI 10.1016/j.celrep.2025.115627↗
- Pancreatic cancer cachexia is mediated by PTHrP-driven disruption of adipose de novo lipogenesis Co-author DOI 10.1101/2025.06.03.657464↗
- Role of tumor cell sialylation in pancreatic cancer progression. Co-author DOI 10.1016/bs.acr.2022.07.003↗
- ST6GAL1 sialyltransferase promotes acinar to ductal metaplasia and pancreatic cancer progression. Co-author DOI 10.1172/jci.insight.161563↗
- Glycosyltransferase ST6Gal-I promotes the epithelial to mesenchymal transition in pancreatic cancer cells. First author DOI 10.1074/jbc.ra120.014126↗
Closest collaborators
The 20 strongest of 20 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Marcus A. Ruscetti · 3 shared papers, 2025–2026 · strength 0.61
- Paper Multiplexed cytokine and antigen mRNA administration generates durable anti-tumor immunity against pancreatic cancer. DOI 10.1038/s41467-026-74574-z↗
- Paper Mitochondrial Ca2+ controls pancreatic cancer growth and metastasis by regulating epithelial cell plasticity. DOI 10.1016/j.celrep.2025.115627↗
- Paper Pancreatic cancer cachexia is mediated by PTHrP-driven disruption of adipose de novo lipogenesis DOI 10.1101/2025.06.03.657464↗
- Jason Robert Pitarresi · 3 shared papers, 2025–2026 · strength 0.61
- Paper Multiplexed cytokine and antigen mRNA administration generates durable anti-tumor immunity against pancreatic cancer. DOI 10.1038/s41467-026-74574-z↗
- Paper Mitochondrial Ca2+ controls pancreatic cancer growth and metastasis by regulating epithelial cell plasticity. DOI 10.1016/j.celrep.2025.115627↗
- Paper Pancreatic cancer cachexia is mediated by PTHrP-driven disruption of adipose de novo lipogenesis DOI 10.1101/2025.06.03.657464↗
- Susan L Bellis · 3 shared papers, 2021–2023 · strength 0.51
- Paper Role of tumor cell sialylation in pancreatic cancer progression. DOI 10.1016/bs.acr.2022.07.003↗
- Paper ST6GAL1 sialyltransferase promotes acinar to ductal metaplasia and pancreatic cancer progression. DOI 10.1172/jci.insight.161563↗
- Paper Glycosyltransferase ST6Gal-I promotes the epithelial to mesenchymal transition in pancreatic cancer cells. DOI 10.1074/jbc.ra120.014126↗
- Jessica Peura · 2 shared papers, 2025 · strength 0.39
- Michael P. Marciel · 2 shared papers, 2023 · strength 0.38
- Donald J Buchsbaum · 2 shared papers, 2021–2023 · strength 0.27
- Colleen M. Britain · 2 shared papers, 2021–2023 · strength 0.27
- Austin D. Silva · 2 shared papers, 2021–2023 · strength 0.27
- Asmi Chakraborty · 2 shared papers, 2021–2023 · strength 0.27
- Loretah Chibaya · 1 shared paper, 2026 · strength 0.22
- Chaitanya Naimesh Parikh · 1 shared paper, 2026 · strength 0.22
- Brian C Lewis · 1 shared paper, 2026 · strength 0.22
- Jillian Weissenrieder · 1 shared paper, 2025 · strength 0.21
- James Kevin FOSKETT · 1 shared paper, 2025 · strength 0.21
- Ben Z. Stanger · 1 shared paper, 2025 · strength 0.21
- Anil K. Rustgi · 1 shared paper, 2025 · strength 0.21
- Robert J. Norgard · 1 shared paper, 2025 · strength 0.18
- Michael Anthony Hollingsworth · 1 shared paper, 2025 · strength 0.18
- Paul M. Grandgenett · 1 shared paper, 2025 · strength 0.18
- Christopher Klug · 1 shared paper, 2023 · strength 0.14
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.