ResearcherUniversity of Alabama at BirminghamActive in the last 2 years
Susan L Bellis
Matched on ORCID iD 0000-0002-5071-527X; OpenAlex author ID A5039063725; NIH RePORTER profile ID 6107732. Sources: OpenAlex.
Their work
- Papers
- 5 (first author 0, last author 5)
- Trials
- 0
- Grants
- 2
- Labs led
- 1
- Collaborators
- 8
2016: 1 grant · 2017: none · 2018: 1 paper · 2019: none · 2020: none · 2021: 1 paper · 2022: 1 grant · 2023: 2 papers · 2024: 1 paper
Papers 5
- The pro-inflammatory cytokines IL-1β and IL-6 promote upregulation of the ST6GAL1 sialyltransferase in pancreatic cancer cells. Last author DOI 10.1016/j.jbc.2024.107752↗
- Role of tumor cell sialylation in pancreatic cancer progression. Last author DOI 10.1016/bs.acr.2022.07.003↗
- ST6GAL1 sialyltransferase promotes acinar to ductal metaplasia and pancreatic cancer progression. Last author DOI 10.1172/jci.insight.161563↗
- Glycosyltransferase ST6Gal-I promotes the epithelial to mesenchymal transition in pancreatic cancer cells. Last author DOI 10.1074/jbc.ra120.014126↗
- ST6Gal-I sialyltransferase promotes chemoresistance in pancreatic ductal adenocarcinoma by abrogating gemcitabine-mediated DNA damage. Last author DOI 10.1074/jbc.m117.808584↗
Grants 2
Closest collaborators
The 8 strongest of 8 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Michael P. Marciel · 3 shared papers, 2023–2024 · strength 0.69
- Paper The pro-inflammatory cytokines IL-1β and IL-6 promote upregulation of the ST6GAL1 sialyltransferase in pancreatic cance… DOI 10.1016/j.jbc.2024.107752↗
- Paper Role of tumor cell sialylation in pancreatic cancer progression. DOI 10.1016/bs.acr.2022.07.003↗
- Paper ST6GAL1 sialyltransferase promotes acinar to ductal metaplasia and pancreatic cancer progression. DOI 10.1172/jci.insight.161563↗
- Lance Wells · 1 shared grant, 2022 · strength 0.66
- Austin D. Silva · 3 shared papers, 2021–2024 · strength 0.58
- Paper The pro-inflammatory cytokines IL-1β and IL-6 promote upregulation of the ST6GAL1 sialyltransferase in pancreatic cance… DOI 10.1016/j.jbc.2024.107752↗
- Paper ST6GAL1 sialyltransferase promotes acinar to ductal metaplasia and pancreatic cancer progression. DOI 10.1172/jci.insight.161563↗
- Paper Glycosyltransferase ST6Gal-I promotes the epithelial to mesenchymal transition in pancreatic cancer cells. DOI 10.1074/jbc.ra120.014126↗
- Nikita Bhalerao · 3 shared papers, 2021–2023 · strength 0.51
- Paper Role of tumor cell sialylation in pancreatic cancer progression. DOI 10.1016/bs.acr.2022.07.003↗
- Paper ST6GAL1 sialyltransferase promotes acinar to ductal metaplasia and pancreatic cancer progression. DOI 10.1172/jci.insight.161563↗
- Paper Glycosyltransferase ST6Gal-I promotes the epithelial to mesenchymal transition in pancreatic cancer cells. DOI 10.1074/jbc.ra120.014126↗
- Donald J Buchsbaum · 3 shared papers, 2018–2023 · strength 0.36
- Paper ST6GAL1 sialyltransferase promotes acinar to ductal metaplasia and pancreatic cancer progression. DOI 10.1172/jci.insight.161563↗
- Paper Glycosyltransferase ST6Gal-I promotes the epithelial to mesenchymal transition in pancreatic cancer cells. DOI 10.1074/jbc.ra120.014126↗
- Paper ST6Gal-I sialyltransferase promotes chemoresistance in pancreatic ductal adenocarcinoma by abrogating gemcitabine-media… DOI 10.1074/jbc.m117.808584↗
- Asmi Chakraborty · 3 shared papers, 2018–2023 · strength 0.36
- Paper ST6GAL1 sialyltransferase promotes acinar to ductal metaplasia and pancreatic cancer progression. DOI 10.1172/jci.insight.161563↗
- Paper Glycosyltransferase ST6Gal-I promotes the epithelial to mesenchymal transition in pancreatic cancer cells. DOI 10.1074/jbc.ra120.014126↗
- Paper ST6Gal-I sialyltransferase promotes chemoresistance in pancreatic ductal adenocarcinoma by abrogating gemcitabine-media… DOI 10.1074/jbc.m117.808584↗
- Colleen M. Britain · 2 shared papers, 2021–2023 · strength 0.27
- Christopher Klug · 1 shared paper, 2023 · strength 0.14
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.