ResearcherPurdue University West LafayetteActive in the last 2 years
Matthew J. Flick
Matched on ORCID iD 0000-0002-5034-3162; OpenAlex author ID A5016211446; NIH RePORTER profile ID 2315255. Sources: OpenAlex.
Their work
- Papers
- 8 (first author 1, last author 2)
- Trials
- 0
- Grants
- 3
- Labs led
- 2
- Collaborators
- 23
2019: 1 paper · 2020: none · 2021: 3 papers · 2022: 2 grants · 2023: none · 2024: 1 paper · 2025: 1 paper · 2026: 2 papers, 1 grant
Papers 8
- Depletion of Fibrinogen Suppresses Growth of Primary Tumors and Metastasis of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1053/j.gastro.2025.09.024↗
- Extravascular coagulation stabilizes pro-fibrotic stromal states via tumor-intrinsic PAR1 signaling in pancreatic ductal adenocarcinoma Co-author DOI 10.64898/2026.06.25.734662↗
- Targeting uPAR with an antibody-drug conjugate suppresses tumor growth and reshapes the immune landscape in pancreatic cancer models. Co-author DOI 10.1126/sciadv.adq0513↗
- Plasminogen deficiency suppresses pancreatic ductal adenocarcinoma disease progression. Last author DOI 10.1002/1878-0261.13552↗
- Thrombin-PAR1 signaling in pancreatic cancer promotes an immunosuppressive microenvironment. Co-author DOI 10.1111/jth.15115↗
- Plasminogen activator inhibitor 1 and venous thrombosis in pancreatic cancer. Co-author DOI 10.1182/bloodadvances.2020003149↗
- Coagulation Signaling through PAR1 as a Therapeutic Target in Pancreatic Ductal Adenocarcinoma. First author DOI 10.3390/ijms22105138↗
- Thrombin Signaling Promotes Pancreatic Adenocarcinoma through PAR-1-Dependent Immune Evasion. Last author DOI 10.1158/0008-5472.can-18-3206↗
Grants 3
- Reprogramming PDAC Stroma by Targeting Coagulation in the Tumor Microenvironment U01CA274304↗
- Thrombin-dependent mechanisms of pancreatic ductal adenocarcinoma disease R01CA211098↗
- Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis U01HL143403↗
Closest collaborators
The 23 strongest of 23 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Melissa L. Fishel · 3 shared papers, 2 shared grants, 2022–2026 · strength 2.10
- Grant Reprogramming PDAC Stroma by Targeting Coagulation in the Tumor Microenvironment U01CA274304↗
- Grant Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis U01HL143403↗
- Paper Extravascular coagulation stabilizes pro-fibrotic stromal states via tumor-intrinsic PAR1 signaling in pancreatic ducta… DOI 10.64898/2026.06.25.734662↗
- Bumsoo Han · 1 shared paper, 2 shared grants, 2022–2026 · strength 1.70
- Grant Reprogramming PDAC Stroma by Targeting Coagulation in the Tumor Microenvironment U01CA274304↗
- Grant Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis U01HL143403↗
- Paper Extravascular coagulation stabilizes pro-fibrotic stromal states via tumor-intrinsic PAR1 signaling in pancreatic ducta… DOI 10.64898/2026.06.25.734662↗
- Stephen F Konieczny · 2 shared papers, 1 shared grant, 2019–2022 · strength 0.87
- Grant Thrombin-dependent mechanisms of pancreatic ductal adenocarcinoma disease R01CA211098↗
- Paper Thrombin-PAR1 signaling in pancreatic cancer promotes an immunosuppressive microenvironment. DOI 10.1111/jth.15115↗
- Paper Thrombin Signaling Promotes Pancreatic Adenocarcinoma through PAR-1-Dependent Immune Evasion. DOI 10.1158/0008-5472.can-18-3206↗
- Alisa S. Wolberg · 2 shared papers, 1 shared grant, 2022–2026 · strength 0.85
- Grant Targeting the Plasminogen Activation System to Limit Pancreatic Cancer Progression and Associated Thrombosis U01HL143403↗
- Paper Depletion of Fibrinogen Suppresses Growth of Primary Tumors and Metastasis of Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2025.09.024↗
- Paper Plasminogen deficiency suppresses pancreatic ductal adenocarcinoma disease progression. DOI 10.1002/1878-0261.13552↗
- Sae Rome Choi · 2 shared papers, 2026 · strength 0.47
- Paper Extravascular coagulation stabilizes pro-fibrotic stromal states via tumor-intrinsic PAR1 signaling in pancreatic ducta… DOI 10.64898/2026.06.25.734662↗
- Paper Depletion of Fibrinogen Suppresses Growth of Primary Tumors and Metastasis of Pancreatic Ductal Adenocarcinoma. DOI 10.1053/j.gastro.2025.09.024↗
- Nayela N. Chowdhury · 2 shared papers, 2024–2026 · strength 0.40
- Yi Yang · 3 shared papers, 2019–2024 · strength 0.39
- Paper Plasminogen deficiency suppresses pancreatic ductal adenocarcinoma disease progression. DOI 10.1002/1878-0261.13552↗
- Paper Thrombin-PAR1 signaling in pancreatic cancer promotes an immunosuppressive microenvironment. DOI 10.1111/jth.15115↗
- Paper Thrombin Signaling Promotes Pancreatic Adenocarcinoma through PAR-1-Dependent Immune Evasion. DOI 10.1158/0008-5472.can-18-3206↗
- Aditi Kothari · 1 shared paper, 2021 · strength 0.28
- Hye‐ran Moon · 1 shared paper, 2026 · strength 0.25
- Claudius H. Conrad · 1 shared paper, 2026 · strength 0.25
- Nigel Mackman · 2 shared papers, 2021 · strength 0.24
- Virginia Metrangolo · 1 shared paper, 2025 · strength 0.24
- Lars Henning Engelholm · 1 shared paper, 2025 · strength 0.24
- Silpa Gampala · 1 shared paper, 2026 · strength 0.22
- Wei Ping Jia · 1 shared paper, 2026 · strength 0.22
- Patrick G. Schweickert · 2 shared papers, 2019–2021 · strength 0.21
- Fenil Shah · 1 shared paper, 2024 · strength 0.18
- Emily White · 1 shared paper, 2021 · strength 0.12
- Florian Moik · 1 shared paper, 2021 · strength 0.12
- Yohei Hisada · 1 shared paper, 2021 · strength 0.12
- Tomohiro Kawano · 1 shared paper, 2021 · strength 0.12
- Ingrid Pabinger · 1 shared paper, 2021 · strength 0.12
- Cihan Ay · 1 shared paper, 2021 · strength 0.12
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.