ResearcherPurdue University West LafayetteNo recent work listed
Stephen F Konieczny
Matched on ORCID iD 0000-0002-3281-3714; OpenAlex author ID A5014807279; NIH RePORTER profile ID 1919792. Sources: OpenAlex.
Their work
- Papers
- 11 (first author 0, last author 2)
- Trials
- 0
- Grants
- 3
- Labs led
- 1
- Collaborators
- 29
2013: 1 paper · 2014: none · 2015: 1 paper · 2016: 2 papers · 2017: 2 grants · 2018: 1 paper · 2019: 2 papers · 2020: 2 papers · 2021: 1 paper · 2022: 1 paper, 1 grant
Papers 11
- Targeting Plk1 Sensitizes Pancreatic Cancer to Immune Checkpoint Therapy. Co-author DOI 10.1158/0008-5472.can-22-0018↗
- Thrombin-PAR1 signaling in pancreatic cancer promotes an immunosuppressive microenvironment. Last author DOI 10.1111/jth.15115↗
- A Biomimetic Tumor Model of Heterogeneous Invasion in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1002/smll.201905500↗
- An engineered pancreatic cancer model with intra-tumoral heterogeneity of driver mutations. Co-author DOI 10.1039/d0lc00707b↗
- Identification and Characterization of AES-135, a Hydroxamic Acid-Based HDAC Inhibitor That Prolongs Survival in an Orthotopic Mouse Model of Pancreatic Cancer. Co-author DOI 10.1021/acs.jmedchem.8b01957↗
- Thrombin Signaling Promotes Pancreatic Adenocarcinoma through PAR-1-Dependent Immune Evasion. Co-author DOI 10.1158/0008-5472.can-18-3206↗
- Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemc… Last author DOI 10.1002/1878-0261.12314↗
- Abrogating cholesterol esterification suppresses growth and metastasis of pancreatic cancer. Co-author DOI 10.1038/onc.2016.168↗
- Plk1 inhibition enhances the efficacy of gemcitabine in human pancreatic cancer. Co-author DOI 10.1080/15384101.2016.1148838↗
- The basic helix-loop-helix transcription factor E47 reprograms human pancreatic cancer cells to a quiescent acinar state with reduced tumorigenic potential. Co-author DOI 10.1097/mpa.0000000000000328↗
- Plk1 phosphorylation of orc2 and hbo1 contributes to gemcitabine resistance in pancreatic cancer. Co-author DOI 10.1158/1535-7163.mct-12-0632↗
Grants 3
Closest collaborators
The 25 strongest of 29 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Matthew J. Flick · 2 shared papers, 1 shared grant, 2019–2022 · strength 0.87
- Grant Thrombin-dependent mechanisms of pancreatic ductal adenocarcinoma disease R01CA211098↗
- Paper Thrombin-PAR1 signaling in pancreatic cancer promotes an immunosuppressive microenvironment. DOI 10.1111/jth.15115↗
- Paper Thrombin Signaling Promotes Pancreatic Adenocarcinoma through PAR-1-Dependent Immune Evasion. DOI 10.1158/0008-5472.can-18-3206↗
- Yi Yang · 6 shared papers, 2016–2021 · strength 0.63
- Paper A Biomimetic Tumor Model of Heterogeneous Invasion in Pancreatic Ductal Adenocarcinoma. DOI 10.1002/smll.201905500↗
- Paper An engineered pancreatic cancer model with intra-tumoral heterogeneity of driver mutations. DOI 10.1039/d0lc00707b↗
- Paper Thrombin-PAR1 signaling in pancreatic cancer promotes an immunosuppressive microenvironment. DOI 10.1111/jth.15115↗
- Patrick G. Schweickert · 4 shared papers, 2016–2021 · strength 0.35
- Paper Thrombin-PAR1 signaling in pancreatic cancer promotes an immunosuppressive microenvironment. DOI 10.1111/jth.15115↗
- Paper Thrombin Signaling Promotes Pancreatic Adenocarcinoma through PAR-1-Dependent Immune Evasion. DOI 10.1158/0008-5472.can-18-3206↗
- Paper Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic … DOI 10.1002/1878-0261.12314↗
- Bumsoo Han · 2 shared papers, 2020 · strength 0.28
- Xiaoqi Liu · 4 shared papers, 2013–2022 · strength 0.27
- Paper Targeting Plk1 Sensitizes Pancreatic Cancer to Immune Checkpoint Therapy. DOI 10.1158/0008-5472.can-22-0018↗
- Paper Plk1 inhibition enhances the efficacy of gemcitabine in human pancreatic cancer. DOI 10.1080/15384101.2016.1148838↗
- Paper Abrogating cholesterol esterification suppresses growth and metastasis of pancreatic cancer. DOI 10.1038/onc.2016.168↗
- Michael J. Bradney · 1 shared paper, 2020 · strength 0.15
- Hye‐ran Moon · 1 shared paper, 2020 · strength 0.13
- Brad L. Jakubison · 2 shared papers, 2015–2018 · strength 0.13
- Paper Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic … DOI 10.1002/1878-0261.12314↗
- Paper The basic helix-loop-helix transcription factor E47 reprograms human pancreatic cancer cells to a quiescent acinar stat… DOI 10.1097/mpa.0000000000000328↗
- Pamela Itkin‐Ansari · 2 shared papers, 2015–2018 · strength 0.13
- Paper Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic … DOI 10.1002/1878-0261.12314↗
- Paper The basic helix-loop-helix transcription factor E47 reprograms human pancreatic cancer cells to a quiescent acinar stat… DOI 10.1097/mpa.0000000000000328↗
- Nigel Mackman · 1 shared paper, 2021 · strength 0.12
- Emily White · 1 shared paper, 2021 · strength 0.12
- Lijun Cheng · 1 shared paper, 2022 · strength 0.12
- Zhuangzhuang Zhang · 1 shared paper, 2022 · strength 0.12
- Yunlong Liu · 1 shared paper, 2018 · strength 0.08
- Melissa L. Fishel · 1 shared paper, 2019 · strength 0.07
- Andrew E. Shouksmith · 1 shared paper, 2019 · strength 0.07
- Fenil Shah · 1 shared paper, 2019 · strength 0.07
- Patrick Thomas Gunning · 1 shared paper, 2019 · strength 0.07
- Jie Li · 1 shared paper, 2016 · strength 0.06
- Nihal Ahmad · 1 shared paper, 2016 · strength 0.06
- J-X Cheng · 1 shared paper, 2016 · strength 0.05
- Shaoxiong Chen · 1 shared paper, 2016 · strength 0.05
- Dongsheng Gu · 1 shared paper, 2016 · strength 0.05
- Junjie Li · 1 shared paper, 2016 · strength 0.05
- Jingwu Xie · 1 shared paper, 2016 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.