ResearcherColumbia University Irving Medical CenterActive in the last 2 years
Basil S. Bakir
Matched on OpenAlex author ID A5064061542; NIH RePORTER profile ID 10222837. Sources: OpenAlex.
Their work
- Papers
- 9 (first author 2, last author 0)
- Trials
- 0
- Grants
- 1
- Labs led
- 0
- Collaborators
- 53
2016: 2 papers, 1 grant · 2017: none · 2018: 2 papers · 2019: none · 2020: none · 2021: 1 paper · 2022: none · 2023: 1 paper · 2024: 2 papers · 2025: 1 paper
Papers 9
- Cellular states associated with metastatic organotropism and survival in patients with pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41588-025-02345-5↗
- Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer. Co-author DOI 10.1038/s41586-024-07379-z↗
- Author Correction: Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer. Co-author DOI 10.1038/s41586-024-08084-7↗
- Phase Ib and Expansion Study of Gemcitabine, Nab-Paclitaxel, and Ficlatuzumab in Patients With Metastatic Pancreatic Cancer. Co-author DOI 10.1093/oncolo/oyad002↗
- PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability. Co-author DOI 10.1158/2159-8290.cd-20-1098↗
- Regulation of Epithelial Plasticity Determines Metastatic Organotropism in Pancreatic Cancer. First author DOI 10.1016/j.devcel.2018.05.025↗
- Pancreas 3D Organoids: Current and Future Aspects as a Research Platform for Personalized Medicine in Pancreatic Cancer. First author DOI 10.1016/j.jcmgh.2017.12.004↗
- ETS-Transcription Factor ETV1 Regulates Stromal Expansion and Metastasis in Pancreatic Cancer. Co-author DOI 10.1053/j.gastro.2016.06.005↗
- Prrx1 isoform switching regulates pancreatic cancer invasion and metastatic colonization. Co-author DOI 10.1101/gad.263327.115↗
Grants 1
Closest collaborators
The 25 strongest of 53 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Anil K. Rustgi · 5 shared papers, 2016–2023 · strength 0.43
- Paper Phase Ib and Expansion Study of Gemcitabine, Nab-Paclitaxel, and Ficlatuzumab in Patients With Metastatic Pancreatic Ca… DOI 10.1093/oncolo/oyad002↗
- Paper Pancreas 3D Organoids: Current and Future Aspects as a Research Platform for Personalized Medicine in Pancreatic Cancer. DOI 10.1016/j.jcmgh.2017.12.004↗
- Paper PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability. DOI 10.1158/2159-8290.cd-20-1098↗
- Jason Robert Pitarresi · 3 shared papers, 2018–2023 · strength 0.29
- Paper Phase Ib and Expansion Study of Gemcitabine, Nab-Paclitaxel, and Ficlatuzumab in Patients With Metastatic Pancreatic Ca… DOI 10.1093/oncolo/oyad002↗
- Paper PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability. DOI 10.1158/2159-8290.cd-20-1098↗
- Paper Regulation of Epithelial Plasticity Determines Metastatic Organotropism in Pancreatic Cancer. DOI 10.1016/j.devcel.2018.05.025↗
- Maximilian Reichert · 4 shared papers, 2016–2018 · strength 0.26
- Paper Pancreas 3D Organoids: Current and Future Aspects as a Research Platform for Personalized Medicine in Pancreatic Cancer. DOI 10.1016/j.jcmgh.2017.12.004↗
- Paper Regulation of Epithelial Plasticity Determines Metastatic Organotropism in Pancreatic Cancer. DOI 10.1016/j.devcel.2018.05.025↗
- Paper Prrx1 isoform switching regulates pancreatic cancer invasion and metastatic colonization. DOI 10.1101/gad.263327.115↗
- Koushik K. Das · 3 shared papers, 2016–2023 · strength 0.23
- Paper Phase Ib and Expansion Study of Gemcitabine, Nab-Paclitaxel, and Ficlatuzumab in Patients With Metastatic Pancreatic Ca… DOI 10.1093/oncolo/oyad002↗
- Paper Prrx1 isoform switching regulates pancreatic cancer invasion and metastatic colonization. DOI 10.1101/gad.263327.115↗
- Paper ETS-Transcription Factor ETV1 Regulates Stromal Expansion and Metastasis in Pancreatic Cancer. DOI 10.1053/j.gastro.2016.06.005↗
- Shigetsugu Takano · 3 shared papers, 2016–2021 · strength 0.20
- Paper PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability. DOI 10.1158/2159-8290.cd-20-1098↗
- Paper Prrx1 isoform switching regulates pancreatic cancer invasion and metastatic colonization. DOI 10.1101/gad.263327.115↗
- Paper ETS-Transcription Factor ETV1 Regulates Stromal Expansion and Metastasis in Pancreatic Cancer. DOI 10.1053/j.gastro.2016.06.005↗
- Anirban Maitra · 3 shared papers, 2016–2021 · strength 0.20
- Paper PTHrP Drives Pancreatic Cancer Growth and Metastasis and Reveals a New Therapeutic Vulnerability. DOI 10.1158/2159-8290.cd-20-1098↗
- Paper Prrx1 isoform switching regulates pancreatic cancer invasion and metastatic colonization. DOI 10.1101/gad.263327.115↗
- Paper ETS-Transcription Factor ETV1 Regulates Stromal Expansion and Metastasis in Pancreatic Cancer. DOI 10.1053/j.gastro.2016.06.005↗
- Michael S. May · 1 shared paper, 2025 · strength 0.18
- Alexander Grenander Raufi · 1 shared paper, 2025 · strength 0.18
- Morteza Chalabi Hajkarim · 1 shared paper, 2025 · strength 0.18
- Jacob K.R. Jamison · 1 shared paper, 2025 · strength 0.18
- Benjamin Izar · 1 shared paper, 2025 · strength 0.18
- Michael D. Kluger · 1 shared paper, 2025 · strength 0.18
- John A. Chabot · 1 shared paper, 2025 · strength 0.18
- Gulam Abbas Manji · 1 shared paper, 2025 · strength 0.18
- Leticia Moreira · 2 shared papers, 2018 · strength 0.16
- Kensuke Suzuki · 2 shared papers, 2018–2021 · strength 0.16
- Benoît Marchand · 2 shared papers, 2016–2021 · strength 0.15
- Douglas Adam Rubinson · 1 shared paper, 2023 · strength 0.13
- Srivatsan Raghavan · 1 shared paper, 2023 · strength 0.13
- Thomas Adam Abrams · 1 shared paper, 2023 · strength 0.13
- Lawrence Scott Blaszkowsky · 1 shared paper, 2023 · strength 0.13
- Andrew J. Aguirre · 1 shared paper, 2023 · strength 0.13
- Matthew B.B. Yurgelun · 1 shared paper, 2023 · strength 0.13
- Brian Matthew Wolpin · 1 shared paper, 2023 · strength 0.13
- Kimberly J. Perez · 1 shared paper, 2023 · strength 0.13
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.