ResearcherFox Chase Cancer CenterActive in the last 2 years
Erica A. Golemis
Matched on ORCID iD 0000-0003-3618-3673; OpenAlex author ID A5004302234. Sources: OpenAlex.
Their work
- Papers
- 6 (first author 0, last author 2)
- Trials
- 0
- Grants
- 0
- Labs led
- 1
- Collaborators
- 23
2016: 1 paper · 2017: 1 paper · 2018: none · 2019: none · 2020: 1 paper · 2021: 1 paper · 2022: none · 2023: 1 paper · 2024: none · 2025: none · 2026: 1 paper
Papers 6
- ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppression. Co-author DOI 10.1038/s43018-026-01265-5↗
- Intrapancreatic fat, pancreatitis, and pancreatic cancer. Last author DOI 10.1007/s00018-023-04855-z↗
- Evaluation of the Small-molecule BRD4 Degrader CFT-2718 in Small-cell Lung Cancer and Pancreatic Cancer Models. Last author DOI 10.1158/1535-7163.mct-20-0831↗
- Cholesterol Pathway Inhibition Induces TGF-β Signaling to Promote Basal Differentiation in Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2020.08.015↗
- Targeted delivery of chemotherapy using HSP90 inhibitor drug conjugates is highly active against pancreatic cancer models. Co-author DOI 10.18632/oncotarget.12642↗
- Screening of Conditionally Reprogrammed Patient-Derived Carcinoma Cells Identifies ERCC3-MYC Interactions as a Target in Pancreatic Cancer. Co-author DOI 10.1158/1078-0432.ccr-16-0149↗
Closest collaborators
The 23 strongest of 23 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Igor A. Astsaturov · 5 shared papers, 2016–2026 · strength 0.68
- Paper Intrapancreatic fat, pancreatitis, and pancreatic cancer. DOI 10.1007/s00018-023-04855-z↗
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Cholesterol Pathway Inhibition Induces TGF-β Signaling to Promote Basal Differentiation in Pancreatic Cancer. DOI 10.1016/j.ccell.2020.08.015↗
- Anna C. Lilly · 3 shared papers, 2021–2026 · strength 0.60
- Paper Intrapancreatic fat, pancreatitis, and pancreatic cancer. DOI 10.1007/s00018-023-04855-z↗
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Evaluation of the Small-molecule BRD4 Degrader CFT-2718 in Small-cell Lung Cancer and Pancreatic Cancer Models. DOI 10.1158/1535-7163.mct-20-0831↗
- Ralph A. Francescone III · 2 shared papers, 2020–2026 · strength 0.26
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Cholesterol Pathway Inhibition Induces TGF-β Signaling to Promote Basal Differentiation in Pancreatic Cancer. DOI 10.1016/j.ccell.2020.08.015↗
- Charline Ogier · 2 shared papers, 2020–2026 · strength 0.26
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Cholesterol Pathway Inhibition Induces TGF-β Signaling to Promote Basal Differentiation in Pancreatic Cancer. DOI 10.1016/j.ccell.2020.08.015↗
- Linara Gabitova-Cornell · 2 shared papers, 2020–2026 · strength 0.26
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Cholesterol Pathway Inhibition Induces TGF-β Signaling to Promote Basal Differentiation in Pancreatic Cancer. DOI 10.1016/j.ccell.2020.08.015↗
- Janusz Franco‐Barraza · 2 shared papers, 2020–2026 · strength 0.26
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Cholesterol Pathway Inhibition Induces TGF-β Signaling to Promote Basal Differentiation in Pancreatic Cancer. DOI 10.1016/j.ccell.2020.08.015↗
- Edna Cukierman · 2 shared papers, 2020–2026 · strength 0.26
- Paper ANO6-mediated trogocytosis of cancer-associated fibroblasts promotes pancreatic cancer cell growth and immune suppressi… DOI 10.1038/s43018-026-01265-5↗
- Paper Cholesterol Pathway Inhibition Induces TGF-β Signaling to Promote Basal Differentiation in Pancreatic Cancer. DOI 10.1016/j.ccell.2020.08.015↗
- Nathaniel W. Snyder · 1 shared paper, 2026 · strength 0.18
- Debora Barbosa Vendramini Costa · 1 shared paper, 2026 · strength 0.18
- Tiffany Luong · 1 shared paper, 2026 · strength 0.18
- Corinne Bousquet · 1 shared paper, 2026 · strength 0.18
- Jerome F. Cros · 1 shared paper, 2026 · strength 0.18
- Serge Y Fuchs · 1 shared paper, 2026 · strength 0.18
- Rolf A. Brekken · 1 shared paper, 2026 · strength 0.18
- Danlin Sun · 1 shared paper, 2021 · strength 0.11
- Egor Bobrov · 2 shared papers, 2016–2017 · strength 0.11
- Paper Targeted delivery of chemotherapy using HSP90 inhibitor drug conjugates is highly active against pancreatic cancer mode… DOI 10.18632/oncotarget.12642↗
- Paper Screening of Conditionally Reprogrammed Patient-Derived Carcinoma Cells Identifies ERCC3-MYC Interactions as a Target i… DOI 10.1158/1078-0432.ccr-16-0149↗
- Natalya Beglyarova · 2 shared papers, 2016–2017 · strength 0.11
- Paper Targeted delivery of chemotherapy using HSP90 inhibitor drug conjugates is highly active against pancreatic cancer mode… DOI 10.18632/oncotarget.12642↗
- Paper Screening of Conditionally Reprogrammed Patient-Derived Carcinoma Cells Identifies ERCC3-MYC Interactions as a Target i… DOI 10.1158/1078-0432.ccr-16-0149↗
- Alana M. O’Reilly · 1 shared paper, 2020 · strength 0.08
- Richard A. Moffitt · 1 shared paper, 2020 · strength 0.08
- Carolyn Y. Fang · 1 shared paper, 2020 · strength 0.08
- Diana Behrens · 1 shared paper, 2016 · strength 0.04
- John Parker Hoffman · 1 shared paper, 2016 · strength 0.04
- Louis M. Weiner · 1 shared paper, 2016 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.