ResearcherMayo ClinicActive in the last 2 years
Nickolas Papadopoulos
Matched on ORCID iD 0000-0001-7135-7451; OpenAlex author ID A5037021577; NIH RePORTER profile ID 9901413. Sources: OpenAlex.
Their work
- Papers
- 9
- Trials
- 0
- Grants
- 1
- Labs led
- 0
- Collaborators
- 66
2012: 1 paper · 2013: none · 2014: 1 paper · 2015: 2 papers · 2016: 1 paper · 2017: 1 paper · 2018: none · 2019: 1 paper · 2020: none · 2021: none · 2022: none · 2023: none · 2024: none · 2025: 2 papers · 2026: 1 grant
Papers 9
- ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxylate transporter MCT1. Co-author DOI 10.7554/elife.94488↗
- ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxylate transporter MCT1 Co-author DOI 10.7554/elife.94488.3↗
- Circulating tumor DNA as a potential marker of adjuvant chemotherapy benefit following surgery for localized pancreatic cancer. Co-author DOI 10.1093/annonc/mdz200↗
- Limited heterogeneity of known driver gene mutations among the metastases of individual patients with pancreatic cancer. Co-author DOI 10.1038/ng.3764↗
- Whole Genome Sequencing Defines the Genetic Heterogeneity of Familial Pancreatic Cancer. Co-author DOI 10.1158/2159-8290.cd-15-0402↗
- Familial and sporadic pancreatic cancer share the same molecular pathogenesis. Co-author DOI 10.1007/s10689-014-9755-y↗
- Very Long-term Survival Following Resection for Pancreatic Cancer Is Not Explained by Commonly Mutated Genes: Results of Whole-Exome Sequencing Analysis. Co-author DOI 10.1158/1078-0432.ccr-14-2600↗
- The early detection of pancreatic cancer: what will it take to diagnose and treat curable pancreatic neoplasia? Co-author DOI 10.1158/0008-5472.can-14-0734↗
- ATM mutations in patients with hereditary pancreatic cancer. Co-author DOI 10.1158/2159-8290.cd-11-0194↗
Grants 1
Closest collaborators
The 25 strongest of 66 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Alison P. Klein · 4 shared papers, 1 shared grant, 2012–2026 · strength 1.41
- Grant Improving Management of patients at High Risk of Pancreatic Cancer R01CA299421↗
- Paper Familial and sporadic pancreatic cancer share the same molecular pathogenesis. DOI 10.1007/s10689-014-9755-y↗
- Paper The early detection of pancreatic cancer: what will it take to diagnose and treat curable pancreatic neoplasia? DOI 10.1158/0008-5472.can-14-0734↗
- Bert Vogelstein · 9 shared papers, 2012–2025 · strength 0.72
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488↗
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488.3↗
- Paper Circulating tumor DNA as a potential marker of adjuvant chemotherapy benefit following surgery for localized pancreatic… DOI 10.1093/annonc/mdz200↗
- Kenneth W. Kinzler · 9 shared papers, 2012–2025 · strength 0.72
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488↗
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488.3↗
- Paper Circulating tumor DNA as a potential marker of adjuvant chemotherapy benefit following surgery for localized pancreatic… DOI 10.1093/annonc/mdz200↗
- Marco Dal Molin · 3 shared papers, 2015–2025 · strength 0.45
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488↗
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488.3↗
- Paper Very Long-term Survival Following Resection for Pancreatic Cancer Is Not Explained by Commonly Mutated Genes: Results o… DOI 10.1158/1078-0432.ccr-14-2600↗
- Surojit Sur · 2 shared papers, 2025 · strength 0.41
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488↗
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488.3↗
- Jordina Rincón-Torroella · 2 shared papers, 2025 · strength 0.41
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488↗
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488.3↗
- Nilofer Saba Azad · 2 shared papers, 2025 · strength 0.41
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488↗
- Paper ME3BP-7 is a targeted cytotoxic agent that rapidly kills pancreatic cancer cells expressing high levels of monocarboxyl… DOI 10.7554/elife.94488.3↗
- Ralph H. Hruban · 6 shared papers, 2012–2017 · strength 0.24
- Paper Limited heterogeneity of known driver gene mutations among the metastases of individual patients with pancreatic cancer. DOI 10.1038/ng.3764↗
- Paper Familial and sporadic pancreatic cancer share the same molecular pathogenesis. DOI 10.1007/s10689-014-9755-y↗
- Paper Very Long-term Survival Following Resection for Pancreatic Cancer Is Not Explained by Commonly Mutated Genes: Results o… DOI 10.1158/1078-0432.ccr-14-2600↗
- Christopher Lee Wolfgang · 4 shared papers, 2014–2019 · strength 0.18
- Paper Circulating tumor DNA as a potential marker of adjuvant chemotherapy benefit following surgery for localized pancreatic… DOI 10.1093/annonc/mdz200↗
- Paper Very Long-term Survival Following Resection for Pancreatic Cancer Is Not Explained by Commonly Mutated Genes: Results o… DOI 10.1158/1078-0432.ccr-14-2600↗
- Paper The early detection of pancreatic cancer: what will it take to diagnose and treat curable pancreatic neoplasia? DOI 10.1158/0008-5472.can-14-0734↗
- Laura DeLong Wood · 3 shared papers, 2015–2017 · strength 0.13
- Paper Limited heterogeneity of known driver gene mutations among the metastases of individual patients with pancreatic cancer. DOI 10.1038/ng.3764↗
- Paper Very Long-term Survival Following Resection for Pancreatic Cancer Is Not Explained by Commonly Mutated Genes: Results o… DOI 10.1158/1078-0432.ccr-14-2600↗
- Paper Whole Genome Sequencing Defines the Genetic Heterogeneity of Familial Pancreatic Cancer. DOI 10.1158/2159-8290.cd-15-0402↗
- Nicholas Jason Roberts · 3 shared papers, 2012–2016 · strength 0.11
- Paper Familial and sporadic pancreatic cancer share the same molecular pathogenesis. DOI 10.1007/s10689-014-9755-y↗
- Paper Whole Genome Sequencing Defines the Genetic Heterogeneity of Familial Pancreatic Cancer. DOI 10.1158/2159-8290.cd-15-0402↗
- Paper ATM mutations in patients with hereditary pancreatic cancer. DOI 10.1158/2159-8290.cd-11-0194↗
- James R. Eshleman · 3 shared papers, 2012–2016 · strength 0.11
- Paper Familial and sporadic pancreatic cancer share the same molecular pathogenesis. DOI 10.1007/s10689-014-9755-y↗
- Paper Whole Genome Sequencing Defines the Genetic Heterogeneity of Familial Pancreatic Cancer. DOI 10.1158/2159-8290.cd-15-0402↗
- Paper ATM mutations in patients with hereditary pancreatic cancer. DOI 10.1158/2159-8290.cd-11-0194↗
- Anne Marie Lennon · 2 shared papers, 2014–2019 · strength 0.10
- Paper Circulating tumor DNA as a potential marker of adjuvant chemotherapy benefit following surgery for localized pancreatic… DOI 10.1093/annonc/mdz200↗
- Paper The early detection of pancreatic cancer: what will it take to diagnose and treat curable pancreatic neoplasia? DOI 10.1158/0008-5472.can-14-0734↗
- Michael Goggins · 3 shared papers, 2012–2016 · strength 0.10
- Paper The early detection of pancreatic cancer: what will it take to diagnose and treat curable pancreatic neoplasia? DOI 10.1158/0008-5472.can-14-0734↗
- Paper Whole Genome Sequencing Defines the Genetic Heterogeneity of Familial Pancreatic Cancer. DOI 10.1158/2159-8290.cd-15-0402↗
- Paper ATM mutations in patients with hereditary pancreatic cancer. DOI 10.1158/2159-8290.cd-11-0194↗
- Alvin P. Makohon-Moore · 2 shared papers, 2016–2017 · strength 0.09
- Christine A. Iacobuzio–Donahue · 2 shared papers, 2016–2017 · strength 0.09
- Alexis L. Norris · 2 shared papers, 2015–2016 · strength 0.08
- Joseph M. Herman · 2 shared papers, 2014–2016 · strength 0.07
- David Goldstein · 1 shared paper, 2019 · strength 0.07
- Benjamin N. J. Thomson · 1 shared paper, 2019 · strength 0.07
- Ben J. Lawrence · 1 shared paper, 2019 · strength 0.07
- Andrew Mark Haydon · 1 shared paper, 2019 · strength 0.07
- Adnan M. Nagrial · 1 shared paper, 2019 · strength 0.07
- Matthew Burge · 1 shared paper, 2019 · strength 0.07
- Niall Christopher Tebbutt · 1 shared paper, 2019 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.