Sho Endo

Closest collaborators

The 25 strongest of 32 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

1. Kohei Nakata · 11 shared papers, 2017–2021 · strength 0.72
   • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
2. Kenoki Ohuchida · 11 shared papers, 2017–2021 · strength 0.72
   • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
3. Toshiya Abe · 10 shared papers, 2017–2019 · strength 0.60
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
   • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
4. Yoshihiro Miyasaka · 10 shared papers, 2017–2019 · strength 0.60
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
   • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
5. Takao Ohtsuka · 10 shared papers, 2017–2019 · strength 0.60
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
   • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
6. Takashi Okumura · 9 shared papers, 2017–2019 · strength 0.54
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
   • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
7. Kazuhiro Koikawa · 9 shared papers, 2017–2019 · strength 0.54
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
   • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
8. Masafumi Nakamura · 7 shared papers, 2017–2021 · strength 0.48
   • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
9. Hiromichi Nakayama · 7 shared papers, 2017–2019 · strength 0.43
   • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
   • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
   • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
10. Shin Takesue · 7 shared papers, 2017–2019 · strength 0.43
    • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
    • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
    • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
11. Chika Iwamoto · 6 shared papers, 2017–2021 · strength 0.42
    • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
    • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
    • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
12. Yohei Ando · 5 shared papers, 2017–2019 · strength 0.33
    • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
    • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
    • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
13. Shin Kibe · 5 shared papers, 2017–2019 · strength 0.33
    • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
    • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
    • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
14. Yoshinao Oda · 5 shared papers, 2017–2019 · strength 0.29
    • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
    • Paper Clinical importance of intraoperative peritoneal cytology in patients with pancreatic cancer. DOI 10.1016/j.surg.2016.10.035↗
    • Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
15. Makoto Hashizume · 5 shared papers, 2017–2018 · strength 0.29
    • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
    • Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
    • Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
16. Akiko Sagara · 3 shared papers, 2017–2021 · strength 0.24
    • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
    • Paper S100P regulates the collective invasion of pancreatic cancer cells into the lymphatic endothelial monolayer. DOI 10.3892/ijo.2019.4812↗
    • Paper Autophagy inhibition enhances antiproliferative effect of salinomycin in pancreatic cancer cells. DOI 10.1016/j.pan.2017.08.009↗
17. Masafumi Nakamura · 4 shared papers, 2017–2018 · strength 0.23
    • Paper Basement membrane destruction by pancreatic stellate cells leads to local invasion in pancreatic ductal adenocarcinoma. DOI 10.1016/j.canlet.2018.03.031↗
    • Paper Clinical importance of intraoperative peritoneal cytology in patients with pancreatic cancer. DOI 10.1016/j.surg.2016.10.035↗
    • Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
18. Masafumi Sada · 4 shared papers, 2017–2018 · strength 0.22
    • Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
    • Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
    • Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
19. Koji Shindo · 2 shared papers, 2019–2021 · strength 0.19
    • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
    • Paper Cancer-associated acinar-to-ductal metaplasia within the invasive front of pancreatic cancer contributes to local invas… DOI 10.1016/j.canlet.2018.12.005↗
20. Kohei Horioka · 3 shared papers, 2017–2018 · strength 0.17
    • Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
    • Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
    • Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
21. Sokichi Matsumoto · 1 shared paper, 2021 · strength 0.11
    • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
22. Naoki Ikenaga · 1 shared paper, 2021 · strength 0.11
    • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
23. Pingshan Zhong · 1 shared paper, 2021 · strength 0.11
    • Paper New high-throughput screening detects compounds that suppress pancreatic stellate cell activation and attenuate pancrea… DOI 10.1016/j.pan.2021.04.002↗
24. Biao Zheng · 2 shared papers, 2017 · strength 0.11
    • Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
    • Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
25. Yusuke Mizuuchi · 2 shared papers, 2017 · strength 0.11
    • Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
    • Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.