ResearcherKyushu UniversityActive in the last 2 years
Yusuke Mizuuchi
Matched on ORCID iD 0000-0003-1460-7404; OpenAlex author ID A5077236645. Sources: OpenAlex.
Their work
- Papers
- 10
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 41
2014: 1 paper · 2015: 1 paper · 2016: 3 papers · 2017: 2 papers · 2018: none · 2019: none · 2020: none · 2021: none · 2022: 1 paper · 2023: 1 paper · 2024: none · 2025: 1 paper
Papers 10
- uPAR expression in M-MDSCs under high LDHA activity in pancreatic ductal adenocarcinoma. Co-author DOI 10.1038/s41598-025-29823-4↗
- Minimally invasive distal pancreatectomy for pancreatic cancer: cranial-to-caudal approach with identification of Gerota's fascia (with video). Co-author DOI 10.1007/s00464-023-10438-7↗
- Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. Co-author DOI 10.1186/s13046-022-02301-9↗
- Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Promotes Growth of Pancreatic Tumors in Mi… Co-author DOI 10.1053/j.gastro.2017.01.010↗
- Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty acids enhancing the invasiveness of pancr… Co-author DOI 10.18632/oncotarget.15430↗
- Nuclear expression of Y-box binding protein-1 is associated with poor prognosis in patients with pancreatic cancer and its knockdown inhibits tumor growth and … Co-author DOI 10.1002/ijc.30075↗
- CD146 attenuation in cancer-associated fibroblasts promotes pancreatic cancer progression. Co-author DOI 10.1002/mc.22409↗
- Overexpression of microRNA-5100 decreases the aggressive phenotype of pancreatic cancer cells by targeting PODXL. Co-author DOI 10.3892/ijo.2016.3389↗
- Anterior gradient 2 downregulation in a subset of pancreatic ductal adenocarcinoma is a prognostic factor indicative of epithelial-mesenchymal transition. Co-author DOI 10.1038/labinvest.2014.138↗
- Follicular pancreatitis, report of a case clinically mimicking pancreatic cancer and literature review. Co-author DOI 10.1016/j.prp.2013.09.005↗
Closest collaborators
The 25 strongest of 41 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Kenoki Ohuchida · 8 shared papers, 2015–2025 · strength 0.77
- Paper uPAR expression in M-MDSCs under high LDHA activity in pancreatic ductal adenocarcinoma. DOI 10.1038/s41598-025-29823-4↗
- Paper Minimally invasive distal pancreatectomy for pancreatic cancer: cranial-to-caudal approach with identification of Gerot… DOI 10.1007/s00464-023-10438-7↗
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Yoshinao Oda · 8 shared papers, 2015–2025 · strength 0.65
- Paper uPAR expression in M-MDSCs under high LDHA activity in pancreatic ductal adenocarcinoma. DOI 10.1038/s41598-025-29823-4↗
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Paper Nuclear expression of Y-box binding protein-1 is associated with poor prognosis in patients with pancreatic cancer and … DOI 10.1002/ijc.30075↗
- Kohei Nakata · 5 shared papers, 2017–2025 · strength 0.62
- Paper uPAR expression in M-MDSCs under high LDHA activity in pancreatic ductal adenocarcinoma. DOI 10.1038/s41598-025-29823-4↗
- Paper Minimally invasive distal pancreatectomy for pancreatic cancer: cranial-to-caudal approach with identification of Gerot… DOI 10.1007/s00464-023-10438-7↗
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Naoki Ikenaga · 3 shared papers, 2022–2025 · strength 0.52
- Paper uPAR expression in M-MDSCs under high LDHA activity in pancreatic ductal adenocarcinoma. DOI 10.1038/s41598-025-29823-4↗
- Paper Minimally invasive distal pancreatectomy for pancreatic cancer: cranial-to-caudal approach with identification of Gerot… DOI 10.1007/s00464-023-10438-7↗
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Masafumi Nakamura · 5 shared papers, 2016–2023 · strength 0.47
- Paper Minimally invasive distal pancreatectomy for pancreatic cancer: cranial-to-caudal approach with identification of Gerot… DOI 10.1007/s00464-023-10438-7↗
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Chika Iwamoto · 4 shared papers, 2017–2025 · strength 0.44
- Paper uPAR expression in M-MDSCs under high LDHA activity in pancreatic ductal adenocarcinoma. DOI 10.1038/s41598-025-29823-4↗
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Toshiya Abe · 4 shared papers, 2017–2023 · strength 0.42
- Paper Minimally invasive distal pancreatectomy for pancreatic cancer: cranial-to-caudal approach with identification of Gerot… DOI 10.1007/s00464-023-10438-7↗
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Noboru Ideno · 2 shared papers, 2022–2023 · strength 0.31
- Paper Minimally invasive distal pancreatectomy for pancreatic cancer: cranial-to-caudal approach with identification of Gerot… DOI 10.1007/s00464-023-10438-7↗
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Hideya Onishi · 2 shared papers, 2016–2025 · strength 0.26
- Takao Ohtsuka · 5 shared papers, 2014–2017 · strength 0.25
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Paper CD146 attenuation in cancer-associated fibroblasts promotes pancreatic cancer progression. DOI 10.1002/mc.22409↗
- Bo Zhang · 1 shared paper, 2025 · strength 0.20
- Chikanori Tsutsumi · 1 shared paper, 2025 · strength 0.20
- Masafumi Nakamura · 1 shared paper, 2025 · strength 0.20
- Masao Tanaka · 4 shared papers, 2014–2016 · strength 0.20
- Paper Nuclear expression of Y-box binding protein-1 is associated with poor prognosis in patients with pancreatic cancer and … DOI 10.1002/ijc.30075↗
- Paper CD146 attenuation in cancer-associated fibroblasts promotes pancreatic cancer progression. DOI 10.1002/mc.22409↗
- Paper Anterior gradient 2 downregulation in a subset of pancreatic ductal adenocarcinoma is a prognostic factor indicative of… DOI 10.1038/labinvest.2014.138↗
- Koji Shindo · 2 shared papers, 2015–2022 · strength 0.18
- Paper Subtypes in pancreatic ductal adenocarcinoma based on niche factor dependency show distinct drug treatment responses. DOI 10.1186/s13046-022-02301-9↗
- Paper Anterior gradient 2 downregulation in a subset of pancreatic ductal adenocarcinoma is a prognostic factor indicative of… DOI 10.1038/labinvest.2014.138↗
- So Nakamura · 1 shared paper, 2023 · strength 0.18
- Masafumi Nakamura · 1 shared paper, 2023 · strength 0.18
- Makoto Hashizume · 3 shared papers, 2016–2017 · strength 0.16
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Paper CD146 attenuation in cancer-associated fibroblasts promotes pancreatic cancer progression. DOI 10.1002/mc.22409↗
- Yoshihiro Miyasaka · 3 shared papers, 2016–2017 · strength 0.16
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Paper Overexpression of microRNA-5100 decreases the aggressive phenotype of pancreatic cancer cells by targeting PODXL. DOI 10.3892/ijo.2016.3389↗
- Tomohiko Shinkawa · 1 shared paper, 2022 · strength 0.13
- Yuki Mochida · 1 shared paper, 2022 · strength 0.13
- Lin Cui · 2 shared papers, 2016 · strength 0.11
- Sho Endo · 2 shared papers, 2017 · strength 0.11
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Takashi Okumura · 2 shared papers, 2017 · strength 0.11
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Masafumi Sada · 2 shared papers, 2017 · strength 0.11
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.