ResearcherKyushu UniversityNo recent work listed
Masafumi Sada
Matched on OpenAlex author ID A5109912785. Sources: OpenAlex.
Their work
- Papers
- 7
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 25
2014: 1 paper · 2015: none · 2016: 2 papers · 2017: 3 papers · 2018: 1 paper
Papers 7
- Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. Co-author DOI 10.1016/j.canlet.2017.10.010↗
- Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Promotes Growth of Pancreatic Tumors in Mi… Co-author DOI 10.1053/j.gastro.2017.01.010↗
- Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty acids enhancing the invasiveness of pancr… Co-author DOI 10.18632/oncotarget.15430↗
- Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. Co-author DOI 10.3892/ijo.2016.3829↗
- Hypoxic stellate cells of pancreatic cancer stroma regulate extracellular matrix fiber organization and cancer cell motility. Co-author DOI 10.1016/j.canlet.2016.01.016↗
- Suppression of CD51 in pancreatic stellate cells inhibits tumor growth by reducing stroma and altering tumor-stromal interaction in pancreatic cancer. Co-author DOI 10.3892/ijo.2016.3374↗
- CD166/ALCAM expression is characteristic of tumorigenicity and invasive and migratory activities of pancreatic cancer cells. Co-author DOI 10.1371/journal.pone.0107247↗
Closest collaborators
The 25 strongest of 25 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Kenoki Ohuchida · 7 shared papers, 2014–2018 · strength 0.37
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Hypoxic stellate cells of pancreatic cancer stroma regulate extracellular matrix fiber organization and cancer cell mot… DOI 10.1016/j.canlet.2016.01.016↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Takao Ohtsuka · 7 shared papers, 2014–2018 · strength 0.37
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Hypoxic stellate cells of pancreatic cancer stroma regulate extracellular matrix fiber organization and cancer cell mot… DOI 10.1016/j.canlet.2016.01.016↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Kohei Horioka · 6 shared papers, 2014–2018 · strength 0.32
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Hypoxic stellate cells of pancreatic cancer stroma regulate extracellular matrix fiber organization and cancer cell mot… DOI 10.1016/j.canlet.2016.01.016↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Takashi Okumura · 5 shared papers, 2016–2018 · strength 0.28
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Hypoxic stellate cells of pancreatic cancer stroma regulate extracellular matrix fiber organization and cancer cell mot… DOI 10.1016/j.canlet.2016.01.016↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Yoshihiro Miyasaka · 5 shared papers, 2016–2018 · strength 0.28
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Hypoxic stellate cells of pancreatic cancer stroma regulate extracellular matrix fiber organization and cancer cell mot… DOI 10.1016/j.canlet.2016.01.016↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Yoshinao Oda · 5 shared papers, 2014–2017 · strength 0.25
- Paper Hypoxic stellate cells of pancreatic cancer stroma regulate extracellular matrix fiber organization and cancer cell mot… DOI 10.1016/j.canlet.2016.01.016↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Masafumi Nakamura · 4 shared papers, 2016–2018 · strength 0.22
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Hypoxic stellate cells of pancreatic cancer stroma regulate extracellular matrix fiber organization and cancer cell mot… DOI 10.1016/j.canlet.2016.01.016↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Sho Endo · 4 shared papers, 2017–2018 · strength 0.22
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Kazuhiro Koikawa · 4 shared papers, 2017–2018 · strength 0.22
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Makoto Hashizume · 4 shared papers, 2017–2018 · strength 0.22
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Kohei Nakata · 4 shared papers, 2017–2018 · strength 0.22
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Toshiya Abe · 4 shared papers, 2017–2018 · strength 0.22
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Chika Iwamoto · 3 shared papers, 2017–2018 · strength 0.17
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Biao Zheng · 3 shared papers, 2016–2017 · strength 0.16
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Paper Suppression of CD51 in pancreatic stellate cells inhibits tumor growth by reducing stroma and altering tumor-stromal in… DOI 10.3892/ijo.2016.3374↗
- Hiromichi Nakayama · 2 shared papers, 2017–2018 · strength 0.11
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Shin Takesue · 2 shared papers, 2017–2018 · strength 0.11
- Paper Pancreatic stellate cells reorganize matrix components and lead pancreatic cancer invasion via the function of Endo180. DOI 10.1016/j.canlet.2017.10.010↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Masafumi Nakamura · 2 shared papers, 2017 · strength 0.11
- Paper Cancer-associated peritoneal mesothelial cells lead the formation of pancreatic cancer peritoneal dissemination. DOI 10.3892/ijo.2016.3829↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Yusuke Mizuuchi · 2 shared papers, 2017 · strength 0.11
- Paper Extra-pancreatic invasion induces lipolytic and fibrotic changes in the adipose microenvironment, with released fatty a… DOI 10.18632/oncotarget.15430↗
- Paper Autophagy Is Required for Activation of Pancreatic Stellate Cells, Associated With Pancreatic Cancer Progression and Pr… DOI 10.1053/j.gastro.2017.01.010↗
- Yohei Ando · 1 shared paper, 2018 · strength 0.06
- Shin Kibe · 1 shared paper, 2018 · strength 0.06
- Masaharu Murata · 1 shared paper, 2017 · strength 0.05
- Hayato Fujita · 1 shared paper, 2016 · strength 0.05
- Kenji Fujiwara · 1 shared paper, 2014 · strength 0.04
- Koji Shindo · 1 shared paper, 2014 · strength 0.04
- Masao Tanaka · 1 shared paper, 2014 · strength 0.04
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.