Pancreatic Cancer Research

ResearcherCornell UniversityActive in the last 2 years

Lewis C. Cantley

ORCID-verified (high confidence, 0.95), identity_conf@1As of 8 November 2026

Matched on ORCID iD 0000-0002-1298-7653; OpenAlex author ID A5057032257; NIH RePORTER profile ID 1879476. Sources: OpenAlex.

Their work

Papers
9 (first author 1, last author 0)
Trials
0
Grants
2
Labs led
0
Collaborators
68
Papers, trials and grants by year

2013: 1 paper · 2014: 2 papers · 2015: 1 paper · 2016: none · 2017: 1 paper · 2018: 1 paper · 2019: none · 2020: 1 paper, 1 grant · 2021: none · 2022: none · 2023: 1 paper · 2024: none · 2025: 1 grant · 2026: 1 paper

Papers 9

  1. Author Correction: Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. Co-author DOI 10.1038/s41586-026-10488-6↗
  2. RBFOX2 modulates a metastatic signature of alternative splicing in pancreatic cancer. Co-author DOI 10.1038/s41586-023-05820-3↗
  3. A covalent small molecule inhibitor of glutamate-oxaloacetate transaminase 1 impairs pancreatic cancer growth. Co-author DOI 10.1016/j.bbrc.2019.11.130↗
  4. Discovery and optimization of aspartate aminotransferase 1 inhibitors to target redox balance in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.bmcl.2018.04.061↗
  5. MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer. Co-author DOI 10.1016/j.ccell.2017.06.004↗
  6. Targeting glutamine metabolism sensitizes pancreatic cancer to PARP-driven metabolic catastrophe induced by ß-lapachone. Co-author DOI 10.1186/s40170-015-0137-1↗
  7. Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function. Co-author DOI 10.1038/nature13611↗
  8. Targeting metabolic scavenging in pancreatic cancer. First author DOI 10.1158/1078-0432.ccr-13-2570↗
  9. Glutamine supports pancreatic cancer growth through a KRAS-regulated metabolic pathway. Co-author DOI 10.1038/nature12040↗

Grants 2

  1. Core C-Computational Core P01CA117969↗
  2. Center on the Physics of Cancer Metabolism U54CA210184↗

Closest collaborators

The 25 strongest of 68 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Ronald Anthony Depinho · 3 shared papers, 1 shared grant, 2013–2026 · strength 0.71
  2. Costas Andreas Lyssiotis · 8 shared papers, 2013–2026 · strength 0.63
  3. Claudia Fischbach · 1 shared grant, 2020 · strength 0.48
  4. Gerald C Chu · 1 shared grant, 2025 · strength 0.45
  5. Phillip Andrew Futreal · 1 shared grant, 2025 · strength 0.45
  6. Cyril Henri Benes · 1 shared grant, 2025 · strength 0.45
  7. NABEEL El-BARDEESY · 1 shared grant, 2025 · strength 0.45
  8. Ralph Weissleder · 1 shared grant, 2025 · strength 0.45
  9. Raghu Kalluri · 1 shared grant, 2025 · strength 0.45
  10. Sarah P. Thayer · 1 shared grant, 2025 · strength 0.45
  11. Anirban Maitra · 1 shared grant, 2025 · strength 0.45
  12. James wesley Horner · 1 shared grant, 2025 · strength 0.45
  13. Huamin Wang · 1 shared grant, 2025 · strength 0.45
  14. Tyler Jacks · 1 shared grant, 2025 · strength 0.45
  15. Alec C. Kimmelman · 5 shared papers, 2013–2026 · strength 0.38
  16. Jason B. Fleming · 4 shared papers, 2013–2026 · strength 0.30
  17. Ya’an Kang · 3 shared papers, 2013–2026 · strength 0.26
  18. Haoqiang Ying · 3 shared papers, 2013–2026 · strength 0.26
  19. Andrea Viale · 2 shared papers, 2014–2026 · strength 0.23
  20. Avnish Kapoor · 2 shared papers, 2014–2026 · strength 0.23
  21. Alessandro Carugo · 2 shared papers, 2014–2026 · strength 0.23
  22. Piergiorgio Pettazzoni · 2 shared papers, 2014–2026 · strength 0.23
  23. Huamin Wang · 2 shared papers, 2014–2026 · strength 0.23
  24. Virginia Giuliani · 2 shared papers, 2014–2026 · strength 0.23
  25. Timothy Paul Heffernan · 2 shared papers, 2014–2026 · strength 0.23

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.