ResearcherNational Institutes of HealthActive in the last 2 years
Katelyn E. Connelly
Matched on ORCID iD 0000-0002-1693-5519; OpenAlex author ID A5018354681. Sources: OpenAlex.
Their work
- Papers
- 8
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 18
2021: 1 paper · 2022: none · 2023: none · 2024: none · 2025: 3 papers · 2026: 4 papers
Papers 8
- Alterations in the CTRB2 gene and response to chemotherapy in pancreatic cancer. Co-author DOI 10.1371/journal.pone.0343022↗
- Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas Co-author DOI 10.64898/2026.04.13.26350790↗
- Novel Genetic Risk Loci for Pancreatic Ductal Adenocarcinoma Identified in a Genome-wide Study of African Ancestry Individuals Co-author DOI 10.64898/2026.04.21.26351329↗
- Long-range chromatin interactions nominate candidate susceptibility genes at non-coding PDAC risk loci Co-author DOI 10.64898/2026.10.28.26366656↗
- Allelic effects on KLHL17 expression underlie a pancreatic cancer genome-wide association signal at chr1p36.33. Co-author DOI 10.1038/s41467-025-59109-2↗
- A Common CTRB misfolding variant associated with pancreatic cancer risk causes ER stress and inflammation in mice. Co-author DOI 10.1136/gutjnl-2024-333406↗
- Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene for pancreatic ductal adenocarcinoma. Co-author DOI 10.1136/gutjnl-2025-335152↗
- A 584 bp deletion in CTRB2 inhibits chymotrypsin B2 activity and secretion and confers risk of pancreatic cancer. Co-author DOI 10.1016/j.ajhg.2021.09.002↗
Closest collaborators
The 18 strongest of 18 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Jason W. Hoskins · 5 shared papers, 2025–2026 · strength 1.19
- Paper Alterations in the CTRB2 gene and response to chemotherapy in pancreatic cancer. DOI 10.1371/journal.pone.0343022↗
- Paper Long-range chromatin interactions nominate candidate susceptibility genes at non-coding PDAC risk loci DOI 10.64898/2026.10.28.26366656↗
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Laufey Thora Amundadottir · 5 shared papers, 2025–2026 · strength 1.19
- Paper Alterations in the CTRB2 gene and response to chemotherapy in pancreatic cancer. DOI 10.1371/journal.pone.0343022↗
- Paper Long-range chromatin interactions nominate candidate susceptibility genes at non-coding PDAC risk loci DOI 10.64898/2026.10.28.26366656↗
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Minal B. Patel · 3 shared papers, 2025–2026 · strength 0.72
- Paper Long-range chromatin interactions nominate candidate susceptibility genes at non-coding PDAC risk loci DOI 10.64898/2026.10.28.26366656↗
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- Ashley A. Jermusyk · 2 shared papers, 2025–2026 · strength 0.45
- Paper Long-range chromatin interactions nominate candidate susceptibility genes at non-coding PDAC risk loci DOI 10.64898/2026.10.28.26366656↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- Jun Zhong · 2 shared papers, 2025–2026 · strength 0.45
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- H. Efsun Arda · 2 shared papers, 2025–2026 · strength 0.45
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- Khalid Jazieh · 1 shared paper, 2026 · strength 0.27
- Kari G. Rabe · 1 shared paper, 2026 · strength 0.27
- Samuel O. Antwi · 1 shared paper, 2026 · strength 0.27
- Ann L. Oberg · 1 shared paper, 2026 · strength 0.27
- Ryan Michael Carr · 1 shared paper, 2026 · strength 0.27
- Hugues Sicotte · 1 shared paper, 2026 · strength 0.27
- Irene Felipe‐Abrio · 1 shared paper, 2025 · strength 0.20
- Evangelina López de Maturana · 1 shared paper, 2025 · strength 0.20
- Cristina Bodas · 1 shared paper, 2025 · strength 0.20
- Brice Chanez · 1 shared paper, 2025 · strength 0.20
- Núria Malats Riera · 1 shared paper, 2025 · strength 0.20
- Francisco X. Real · 1 shared paper, 2025 · strength 0.20
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.