ResearcherNational Institutes of HealthActive in the last 2 years
Jun Zhong
Matched on ORCID iD 0000-0002-3700-9434; OpenAlex author ID A5058056521, A5134403989. Sources: OpenAlex.
Their work
- Papers
- 14 (first author 1, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 6
2018: 1 paper · 2019: none · 2020: 2 papers · 2021: 2 papers · 2022: none · 2023: 2 papers · 2024: 1 paper · 2025: 2 papers · 2026: 4 papers
Papers 14
- Polygenic scores for risk of pancreatic ductal adenocarcinoma: evaluation of novel and published models. Co-author DOI 10.1038/s41698-026-01479-x↗
- Different diabetes types and pancreatic ductal adenocarcinoma: a Mendelian randomization and pathway/gene-set analysis. Co-author DOI 10.1093/jnci/djaf308↗
- Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas Co-author DOI 10.64898/2026.04.13.26350790↗
- Novel Genetic Risk Loci for Pancreatic Ductal Adenocarcinoma Identified in a Genome-wide Study of African Ancestry Individuals Co-author DOI 10.64898/2026.04.21.26351329↗
- Allelic effects on KLHL17 expression underlie a pancreatic cancer genome-wide association signal at chr1p36.33. Co-author DOI 10.1038/s41467-025-59109-2↗
- Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene for pancreatic ductal adenocarcinoma. Co-author DOI 10.1136/gutjnl-2025-335152↗
- Genome-Wide Analysis to Assess if Heavy Alcohol Consumption Modifies the Association between SNPs and Pancreatic Cancer Risk. Co-author DOI 10.1158/1055-9965.epi-24-0096↗
- Artificial intelligence and improved early detection for pancreatic cancer Co-author DOI 10.1016/j.xinn.2023.100457↗
- Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization. Co-author DOI 10.1158/1055-9965.epi-23-0453↗
- A 584 bp deletion in CTRB2 inhibits chymotrypsin B2 activity and secretion and confers risk of pancreatic cancer. First author DOI 10.1016/j.ajhg.2021.09.002↗
- Hepcidin-regulating iron metabolism genes and pancreatic ductal adenocarcinoma: a pathway analysis of genome-wide association studies. Co-author DOI 10.1093/ajcn/nqab217↗
- Genome-wide association meta-analysis identifies GP2 gene risk variants for pancreatic cancer. Co-author DOI 10.1038/s41467-020-16711-w↗
- A Transcriptome-Wide Association Study Identifies Novel Candidate Susceptibility Genes for Pancreatic Cancer. Co-author DOI 10.1093/jnci/djz246↗
- Genome-wide meta-analysis identifies five new susceptibility loci for pancreatic cancer. Co-author DOI 10.1038/s41467-018-02942-5↗
Closest collaborators
The 6 strongest of 6 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Laufey Thora Amundadottir · 3 shared papers, 2023–2026 · strength 0.75
- Paper Artificial intelligence and improved early detection for pancreatic cancer DOI 10.1016/j.xinn.2023.100457↗
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- Jason W. Hoskins · 2 shared papers, 2025–2026 · strength 0.45
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- Katelyn E. Connelly · 2 shared papers, 2025–2026 · strength 0.45
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- H. Efsun Arda · 2 shared papers, 2025–2026 · strength 0.45
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- Minal B. Patel · 2 shared papers, 2025–2026 · strength 0.45
- Paper Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas DOI 10.64898/2026.04.13.26350790↗
- Paper Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene fo… DOI 10.1136/gutjnl-2025-335152↗
- Ashley A. Jermusyk · 1 shared paper, 2025 · strength 0.18
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.