Pancreatic Cancer Research

ResearcherLeidos Biomedical Research Inc. (United States)Active in the last 2 years

Laufey Thora Amundadottir

Matched on ORCID iD 0000-0003-1859-8971; OpenAlex author ID A5042493218. Sources: OpenAlex.

Their work

Papers
52 (first author 2, last author 12)
Trials
0
Grants
0
Labs led
1
Collaborators
134
Papers, trials and grants by year

2011: 1 paper · 2012: 1 paper · 2013: 5 papers · 2014: 4 papers · 2015: 4 papers · 2016: 5 papers · 2017: 3 papers · 2018: 1 paper · 2019: 2 papers · 2020: 6 papers · 2021: 6 papers · 2022: 2 papers · 2023: 3 papers · 2024: 1 paper · 2025: 3 papers · 2026: 5 papers

Papers 50 of 52

  1. Different diabetes types and pancreatic ductal adenocarcinoma: a Mendelian randomization and pathway/gene-set analysis. Co-author DOI 10.1093/jnci/djaf308↗
  2. Alterations in the CTRB2 gene and response to chemotherapy in pancreatic cancer. Co-author DOI 10.1371/journal.pone.0343022↗
  3. Characterization of a pancreatic cancer GWAS signal suggests PDX1 buffers stress in the exocrine pancreas Last author DOI 10.64898/2026.04.13.26350790↗
  4. Novel Genetic Risk Loci for Pancreatic Ductal Adenocarcinoma Identified in a Genome-wide Study of African Ancestry Individuals Co-author DOI 10.64898/2026.04.21.26351329↗
  5. Long-range chromatin interactions nominate candidate susceptibility genes at non-coding PDAC risk loci Last author DOI 10.64898/2026.10.28.26366656↗
  6. Allelic effects on KLHL17 expression underlie a pancreatic cancer genome-wide association signal at chr1p36.33. Last author DOI 10.1038/s41467-025-59109-2↗
  7. A Common CTRB misfolding variant associated with pancreatic cancer risk causes ER stress and inflammation in mice. Co-author DOI 10.1136/gutjnl-2024-333406↗
  8. Large-scale multiomic analysis identifies non-coding somatic driver mutations and nominates ZFP36L2 as a driver gene for pancreatic ductal adenocarcinoma. Last author DOI 10.1136/gutjnl-2025-335152↗
  9. Genome-Wide Analysis to Assess if Heavy Alcohol Consumption Modifies the Association between SNPs and Pancreatic Cancer Risk. Co-author DOI 10.1158/1055-9965.epi-24-0096↗
  10. Artificial intelligence and improved early detection for pancreatic cancer Last author DOI 10.1016/j.xinn.2023.100457↗
  11. Relationship between ABO Blood Group Alleles and Pancreatic Cancer Is Modulated by Secretor (FUT2) Genotype, but Not Lewis Antigen (FUT3) Genotype. Co-author DOI 10.1158/1055-9965.epi-23-0009↗
  12. Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization. Co-author DOI 10.1158/1055-9965.epi-23-0453↗
  13. The age-dependent association of risk factors with pancreatic cancer. Co-author DOI 10.1016/j.annonc.2022.03.276↗
  14. Standardization of EUS imaging and reporting in high-risk individuals of pancreatic adenocarcinoma: consensus statement of the Pancreatic Cancer Early Detectio… Co-author DOI 10.1016/j.gie.2021.10.025↗
  15. A pooled genome-wide association study identifies pancreatic cancer susceptibility loci on chromosome 19p12 and 19p13.3 in the full-Jewish population. Co-author DOI 10.1007/s00439-020-02205-8↗
  16. A 584 bp deletion in CTRB2 inhibits chymotrypsin B2 activity and secretion and confers risk of pancreatic cancer. Last author DOI 10.1016/j.ajhg.2021.09.002↗
  17. Recommendations for a More Organized and Effective Approach to the Early Detection of Pancreatic Cancer From the PRECEDE (Pancreatic Cancer Early Detection) Co… Co-author DOI 10.1053/j.gastro.2021.08.036↗
  18. Hepcidin-regulating iron metabolism genes and pancreatic ductal adenocarcinoma: a pathway analysis of genome-wide association studies. Co-author DOI 10.1093/ajcn/nqab217↗
  19. Smoking Modifies Pancreatic Cancer Risk Loci on 2q21.3. Co-author DOI 10.1158/0008-5472.can-20-3267↗
  20. A multilayered post-GWAS assessment on genetic susceptibility to pancreatic cancer. Co-author DOI 10.1186/s13073-020-00816-4↗
  21. Genome-wide association meta-analysis identifies GP2 gene risk variants for pancreatic cancer. Co-author DOI 10.1038/s41467-020-16711-w↗
  22. A Transcriptome-Wide Association Study Identifies Novel Candidate Susceptibility Genes for Pancreatic Cancer. Last author DOI 10.1093/jnci/djz246↗
  23. Genome-Wide Association Study Data Reveal Genetic Susceptibility to Chronic Inflammatory Intestinal Diseases and Pancreatic Ductal Adenocarcinoma Risk. Co-author DOI 10.1158/0008-5472.can-20-0447↗
  24. Genetic and Circulating Biomarker Data Improve Risk Prediction for Pancreatic Cancer in the General Population. Co-author DOI 10.1158/1055-9965.epi-19-1389↗
  25. Genome-Wide Gene-Diabetes and Gene-Obesity Interaction Scan in 8,255 Cases and 11,900 Controls from PanScan and PanC4 Consortia. Co-author DOI 10.1158/1055-9965.epi-20-0275↗
  26. Mendelian Randomization Analysis of n-6 Polyunsaturated Fatty Acid Levels and Pancreatic Cancer Risk. Co-author DOI 10.1158/1055-9965.epi-20-0651↗
  27. CLPTM1L/CRR9 ectodomain interaction with GRP78 at the cell surface signals for survival and chemoresistance upon ER stress in pancreatic adenocarcinoma cells. First author DOI 10.1002/ijc.32012↗
  28. Prediagnostic Leukocyte Telomere Length and Pancreatic Cancer Survival. Co-author DOI 10.1158/1055-9965.epi-19-0577↗
  29. Pancreatic cancer risk is modulated by inflammatory potential of diet and ABO genotype: a consortia-based evaluation and replication study. Co-author DOI 10.1093/carcin/bgy072↗
  30. Integrated Genomic Characterization of Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.1016/j.ccell.2017.07.007↗
  31. Leucocyte telomere length, genetic variants at the TERT gene region and risk of pancreatic cancer. Co-author DOI 10.1136/gutjnl-2016-312510↗
  32. Impact of Sixteen Established Pancreatic Cancer Susceptibility Loci in American Jews. Co-author DOI 10.1158/1055-9965.epi-17-0262↗
  33. Functional characterization of a chr13q22.1 pancreatic cancer risk locus reveals long-range interaction and allele-specific effects on DIS3 expression. Last author DOI 10.1093/hmg/ddw300↗
  34. Circulating Metabolites and Survival Among Patients With Pancreatic Cancer. Co-author DOI 10.1093/jnci/djv409↗
  35. Pancreatic Cancer Risk Associated with Prediagnostic Plasma Levels of Leptin and Leptin Receptor Genetic Polymorphisms. Co-author DOI 10.1158/0008-5472.can-16-1699↗
  36. Association of Common Susceptibility Variants of Pancreatic Cancer in Higher-Risk Patients: A PACGENE Study. Co-author DOI 10.1158/1055-9965.epi-15-1217↗
  37. Pancreatic Cancer Genetics. Co-author DOI 10.7150/ijbs.15001↗
  38. TERT gene harbors multiple variants associated with pancreatic cancer susceptibility. Co-author DOI 10.1002/ijc.29590↗
  39. Epidemiology and Inherited Predisposition for Sporadic Pancreatic Adenocarcinoma. First author DOI 10.1016/j.hoc.2015.04.009↗
  40. Common variation at 2p13.3, 3q29, 7p13 and 17q25.1 associated with susceptibility to pancreatic cancer. Co-author DOI 10.1038/ng.3341↗
  41. Correction: vitamin d metabolic pathway genes and pancreatic cancer risk. Co-author DOI 10.1371/journal.pone.0129983↗
  42. Transcriptome analysis of pancreatic cancer reveals a tumor suppressor function for HNF1A. Last author DOI 10.1093/carcin/bgu193↗
  43. Genome-wide association study of survival in patients with pancreatic adenocarcinoma. Co-author DOI 10.1136/gutjnl-2012-303477↗
  44. CLPTM1L promotes growth and enhances aneuploidy in pancreatic cancer cells. Last author DOI 10.1158/0008-5472.can-13-3176↗
  45. Variants associated with susceptibility to pancreatic cancer and melanoma do not reciprocally affect risk. Co-author DOI 10.1158/1055-9965.epi-13-0627↗
  46. Diabetes and risk of pancreatic cancer: a pooled analysis from the pancreatic cancer cohort consortium. Co-author DOI 10.1007/s10552-012-0078-8↗
  47. Polymorphisms in genes related to one-carbon metabolism are not related to pancreatic cancer in PanScan and PanC4. Co-author DOI 10.1007/s10552-012-0138-0↗
  48. A resequence analysis of genomic loci on chromosomes 1q32.1, 5p15.33, and 13q22.1 associated with pancreatic cancer risk. Last author DOI 10.1097/mpa.0b013e318264cea5↗
  49. An integrated transcriptome and epigenome analysis identifies a novel candidate gene for pancreatic cancer. Last author DOI 10.1186/1755-8794-6-33↗
  50. An absolute risk model to identify individuals at elevated risk for pancreatic cancer in the general population. Co-author DOI 10.1371/journal.pone.0072311↗

Closest collaborators

The 25 strongest of 134 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Jason W. Hoskins · 10 shared papers, 2013–2026 · strength 1.35
  2. Katelyn E. Connelly · 5 shared papers, 2025–2026 · strength 1.19
  3. Brian Matthew Wolpin · 12 shared papers, 2013–2023 · strength 0.77
  4. Jun Zhong · 3 shared papers, 2023–2026 · strength 0.75
  5. Minal B. Patel · 3 shared papers, 2025–2026 · strength 0.72
  6. Alison P. Klein · 7 shared papers, 2016–2023 · strength 0.69
  7. Harvey A. Risch · 7 shared papers, 2013–2023 · strength 0.65
  8. Peter Kraft · 8 shared papers, 2013–2023 · strength 0.62
  9. Chen Yuan · 7 shared papers, 2016–2023 · strength 0.59
  10. Rachael Z. Stolzenberg‐Solomon · 10 shared papers, 2013–2022 · strength 0.56
  11. Ashley A. Jermusyk · 2 shared papers, 2025–2026 · strength 0.45
  12. Gloria M. Petersen · 10 shared papers, 2013–2022 · strength 0.42
  13. Edward L. Giovannucci · 7 shared papers, 2013–2022 · strength 0.41
  14. Kari G. Rabe · 4 shared papers, 2014–2026 · strength 0.41
  15. Kimmie Ng · 6 shared papers, 2016–2022 · strength 0.39
  16. Charles S. Fuchs · 8 shared papers, 2013–2020 · strength 0.39
  17. Ana Babić · 5 shared papers, 2016–2022 · strength 0.35
  18. Núria Malats · 3 shared papers, 2014–2025 · strength 0.33
  19. Samuel O. Antwi · 2 shared papers, 2018–2026 · strength 0.32
  20. Ann L. Oberg · 2 shared papers, 2018–2026 · strength 0.32
  21. JoAnn E. Manson · 5 shared papers, 2016–2020 · strength 0.29
  22. Jihye Kim · 2 shared papers, 2020–2023 · strength 0.29
  23. Ying Bao · 5 shared papers, 2013–2020 · strength 0.27
  24. Khalid Jazieh · 1 shared paper, 2026 · strength 0.27
  25. Ryan Michael Carr · 1 shared paper, 2026 · strength 0.27

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.