ResearcherMayo ClinicNo recent work listed
Jin‐San Zhang
Matched on ORCID iD 0000-0002-4436-9593; OpenAlex author ID A5048721439. Sources: OpenAlex.
Their work
- Papers
- 16 (first author 1, last author 6)
- Trials
- 0
- Grants
- 0
- Labs led
- 2
- Collaborators
- 50
2012: 1 paper · 2013: none · 2014: 1 paper · 2015: 2 papers · 2016: 3 papers · 2017: none · 2018: 1 paper · 2019: 2 papers · 2020: 4 papers · 2021: 1 paper · 2022: none · 2023: 1 paper
Papers 16
- ESE1/AGR2 axis antagonizes TGF-β-induced epithelial-mesenchymal transition in low-grade pancreatic cancer. Last author DOI 10.1002/cam4.5397↗
- Yap1-2 Isoform Is the Primary Mediator in TGF-β1 Induced EMT in Pancreatic Cancer. Last author DOI 10.3389/fonc.2021.649290↗
- EZH2 Regulates Pancreatic Cancer Subtype Identity and Tumor Progression via Transcriptional Repression of GATA6. Co-author DOI 10.1158/0008-5472.can-20-0672↗
- An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. Last author DOI 10.3389/fcell.2020.00287↗
- Corrigendum: An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. Last author DOI 10.3389/fcell.2020.594589↗
- The WW domains dictate isoform-specific regulation of YAP1 stability and pancreatic cancer cell malignancy. Last author DOI 10.7150/thno.42795↗
- Blockage of SLC31A1-dependent copper absorption increases pancreatic cancer cell autophagy to resist cell death. Co-author DOI 10.1111/cpr.12568↗
- Glycogen Synthase Kinase-3 Inhibition Sensitizes Pancreatic Cancer Cells to Chemotherapy by Abrogating the TopBP1/ATR-Mediated DNA Damage Response. Co-author DOI 10.1158/1078-0432.ccr-19-0799↗
- Fibroblast Growth Factor 10 in Pancreas Development and Pancreatic Cancer. Last author DOI 10.3389/fgene.2018.00482↗
- NFATc4 Regulates Sox9 Gene Expression in Acinar Cell Plasticity and Pancreatic Cancer Initiation. First author DOI 10.1155/2016/5272498↗
- SIRT1-Activating Compounds (STAC) Negatively Regulate Pancreatic Cancer Cell Growth and Viability Through a SIRT1 Lysosomal-Dependent Pathway. Co-author DOI 10.1158/1078-0432.ccr-15-1760↗
- GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. Co-author DOI 10.1158/1535-7163.mct-15-0309↗
- Nuclear FAM21 participates in NF-κB-dependent gene regulation in pancreatic cancer cells. Co-author DOI 10.1242/jcs.161513↗
- Antithetical NFATc1-Sox2 and p53-miR200 signaling networks govern pancreatic cancer cell plasticity. Co-author DOI 10.15252/embj.201489574↗
- Inflammation-induced NFATc1-STAT3 transcription complex promotes pancreatic cancer initiation by KrasG12D. Co-author DOI 10.1158/2159-8290.cd-13-0593↗
- Restricted heterochromatin formation links NFATc2 repressor activity with growth promotion in pancreatic cancer. Co-author DOI 10.1053/j.gastro.2011.11.001↗
Closest collaborators
The 25 strongest of 50 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Daniel D. Billadeau · 11 shared papers, 2012–2020 · strength 0.67
- Paper An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.00287↗
- Paper Corrigendum: An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.594589↗
- Paper The WW domains dictate isoform-specific regulation of YAP1 stability and pancreatic cancer cell malignancy. DOI 10.7150/thno.42795↗
- Meiyu Quan · 5 shared papers, 2020–2023 · strength 0.55
- Paper ESE1/AGR2 axis antagonizes TGF-β-induced epithelial-mesenchymal transition in low-grade pancreatic cancer. DOI 10.1002/cam4.5397↗
- Paper Yap1-2 Isoform Is the Primary Mediator in TGF-β1 Induced EMT in Pancreatic Cancer. DOI 10.3389/fonc.2021.649290↗
- Paper An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.00287↗
- Jiayu Liu · 3 shared papers, 2020–2021 · strength 0.30
- Paper Yap1-2 Isoform Is the Primary Mediator in TGF-β1 Induced EMT in Pancreatic Cancer. DOI 10.3389/fonc.2021.649290↗
- Paper An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.00287↗
- Paper Corrigendum: An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.594589↗
- Volker Ellenrieder · 6 shared papers, 2012–2020 · strength 0.29
- Paper EZH2 Regulates Pancreatic Cancer Subtype Identity and Tumor Progression via Transcriptional Repression of GATA6. DOI 10.1158/0008-5472.can-20-0672↗
- Paper NFATc4 Regulates Sox9 Gene Expression in Acinar Cell Plasticity and Pancreatic Cancer Initiation. DOI 10.1155/2016/5272498↗
- Paper GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. DOI 10.1158/1535-7163.mct-15-0309↗
- Zhenkun Lou · 3 shared papers, 2020 · strength 0.28
- Paper An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.00287↗
- Paper Corrigendum: An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.594589↗
- Paper The WW domains dictate isoform-specific regulation of YAP1 stability and pancreatic cancer cell malignancy. DOI 10.7150/thno.42795↗
- Qiang Guo · 3 shared papers, 2020 · strength 0.28
- Paper An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.00287↗
- Paper Corrigendum: An FGFR/AKT/SOX2 Signaling Axis Controls Pancreatic Cancer Stemness. DOI 10.3389/fcell.2020.594589↗
- Paper The WW domains dictate isoform-specific regulation of YAP1 stability and pancreatic cancer cell malignancy. DOI 10.7150/thno.42795↗
- Elisabeth Heßmann · 5 shared papers, 2014–2020 · strength 0.26
- Paper EZH2 Regulates Pancreatic Cancer Subtype Identity and Tumor Progression via Transcriptional Repression of GATA6. DOI 10.1158/0008-5472.can-20-0672↗
- Paper NFATc4 Regulates Sox9 Gene Expression in Acinar Cell Plasticity and Pancreatic Cancer Initiation. DOI 10.1155/2016/5272498↗
- Paper GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. DOI 10.1158/1535-7163.mct-15-0309↗
- Huijing Xu · 2 shared papers, 2020–2023 · strength 0.25
- Shiv K. Singh · 5 shared papers, 2012–2020 · strength 0.23
- Paper EZH2 Regulates Pancreatic Cancer Subtype Identity and Tumor Progression via Transcriptional Repression of GATA6. DOI 10.1158/0008-5472.can-20-0672↗
- Paper GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. DOI 10.1158/1535-7163.mct-15-0309↗
- Paper Antithetical NFATc1-Sox2 and p53-miR200 signaling networks govern pancreatic cancer cell plasticity. DOI 10.15252/embj.201489574↗
- Albrecht Neeße · 4 shared papers, 2014–2020 · strength 0.20
- Paper EZH2 Regulates Pancreatic Cancer Subtype Identity and Tumor Progression via Transcriptional Repression of GATA6. DOI 10.1158/0008-5472.can-20-0672↗
- Paper GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. DOI 10.1158/1535-7163.mct-15-0309↗
- Paper Antithetical NFATc1-Sox2 and p53-miR200 signaling networks govern pancreatic cancer cell plasticity. DOI 10.15252/embj.201489574↗
- Alexander Otto König · 4 shared papers, 2014–2016 · strength 0.17
- Paper NFATc4 Regulates Sox9 Gene Expression in Acinar Cell Plasticity and Pancreatic Cancer Initiation. DOI 10.1155/2016/5272498↗
- Paper GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. DOI 10.1158/1535-7163.mct-15-0309↗
- Paper Antithetical NFATc1-Sox2 and p53-miR200 signaling networks govern pancreatic cancer cell plasticity. DOI 10.15252/embj.201489574↗
- Li Ding · 2 shared papers, 2019–2020 · strength 0.17
- Jie Wang · 1 shared paper, 2023 · strength 0.16
- Jochen Gaedcke · 2 shared papers, 2015–2020 · strength 0.12
- Chao Gao · 1 shared paper, 2021 · strength 0.11
- Sandra Baumgart · 3 shared papers, 2012–2016 · strength 0.11
- Paper GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. DOI 10.1158/1535-7163.mct-15-0309↗
- Paper Inflammation-induced NFATc1-STAT3 transcription complex promotes pancreatic cancer initiation by KrasG12D. DOI 10.1158/2159-8290.cd-13-0593↗
- Paper Restricted heterochromatin formation links NFATc2 repressor activity with growth promotion in pancreatic cancer. DOI 10.1053/j.gastro.2011.11.001↗
- Thomas Mathias Gress · 3 shared papers, 2012–2016 · strength 0.11
- Paper GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression. DOI 10.1158/1535-7163.mct-15-0309↗
- Paper Inflammation-induced NFATc1-STAT3 transcription complex promotes pancreatic cancer initiation by KrasG12D. DOI 10.1158/2159-8290.cd-13-0593↗
- Paper Restricted heterochromatin formation links NFATc2 repressor activity with growth promotion in pancreatic cancer. DOI 10.1053/j.gastro.2011.11.001↗
- Rodrick Ndlovu · 1 shared paper, 2018 · strength 0.11
- Rongtao Zhou · 1 shared paper, 2019 · strength 0.10
- Chun‐Bo Teng · 1 shared paper, 2019 · strength 0.10
- Liang Jin · 1 shared paper, 2019 · strength 0.10
- Yi Pan · 1 shared paper, 2019 · strength 0.10
- Ze Yu · 1 shared paper, 2019 · strength 0.10
- Sheng Tai · 1 shared paper, 2019 · strength 0.10
- Wei Wang · 1 shared paper, 2020 · strength 0.09
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.