ResearcherKumamoto UniversityNo recent work listed
Kazuki Matsumura
Matched on ORCID iD 0000-0002-6204-9147; OpenAlex author ID A5102835211. Sources: OpenAlex.
Their work
- Papers
- 7
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 22
2020: 1 paper · 2021: 3 papers · 2022: 1 paper · 2023: 2 papers
Papers 7
- Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transitions via YAP/TAZ-Hedgehog signalling axis… Co-author DOI 10.1038/s41416-022-02106-9↗
- Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune suppression by downregulating PD-L1 exp… Co-author PMID 37293171↗
- Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.tranon.2022.101533↗
- Frailty is associated with poor prognosis after resection for pancreatic cancer. Co-author DOI 10.1007/s10147-021-01983-z↗
- Prognostic Value of the Preoperative Tumor Marker Index in Resected Pancreatic Ductal Adenocarcinoma: A Retrospective Single-Institution Study. Co-author DOI 10.1245/s10434-020-09022-3↗
- Biological Significance of YAP/TAZ in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3389/fonc.2021.700315↗
- Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.21873/invivo.12111↗
Closest collaborators
The 22 strongest of 22 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hiromitsu Hayashi · 7 shared papers, 2020–2023 · strength 0.93
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Biological Significance of YAP/TAZ in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fonc.2021.700315↗
- Norio Uemura · 6 shared papers, 2020–2023 · strength 0.81
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Biological Significance of YAP/TAZ in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fonc.2021.700315↗
- Zhao Liu · 5 shared papers, 2020–2023 · strength 0.70
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Biological Significance of YAP/TAZ in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fonc.2021.700315↗
- Katsunori Imai · 5 shared papers, 2020–2023 · strength 0.62
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Paper Prognostic Value of the Preoperative Tumor Marker Index in Resected Pancreatic Ductal Adenocarcinoma: A Retrospective S… DOI 10.1245/s10434-020-09022-3↗
- Hiroki Sato · 4 shared papers, 2021–2023 · strength 0.60
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Biological Significance of YAP/TAZ in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fonc.2021.700315↗
- Kosuke Mima · 4 shared papers, 2021–2023 · strength 0.56
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Shigeki Nakagawa · 4 shared papers, 2021–2023 · strength 0.56
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Tatsunori Miyata · 3 shared papers, 2021–2023 · strength 0.44
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Prognostic Value of the Preoperative Tumor Marker Index in Resected Pancreatic Ductal Adenocarcinoma: A Retrospective S… DOI 10.1245/s10434-020-09022-3↗
- Hideo Baba · 3 shared papers, 2020–2023 · strength 0.39
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Paper Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinom… DOI 10.21873/invivo.12111↗
- Fumimasa Kitamura · 3 shared papers, 2020–2022 · strength 0.34
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Paper Frailty is associated with poor prognosis after resection for pancreatic cancer. DOI 10.1007/s10147-021-01983-z↗
- Paper Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinom… DOI 10.21873/invivo.12111↗
- Rumi Itoyama · 3 shared papers, 2020–2021 · strength 0.33
- Paper Prognostic Value of the Preoperative Tumor Marker Index in Resected Pancreatic Ductal Adenocarcinoma: A Retrospective S… DOI 10.1245/s10434-020-09022-3↗
- Paper Frailty is associated with poor prognosis after resection for pancreatic cancer. DOI 10.1007/s10147-021-01983-z↗
- Paper Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinom… DOI 10.21873/invivo.12111↗
- Hideo Andreas Baba · 2 shared papers, 2021 · strength 0.26
- Takayoshi Kaida · 2 shared papers, 2021–2022 · strength 0.24
- Yosuke Nakao · 2 shared papers, 2021 · strength 0.23
- Takanobu Yamao · 2 shared papers, 2020–2021 · strength 0.22
- Paper Prognostic Value of the Preoperative Tumor Marker Index in Resected Pancreatic Ductal Adenocarcinoma: A Retrospective S… DOI 10.1245/s10434-020-09022-3↗
- Paper Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinom… DOI 10.21873/invivo.12111↗
- Hideo Baba · 1 shared paper, 2023 · strength 0.16
- Hidetoshi Nitta · 1 shared paper, 2023 · strength 0.16
- Masayo Tsukamoto · 1 shared paper, 2021 · strength 0.12
- Akira Chikamoto · 1 shared paper, 2021 · strength 0.12
- Hirohisa Okabe · 1 shared paper, 2021 · strength 0.12
- Takashi Matsumoto · 1 shared paper, 2021 · strength 0.11
- Hideo Baba · 1 shared paper, 2021 · strength 0.11
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.