ResearcherKumamoto UniversityActive in the last 2 years
Norio Uemura
Matched on OpenAlex author ID A5102340560. Sources: OpenAlex.
Their work
- Papers
- 12 (first author 2, last author 0)
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 29
2020: 2 papers · 2021: 4 papers · 2022: 1 paper · 2023: 4 papers · 2024: none · 2025: none · 2026: 1 paper
Papers 12
- Neoadjuvant Chemotherapy Diminishes Ki-67 Overexpression and Persistent Overexpression Is a Poor Prognostic Factor in Pancreatic Ductal Adenocarcinoma After Pa… Co-author DOI 10.1097/mpa.0000000000002641↗
- Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transitions via YAP/TAZ-Hedgehog signalling axis… Co-author DOI 10.1038/s41416-022-02106-9↗
- Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironment in pancreatic cancer. Co-author DOI 10.1172/jci.insight.163022↗
- Biological and Clinical Impacts of Glucose Metabolism in Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.3390/cancers15020498↗
- Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune suppression by downregulating PD-L1 exp… Co-author PMID 37293171↗
- Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.tranon.2022.101533↗
- Frailty is associated with poor prognosis after resection for pancreatic cancer. Co-author DOI 10.1007/s10147-021-01983-z↗
- Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.21873/anticanres.15071↗
- Biological Significance of YAP/TAZ in Pancreatic Ductal Adenocarcinoma. First author DOI 10.3389/fonc.2021.700315↗
- Recent advances in artificial intelligence for pancreatic ductal adenocarcinoma. First author DOI 10.3748/wjg.v27.i43.7480↗
- Successful Treatment for the Recurrent Liver Metastases of the Pancreatic Cancer by Multimodality Therapy. Co-author DOI 10.1097/mpa.0000000000001622↗
- Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.21873/invivo.12111↗
Closest collaborators
The 25 strongest of 29 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hiromitsu Hayashi · 12 shared papers, 2020–2026 · strength 1.79
- Paper Neoadjuvant Chemotherapy Diminishes Ki-67 Overexpression and Persistent Overexpression Is a Poor Prognostic Factor in P… DOI 10.1097/mpa.0000000000002641↗
- Paper Biological and Clinical Impacts of Glucose Metabolism in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/cancers15020498↗
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Zhao Liu · 7 shared papers, 2020–2023 · strength 1.04
- Paper Biological and Clinical Impacts of Glucose Metabolism in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/cancers15020498↗
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Katsunori Imai · 7 shared papers, 2020–2023 · strength 0.86
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Shigeki Nakagawa · 5 shared papers, 2021–2026 · strength 0.83
- Paper Neoadjuvant Chemotherapy Diminishes Ki-67 Overexpression and Persistent Overexpression Is a Poor Prognostic Factor in P… DOI 10.1097/mpa.0000000000002641↗
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Kazuki Matsumura · 6 shared papers, 2020–2023 · strength 0.81
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Biological Significance of YAP/TAZ in Pancreatic Ductal Adenocarcinoma. DOI 10.3389/fonc.2021.700315↗
- Hiroki Sato · 5 shared papers, 2021–2023 · strength 0.80
- Paper Biological and Clinical Impacts of Glucose Metabolism in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/cancers15020498↗
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Kosuke Mima · 5 shared papers, 2021–2023 · strength 0.69
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Fumimasa Kitamura · 5 shared papers, 2020–2023 · strength 0.60
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Hideo Baba · 4 shared papers, 2020–2023 · strength 0.52
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Tatsunori Miyata · 3 shared papers, 2021–2023 · strength 0.45
- Paper Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transiti… DOI 10.1038/s41416-022-02106-9↗
- Paper Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune… PMID 37293171↗
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Hideo Baba · 3 shared papers, 2021–2023 · strength 0.45
- Paper Biological and Clinical Impacts of Glucose Metabolism in Pancreatic Ductal Adenocarcinoma. DOI 10.3390/cancers15020498↗
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Frailty is associated with poor prognosis after resection for pancreatic cancer. DOI 10.1007/s10147-021-01983-z↗
- Toru Takematsu · 2 shared papers, 2020–2026 · strength 0.38
- Paper Neoadjuvant Chemotherapy Diminishes Ki-67 Overexpression and Persistent Overexpression Is a Poor Prognostic Factor in P… DOI 10.1097/mpa.0000000000002641↗
- Paper Successful Treatment for the Recurrent Liver Metastases of the Pancreatic Cancer by Multimodality Therapy. DOI 10.1097/mpa.0000000000001622↗
- Rumi Itoyama · 3 shared papers, 2020–2023 · strength 0.34
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Frailty is associated with poor prognosis after resection for pancreatic cancer. DOI 10.1007/s10147-021-01983-z↗
- Paper Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinom… DOI 10.21873/invivo.12111↗
- Yuki Kitano · 1 shared paper, 2026 · strength 0.27
- Hirohisa Okabe · 1 shared paper, 2026 · strength 0.27
- Shinsei Yumoto · 1 shared paper, 2026 · strength 0.27
- Masaaki Iwatsuki · 1 shared paper, 2026 · strength 0.27
- Takatsugu Ishimoto · 2 shared papers, 2021–2023 · strength 0.26
- Hideo Baba · 2 shared papers, 2020–2021 · strength 0.25
- Takayoshi Kaida · 2 shared papers, 2021–2022 · strength 0.24
- Takanobu Yamao · 2 shared papers, 2020 · strength 0.20
- Paper Successful Treatment for the Recurrent Liver Metastases of the Pancreatic Cancer by Multimodality Therapy. DOI 10.1097/mpa.0000000000001622↗
- Paper Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinom… DOI 10.21873/invivo.12111↗
- Hideo Baba · 1 shared paper, 2023 · strength 0.16
- Hidetoshi Nitta · 1 shared paper, 2023 · strength 0.16
- Hideo Andreas Baba · 1 shared paper, 2021 · strength 0.15
- Luke Bu · 1 shared paper, 2023 · strength 0.13
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.