ResearcherInternational Research Center for Medical Sciences, Kumamoto UniversityNo recent work listed
Fumimasa Kitamura
Matched on ORCID iD 0000-0002-6717-3788; OpenAlex author ID A5028668436. Sources: OpenAlex.
Their work
- Papers
- 7
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 23
2020: 1 paper · 2021: 3 papers · 2022: 2 papers · 2023: 1 paper
Papers 7
- Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironment in pancreatic cancer. Co-author DOI 10.1172/jci.insight.163022↗
- Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation in pancreatic ductal adenocarcinoma. Co-author DOI 10.1016/j.tranon.2022.101533↗
- Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. Co-author DOI 10.1111/cas.15495↗
- Frailty is associated with poor prognosis after resection for pancreatic cancer. Co-author DOI 10.1007/s10147-021-01983-z↗
- Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2021.09.007↗
- Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.21873/anticanres.15071↗
- Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.21873/invivo.12111↗
Closest collaborators
The 23 strongest of 23 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Hiromitsu Hayashi · 6 shared papers, 2020–2023 · strength 0.69
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Katsunori Imai · 6 shared papers, 2020–2023 · strength 0.69
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Norio Uemura · 5 shared papers, 2020–2023 · strength 0.60
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Rumi Itoyama · 5 shared papers, 2020–2023 · strength 0.55
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Frailty is associated with poor prognosis after resection for pancreatic cancer. DOI 10.1007/s10147-021-01983-z↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Hideo Baba · 4 shared papers, 2020–2023 · strength 0.47
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Takatsugu Ishimoto · 4 shared papers, 2021–2023 · strength 0.46
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Kosuke Mima · 3 shared papers, 2021–2023 · strength 0.37
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Paper Frailty is associated with poor prognosis after resection for pancreatic cancer. DOI 10.1007/s10147-021-01983-z↗
- Kazuki Matsumura · 3 shared papers, 2020–2022 · strength 0.34
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Paper Frailty is associated with poor prognosis after resection for pancreatic cancer. DOI 10.1007/s10147-021-01983-z↗
- Paper Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinom… DOI 10.21873/invivo.12111↗
- Luke Bu · 3 shared papers, 2021–2023 · strength 0.34
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Tomoyuki Uchihara · 3 shared papers, 2021–2022 · strength 0.34
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Hideo Baba · 2 shared papers, 2021 · strength 0.24
- Takayoshi Kaida · 2 shared papers, 2021–2022 · strength 0.24
- Shigeki Nakagawa · 2 shared papers, 2021–2022 · strength 0.24
- Zhao Liu · 2 shared papers, 2020–2022 · strength 0.23
- Paper Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation… DOI 10.1016/j.tranon.2022.101533↗
- Paper Prognostic Impact of Coagulation Activity in Patients Undergoing Curative Resection for Pancreatic Ductal Adenocarcinom… DOI 10.21873/invivo.12111↗
- Tatsunori Miyata · 2 shared papers, 2021 · strength 0.22
- Yosuke Nakao · 2 shared papers, 2021 · strength 0.21
- Kota Arima · 2 shared papers, 2021–2022 · strength 0.21
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Hiroki Sato · 1 shared paper, 2022 · strength 0.13
- Takashi Matsumoto · 1 shared paper, 2021 · strength 0.11
- Jun Zhang · 1 shared paper, 2022 · strength 0.11
- Takanobu Yamao · 1 shared paper, 2020 · strength 0.10
- Hideo Baba · 1 shared paper, 2021 · strength 0.09
- Yuki Kitano · 1 shared paper, 2021 · strength 0.09
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.