ResearcherKyushu UniversityNo recent work listed
Tomoyuki Uchihara
Matched on ORCID iD 0000-0002-8344-8761; OpenAlex author ID A5062543717. Sources: OpenAlex.
Their work
- Papers
- 5
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 26
2015: 1 paper · 2016: none · 2017: none · 2018: 1 paper · 2019: none · 2020: none · 2021: 2 papers · 2022: 1 paper
Papers 5
- Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. Co-author DOI 10.1111/cas.15495↗
- Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2021.09.007↗
- Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. Co-author DOI 10.21873/anticanres.15071↗
- Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prognosis in pancreatic cancer. Co-author DOI 10.1111/cas.13467↗
- Recurrence 11 years after complete response to gemcitabine, 5-Fluorouracil, and Cisplatin chemotherapy followed by radiotherapy in a patient with advanced panc… Co-author PMID 25964569↗
Closest collaborators
The 25 strongest of 26 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Takatsugu Ishimoto · 4 shared papers, 2018–2022 · strength 0.40
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Fumimasa Kitamura · 3 shared papers, 2021–2022 · strength 0.34
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Katsunori Imai · 3 shared papers, 2018–2021 · strength 0.29
- Paper Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma. DOI 10.21873/anticanres.15071↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Kota Arima · 3 shared papers, 2018–2022 · strength 0.27
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Luke Bu · 3 shared papers, 2018–2022 · strength 0.27
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Rumi Itoyama · 3 shared papers, 2018–2022 · strength 0.27
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Hideo Baba · 3 shared papers, 2015–2022 · strength 0.23
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Paper Recurrence 11 years after complete response to gemcitabine, 5-Fluorouracil, and Cisplatin chemotherapy followed by radi… PMID 25964569↗
- Hiromitsu Hayashi · 2 shared papers, 2021 · strength 0.22
- Tatsunori Miyata · 2 shared papers, 2021 · strength 0.22
- Norio Uemura · 1 shared paper, 2021 · strength 0.13
- Hideo Baba · 1 shared paper, 2021 · strength 0.13
- Jun Zhang · 1 shared paper, 2022 · strength 0.11
- Yosuke Nakao · 1 shared paper, 2021 · strength 0.09
- Hideo Baba · 1 shared paper, 2021 · strength 0.09
- Yuki Kitano · 1 shared paper, 2021 · strength 0.09
- Masayo Tsukamoto · 1 shared paper, 2018 · strength 0.07
- Akira Chikamoto · 1 shared paper, 2018 · strength 0.07
- Daisuke Hashimoto · 1 shared paper, 2018 · strength 0.07
- Shigeki Nakagawa · 1 shared paper, 2018 · strength 0.07
- Hirohisa Okabe · 1 shared paper, 2018 · strength 0.07
- Yo Ichi Yamashita · 1 shared paper, 2015 · strength 0.05
- Tomoharu Yoshizumi · 1 shared paper, 2015 · strength 0.05
- Norifumi Harimoto · 1 shared paper, 2015 · strength 0.05
- Shinji Itoh · 1 shared paper, 2015 · strength 0.05
- Ken Shirabe · 1 shared paper, 2015 · strength 0.05
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.