ResearcherInternational Research Center for Medical Sciences, Kumamoto UniversityNo recent work listed
Luke Bu
Matched on OpenAlex author ID A5110024934. Sources: OpenAlex.
Their work
- Papers
- 4
- Trials
- 0
- Grants
- 0
- Labs led
- 0
- Collaborators
- 20
2018: 1 paper · 2019: none · 2020: none · 2021: 1 paper · 2022: 1 paper · 2023: 1 paper
Papers 4
- Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironment in pancreatic cancer. Co-author DOI 10.1172/jci.insight.163022↗
- Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. Co-author DOI 10.1111/cas.15495↗
- Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreatic cancer. Co-author DOI 10.1016/j.canlet.2021.09.007↗
- Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prognosis in pancreatic cancer. Co-author DOI 10.1111/cas.13467↗
Closest collaborators
The 20 strongest of 20 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.
- Rumi Itoyama · 4 shared papers, 2018–2023 · strength 0.40
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Takatsugu Ishimoto · 4 shared papers, 2018–2023 · strength 0.40
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Fumimasa Kitamura · 3 shared papers, 2021–2023 · strength 0.34
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Hideo Baba · 3 shared papers, 2018–2023 · strength 0.31
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Katsunori Imai · 3 shared papers, 2018–2023 · strength 0.29
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Kota Arima · 3 shared papers, 2018–2022 · strength 0.27
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Tomoyuki Uchihara · 3 shared papers, 2018–2022 · strength 0.27
- Paper Tumor microenvironmental 15-PGDH depletion promotes fibrotic tumor formation and angiogenesis in pancreatic cancer. DOI 10.1111/cas.15495↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Paper Downregulation of 15-hydroxyprostaglandin dehydrogenase by interleukin-1β from activated macrophages leads to poor prog… DOI 10.1111/cas.13467↗
- Hiromitsu Hayashi · 2 shared papers, 2021–2023 · strength 0.22
- Paper Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironmen… DOI 10.1172/jci.insight.163022↗
- Paper Metabolic shift to serine biosynthesis through 3-PG accumulation and PHGDH induction promotes tumor growth in pancreati… DOI 10.1016/j.canlet.2021.09.007↗
- Norio Uemura · 1 shared paper, 2023 · strength 0.13
- Kosuke Mima · 1 shared paper, 2023 · strength 0.13
- Jun Zhang · 1 shared paper, 2022 · strength 0.11
- Yosuke Nakao · 1 shared paper, 2021 · strength 0.09
- Hideo Baba · 1 shared paper, 2021 · strength 0.09
- Yuki Kitano · 1 shared paper, 2021 · strength 0.09
- Tatsunori Miyata · 1 shared paper, 2021 · strength 0.09
- Masayo Tsukamoto · 1 shared paper, 2018 · strength 0.07
- Akira Chikamoto · 1 shared paper, 2018 · strength 0.07
- Daisuke Hashimoto · 1 shared paper, 2018 · strength 0.07
- Shigeki Nakagawa · 1 shared paper, 2018 · strength 0.07
- Hirohisa Okabe · 1 shared paper, 2018 · strength 0.07
How to reach this person
A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.