Pancreatic Cancer Research

ResearcherWuhan Union HospitalNo recent work listed

Qi Qin

ORCID-verified (high confidence, 0.95), identity_conf@1As of 8 November 2026

Matched on ORCID iD 0000-0003-3957-8902; OpenAlex author ID A5101895615. Sources: OpenAlex.

Their work

Papers
11 (first author 2, last author 1)
Trials
0
Grants
0
Labs led
0
Collaborators
22
Papers, trials and grants by year

2011: 1 paper · 2012: none · 2013: 5 papers · 2014: 1 paper · 2015: 1 paper · 2016: none · 2017: none · 2018: 1 paper · 2019: none · 2020: 2 papers

Papers 11

  1. Long noncoding RNA ITGB2-AS1 promotes growth and metastasis through miR-4319/RAF1 axis in pancreatic ductal adenocarcinoma. First author DOI 10.1002/jcp.29471↗
  2. LncRNA differentiation antagonizing non-protein coding RNA promotes proliferation and invasion through regulating miR-135a/NLRP37 axis in pancreatic cancer. Last author DOI 10.1007/s10637-019-00798-0↗
  3. The responsively decreased PKM2 facilitates the survival of pancreatic cancer cells in hypoglucose. Co-author DOI 10.1038/s41419-017-0158-5↗
  4. Ectopic expression of miR-494 inhibited the proliferation, invasion and chemoresistance of pancreatic cancer by regulating SIRT1 and c-Myc. Co-author DOI 10.1038/gt.2015.39↗
  5. Chronic pancreatitis and pancreatic cancer demonstrate active epithelial-mesenchymal transition profile, regulated by miR-217-SIRT1 pathway. Co-author DOI 10.1016/j.canlet.2014.08.007↗
  6. Nicotinamide prohibits proliferation and enhances chemosensitivity of pancreatic cancer cells through deregulating SIRT1 and Ras/Akt pathways. Co-author DOI 10.1016/j.pan.2013.01.001↗
  7. miR-148b functions as a tumor suppressor in pancreatic cancer by targeting AMPKα1. Co-author DOI 10.1158/1535-7163.mct-12-0534-t↗
  8. miRNA-141, downregulated in pancreatic cancer, inhibits cell proliferation and invasion by directly targeting MAP4K4. Co-author DOI 10.1158/1535-7163.mct-13-0296↗
  9. Hypermethylation of HIC1 promoter and aberrant expression of HIC1/SIRT1 might contribute to the carcinogenesis of pancreatic cancer. First author DOI 10.1245/s10434-012-2364-9↗
  10. MiR-130b is a prognostic marker and inhibits cell proliferation and invasion in pancreatic cancer through targeting STAT3. Co-author DOI 10.1371/journal.pone.0073803↗
  11. SIRT1 RNAi knockdown induces apoptosis and senescence, inhibits invasion and enhances chemosensitivity in pancreatic cancer cells. Co-author DOI 10.1038/gt.2011.81↗

Closest collaborators

The 22 strongest of 22 by collab_strength@1: shared papers, trials and grants, recent and small-team work weighing most.

  1. Chunyou Wang · 9 shared papers, 2013–2020 · strength 0.55
  2. Gang Zhao · 9 shared papers, 2011–2018 · strength 0.35
  3. Shichang Deng · 7 shared papers, 2013–2018 · strength 0.28
  4. Xiang Li · 6 shared papers, 2013–2018 · strength 0.25
  5. Shuai Zhu · 4 shared papers, 2013–2018 · strength 0.17
  6. Jungang Zhang · 5 shared papers, 2013 · strength 0.17
  7. Ming Yang · 2 shared papers, 2014–2020 · strength 0.16
  8. Heshui Wu · 2 shared papers, 2014–2020 · strength 0.16
  9. Gang Zhao · 1 shared paper, 2020 · strength 0.15
  10. Yong Tang · 1 shared paper, 2020 · strength 0.15
  11. Yao Guo · 1 shared paper, 2020 · strength 0.13
  12. Tao Yin · 1 shared paper, 2020 · strength 0.13
  13. Shi-Jiang Deng · 2 shared papers, 2014–2018 · strength 0.11
  14. Yan Jin · 2 shared papers, 2014–2018 · strength 0.11
  15. Mingliang Liu · 1 shared paper, 2018 · strength 0.08
  16. Tao Yin · 2 shared papers, 2011–2014 · strength 0.06
  17. Ming Yang · 1 shared paper, 2015 · strength 0.04
  18. Y. Liu · 1 shared paper, 2015 · strength 0.04
  19. Shikai Zhu · 1 shared paper, 2015 · strength 0.04
  20. Yi Niu · 1 shared paper, 2013 · strength 0.03
  21. Wang Cy · 1 shared paper, 2011 · strength 0.02
  22. Jing Cui · 1 shared paper, 2011 · strength 0.02

How to reach this person

A chain of introductions through people who have published, run trials or held grants together (warm_path@1: fewest links first, then the strongest). Built from public records only; no contact details are kept.